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OpenVigil FDA - Inspection of U.S. American Adverse Drug Events Pharmacovigilance Data and Novel Clinical Applications

PloS one, 2016-06, Vol.11 (6), p.e0157753-e0157753

2016

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The bottleneck of JNK signaling: Molecular and functional characteristics of MKK4 and MKK7

European journal of cell biology, 2011-06, Vol.90 (6), p.536-544

2011

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TNF-induced necroptosis and PARP-1-mediated necrosis represent distinct routes to programmed necrotic cell death

Cellular and molecular life sciences : CMLS, 2014-01, Vol.71 (2), p.331-348

2014

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Drug interactions of tetrahydrocannabinol and cannabidiol in cannabinoid drugs

Deutsches Ärzteblatt international, 2023-12

2023

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Verminderung des kardialen Risikoprofils von COX-Inhibitoren

Schmerzmedizin, 2021-11, Vol.37 (6), p.35-39

Herdegen, Thomas

2021

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The bright side of JNKs-Multitalented mediators in neuronal sprouting, brain development and nerve fiber regeneration

Progress in neurobiology, 2006-10, Vol.80 (2), p.84-97

2006

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Activation of cerebral peroxisome proliferator‐activated receptors gamma promotes neuroprotection by attenuation of neuronal cyclooxygenase‐2 overexpression after focal cerebral ischemia in rats

The FASEB journal, 2006-06, Vol.20 (8), p.1162-1175

2006

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c-Jun N-terminal kinase 1 (JNK1) modulates oligodendrocyte progenitor cell architecture, proliferation and myelination

Scientific reports, 2021-03, Vol.11 (1), p.7264-7264, Article 7264

2021

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AP-1 proteins in the adult brain : facts and fiction about effectors of neuroprotection and neurodegeneration: AP-1

Oncogene, 2001-04, Vol.20 (19), p.2424-2437

2001

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Neuronal c-Jun is required for successful axonal regeneration, but the effects of phosphorylation of its N-terminus are moderate

Journal of neurochemistry, 2012-05, Vol.121 (4), p.607-618

2012

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The JNK inhibitor XG-102 protects against TNBS-induced colitis

PloS one, 2012-03, Vol.7 (3), p.e30985

2012

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JNK1 Phosphorylation of SCG10 Determines Microtubule Dynamics and Axodendritic Length

The Journal of cell biology, 2006-04, Vol.173 (2), p.265-277

2006

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Specific pathophysiological functions of JNK isoforms in the brain

The European journal of neuroscience, 2005-01, Vol.21 (2), p.363-377

2005

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Drug Interactions of Tetrahydrocannabinol and Cannabidiol in Cannabinoid Drugs: Recommendations for Clinical Practice

Deutsches Ärzteblatt international, 2023-12, Vol.120 (49), p.833-840

2023

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The c-Jun transcription factor – bipotential mediator of neuronal death, survival and regeneration

Trends in neurosciences (Regular ed.), 1997-05, Vol.20 (5), p.227-231

1997

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Drug Interactions of Tetrahydrocannabinol and Cannabidiol in Cannabinoid Drugs

Deutsches Ärzteblatt international, 2023-12, Vol.120 (49), p.833-840

2023

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Cardioprotective Effect of NSAIDs

Deutsches Ärzteblatt international, 2017-08

2017

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All JNKs Can Kill, but Nuclear Localization Is Critical for Neuronal Death

The Journal of biological chemistry, 2008-07, Vol.283 (28), p.19704-19713

2008

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JNK2 Translocates to the Mitochondria and Mediates Cytochrome c Release in PC12 Cells in Response to 6-Hydroxydopamine

The Journal of biological chemistry, 2004-12, Vol.279 (53), p.55385-55392

2004

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MKK7γ1 reverses nerve growth factor signals: Proliferation and cell death instead of neuritogenesis and protection

Cellular signalling, 2011-08, Vol.23 (8), p.1281-1290

2011

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c-Jun N-Terminal Protein Kinase (JNK) 2/3 Is Specifically Activated by Stress, Mediating c-Jun Activation, in the Presence of Constitutive JNK1 Activity in Cerebellar Neurons

The Journal of neuroscience, 2002-06, Vol.22 (11), p.4335-4345

2002

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A Single c-Jun N-terminal Kinase Isoform (JNK3-p54) Is an Effector in Both Neuronal Differentiation and Cell Death

The Journal of biological chemistry, 2003-01, Vol.278 (1), p.567-572

2003

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The concerted signaling of ERK1/2 and JNKs is essential for PC12 cell neuritogenesis and converges at the level of target proteins

Molecular and cellular neuroscience, 2003-09, Vol.24 (1), p.238-249

2003

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Lithium Blocks the c-Jun Stress Response and Protects Neurons via Its Action on Glycogen Synthase Kinase 3

Molecular and Cellular Biology, 2003-09, Vol.23 (17), p.6027-6036

2003

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CD95 Ligand (Fas-L/APO-1L) and Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand Mediate Ischemia-Induced Apoptosis in Neurons

The Journal of neuroscience, 1999-05, Vol.19 (10), p.3809-3817

1999

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OpenVigil FDA - Inspection of U.S. American Adverse Drug Events Pharmacovigilance Data and Novel Clinical Applications

The bottleneck of JNK signaling: Molecular and functional characteristics of MKK4 and MKK7

TNF-induced necroptosis and PARP-1-mediated necrosis represent distinct routes to programmed necrotic cell death

Drug interactions of tetrahydrocannabinol and cannabidiol in cannabinoid drugs

Verminderung des kardialen Risikoprofils von COX-Inhibitoren

The bright side of JNKs-Multitalented mediators in neuronal sprouting, brain development and nerve fiber regeneration

Activation of cerebral peroxisome proliferator‐activated receptors gamma promotes neuroprotection by attenuation of neuronal cyclooxygenase‐2 overexpression after focal cerebral ischemia in rats

c-Jun N-terminal kinase 1 (JNK1) modulates oligodendrocyte progenitor cell architecture, proliferation and myelination

AP-1 proteins in the adult brain : facts and fiction about effectors of neuroprotection and neurodegeneration: AP-1

Neuronal c-Jun is required for successful axonal regeneration, but the effects of phosphorylation of its N-terminus are moderate

The JNK inhibitor XG-102 protects against TNBS-induced colitis

JNK1 Phosphorylation of SCG10 Determines Microtubule Dynamics and Axodendritic Length

Specific pathophysiological functions of JNK isoforms in the brain

Drug Interactions of Tetrahydrocannabinol and Cannabidiol in Cannabinoid Drugs: Recommendations for Clinical Practice

The c-Jun transcription factor – bipotential mediator of neuronal death, survival and regeneration

Drug Interactions of Tetrahydrocannabinol and Cannabidiol in Cannabinoid Drugs

Cardioprotective Effect of NSAIDs

All JNKs Can Kill, but Nuclear Localization Is Critical for Neuronal Death

JNK2 Translocates to the Mitochondria and Mediates Cytochrome c Release in PC12 Cells in Response to 6-Hydroxydopamine

MKK7γ1 reverses nerve growth factor signals: Proliferation and cell death instead of neuritogenesis and protection

c-Jun N-Terminal Protein Kinase (JNK) 2/3 Is Specifically Activated by Stress, Mediating c-Jun Activation, in the Presence of Constitutive JNK1 Activity in Cerebellar Neurons

A Single c-Jun N-terminal Kinase Isoform (JNK3-p54) Is an Effector in Both Neuronal Differentiation and Cell Death

The concerted signaling of ERK1/2 and JNKs is essential for PC12 cell neuritogenesis and converges at the level of target proteins

Lithium Blocks the c-Jun Stress Response and Protects Neurons via Its Action on Glycogen Synthase Kinase 3

CD95 Ligand (Fas-L/APO-1L) and Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand Mediate Ischemia-Induced Apoptosis in Neurons

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