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Nkx6.1 controls a gene regulatory network required for establishing and maintaining pancreatic Beta cell identity

PLoS genetics, 2013-01, Vol.9 (1), p.e1003274-e1003274

2013

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Pancreatic neurogenin 3-expressing cells are unipotent islet precursors

Development (Cambridge), 2009-11, Vol.136 (21), p.3567-3574

2009

Volltextzugriff (PDF)

Dicer is required for haploid male germ cell differentiation in mice

PloS one, 2011-09, Vol.6 (9), p.e24821

2011

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Regeneration of pancreatic insulin-producing cells by in situ adaptive cell conversion

Current opinion in genetics & development, 2016-10, Vol.40, p.1-10

2016

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Generation of human islet cell type-specific identity genesets

Nature communications, 2022-04, Vol.13 (1), p.2020-2020, Article 2020

2022

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Pancreatic Ppy-expressing γ-cells display mixed phenotypic traits and the adaptive plasticity to engage insulin production

Nature communications, 2021-07, Vol.12 (1), p.4458-16, Article 4458

2021

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Leptin Engages a Hypothalamic Neurocircuitry to Permit Survival in the Absence of Insulin

Cell metabolism, 2013-09, Vol.18 (3), p.431-444

2013

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Author Correction: Generation of human islet cell type-specific identity genesets

Nature communications, 2024-03, Vol.15 (1), p.2574-2574, Article 2574

2024

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Tissue repair brakes: A common paradigm in the biology of regeneration

Stem cells (Dayton, Ohio), 2020-03, Vol.38 (3), p.330-339

2020

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Stress-impaired transcription factor expression and insulin secretion in transplanted human islets

The Journal of clinical investigation, 2016-05, Vol.126 (5), p.1857-1870

2016

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Pancreatic α Cell-Derived Glucagon-Related Peptides Are Required for β Cell Adaptation and Glucose Homeostasis

Cell reports (Cambridge), 2017-03, Vol.18 (13), p.3192-3203

2017

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Argonaute2 Mediates Compensatory Expansion of the Pancreatic β Cell

Cell metabolism, 2014-01, Vol.19 (1), p.122-134

2014

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Cell Heterogeneity and Paracrine Interactions in Human Islet Function: A Perspective Focused in β-Cell Regeneration Strategies

Frontiers in endocrinology (Lausanne), 2021-02, Vol.11, p.619150-619150

2021

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Stage-specific transcriptomic changes in pancreatic α-cells after massive β-cell loss

BMC genomics, 2021-08, Vol.22 (1), p.1-585, Article 585

2021

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Adult pancreatic islet endocrine cells emerge as fetal hormone-expressing cells

Cell reports (Cambridge), 2022-02, Vol.38 (7), p.110377-110377, Article 110377

2022

Volltextzugriff (PDF)

β Cell-Specific Deletion of the IL-1 Receptor Antagonist Impairs β Cell Proliferation and Insulin Secretion

Cell reports (Cambridge), 2018-02, Vol.22 (7), p.1774-1786

2018

Volltextzugriff (PDF)

Blockade of glucagon signaling prevents or reverses diabetes onset only if residual β-cells persist

eLife, 2016-04, Vol.5

2016

Volltextzugriff (PDF)

Islet cell replacement and transplantation immunology in a mouse strain with inducible diabetes

Scientific reports, 2022-05, Vol.12 (1), p.9033-9033, Article 9033

2022

Volltextzugriff (PDF)

Progenitor cell domains in the developing and adult pancreas

Cell cycle (Georgetown, Tex.), 2011-06, Vol.10 (12), p.1921-1927

2011

Volltextzugriff (PDF)

Author Correction: Pancreatic Ppy-expressing γ-cells display mixed phenotypic traits and the adaptive plasticity to engage insulin production

Nature communications, 2021-09, Vol.12 (1), p.5783-5783, Article 5783

2021

Volltextzugriff (PDF)

KATP channel blockers control glucagon secretion by distinct mechanisms: A direct stimulation of α-cells involving a [Ca2+]c rise and an indirect inhibition mediated by somatostatin

Molecular metabolism (Germany), 2021-11, Vol.53, p.101268-101268, Article 101268

2021

Volltextzugriff (PDF)

SGLT2 is not expressed in pancreatic α- and β-cells, and its inhibition does not directly affect glucagon and insulin secretion in rodents and humans

Molecular metabolism (Germany), 2020-12, Vol.42, p.101071-101071, Article 101071

2020

Volltextzugriff (PDF)

UCP2 Regulates the Glucagon Response to Fasting and Starvation

Diabetes (New York, N.Y.), 2013-05, Vol.62 (5), p.1623-1633

2013

Volltextzugriff (PDF)

Conversion of adult pancreatic α-cells to β-cells after extreme β-cell loss

Nature (London), 2010-04, Vol.464 (7292), p.1149-1154

2010

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Diabetes recovery by age-dependent conversion of pancreatic δ-cells into insulin producers

Nature (London), 2014-10, Vol.514 (7523), p.503-507

2014

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Nkx6.1 controls a gene regulatory network required for establishing and maintaining pancreatic Beta cell identity

Pancreatic neurogenin 3-expressing cells are unipotent islet precursors

Dicer is required for haploid male germ cell differentiation in mice

Regeneration of pancreatic insulin-producing cells by in situ adaptive cell conversion

Generation of human islet cell type-specific identity genesets

Pancreatic Ppy-expressing γ-cells display mixed phenotypic traits and the adaptive plasticity to engage insulin production

Leptin Engages a Hypothalamic Neurocircuitry to Permit Survival in the Absence of Insulin

Author Correction: Generation of human islet cell type-specific identity genesets

Tissue repair brakes: A common paradigm in the biology of regeneration

Stress-impaired transcription factor expression and insulin secretion in transplanted human islets

Pancreatic α Cell-Derived Glucagon-Related Peptides Are Required for β Cell Adaptation and Glucose Homeostasis

Argonaute2 Mediates Compensatory Expansion of the Pancreatic β Cell

Cell Heterogeneity and Paracrine Interactions in Human Islet Function: A Perspective Focused in β-Cell Regeneration Strategies

Stage-specific transcriptomic changes in pancreatic α-cells after massive β-cell loss

Adult pancreatic islet endocrine cells emerge as fetal hormone-expressing cells

β Cell-Specific Deletion of the IL-1 Receptor Antagonist Impairs β Cell Proliferation and Insulin Secretion

Blockade of glucagon signaling prevents or reverses diabetes onset only if residual β-cells persist

Islet cell replacement and transplantation immunology in a mouse strain with inducible diabetes

Progenitor cell domains in the developing and adult pancreas

Author Correction: Pancreatic Ppy-expressing γ-cells display mixed phenotypic traits and the adaptive plasticity to engage insulin production

KATP channel blockers control glucagon secretion by distinct mechanisms: A direct stimulation of α-cells involving a [Ca2+]c rise and an indirect inhibition mediated by somatostatin

SGLT2 is not expressed in pancreatic α- and β-cells, and its inhibition does not directly affect glucagon and insulin secretion in rodents and humans

UCP2 Regulates the Glucagon Response to Fasting and Starvation

Conversion of adult pancreatic α-cells to β-cells after extreme β-cell loss

Diabetes recovery by age-dependent conversion of pancreatic δ-cells into insulin producers

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