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Context-specific inhibition of JNKs: overcoming the dilemma of protection and damage

Trends in pharmacological sciences (Regular ed.), 2005-09, Vol.26 (9), p.455-461

2005

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Cannabinoids reduce opioid use in older patients with pain : Retrospective three-year analysis of data from a general practice

Schmerz (Berlin, Germany), 2023-02, Vol.37 (1), p.29-37

2023

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Specific activities of individual c-Jun N-terminal kinases in the brain

Neuroscience, 2009-07, Vol.161 (4), p.951-959

2009

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AP-1 proteins in the adult brain : facts and fiction about effectors of neuroprotection and neurodegeneration: AP-1

Oncogene, 2001-04, Vol.20 (19), p.2424-2437

2001

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c-Jun N-terminal kinases trigger both degeneration and neurite outgrowth in primary hippocampal and cortical neurons

Journal of neurochemistry, 2008-02, Vol.104 (4), p.957-969

2008

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Inducible and constitutive transcription factors in the mammalian nervous system: control of gene expression by Jun, Fos and Krox, and CREB/ATF proteins

Brain Research Reviews, 1998-12, Vol.28 (3), p.370-490

1998

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The c-Jun N-terminal kinase (JNK) inhibitor XG-102 enhances the neuroprotection of hyperbaric oxygen after cerebral ischaemia in adult rats

Neuropathology and applied neurobiology, 2010-04, Vol.36 (3), p.211-224

2010

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Memory retrieval after contextual fear conditioning induces c‐Fos and JunB expression in CA1 hippocampus

Genes, brain and behavior, 2003-02, Vol.2 (1), p.3-10

2003

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Induction of immediate early gene expression by high-frequency stimulation of the subthalamic nucleus in rats

Neuroscience, 2006, Vol.138 (4), p.1377-1385

2006

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Specific temporal and spatial distribution of JUN, FOS, and KROX-24 proteins in spinal neurons following noxious transsynaptic stimulation

Journal of comparative neurology (1911), 1991-11, Vol.313 (1), p.178-191

1991

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Neuroprotection by Oxygen in Acute Transient Focal Cerebral Ischemia Is Dose Dependent and Shows Superiority of Hyperbaric Oxygenation

Cerebrovascular diseases (Basel, Switzerland), 2008-03, Vol.25 (3), p.193-201

2008

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Activity and expression of JNK1, p38 and ERK kinases, c-Jun N-terminal phosphorylation, and c- jun promoter binding in the adult rat brain following kainate-induced seizures

Neuroscience, 1999-01, Vol.91 (2), p.471-483

1999

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OpenVigil FDA - Inspection of U.S. American Adverse Drug Events Pharmacovigilance Data and Novel Clinical Applications

PloS one, 2016-06, Vol.11 (6), p.e0157753-e0157753

2016

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Normal values of quantitative T2′ in a spontaneously hypertensive stroke prone rat stem at 3 T

European journal of radiology, 2012-05, Vol.81 (5), p.985-987

2012

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Normal values of quantitative T2′ in a spontaneously hypertensive stroke prone rat stem at 3T

European journal of radiology, 2012-05, Vol.81 (5), p.985-987

2012

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Depletion of the AP-1 repressor JDP2 induces cell death similar to apoptosis

Biochimica et biophysica acta, 2005-08, Vol.1745 (1), p.29-37

2005

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JNK2 mediates TNF-induced cell death in mouse embryonic fibroblasts via regulation of both caspase and cathepsin protease pathways

Cell death and differentiation, 2004-03, Vol.11 (3), p.301-313

2004

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Block of c-Fos and JunB Expression by Antisense Oligonucleotides Inhibits Light-induced-Phase Shifts of the Mammalian Circadian Clock

The European journal of neuroscience, 1995-03, Vol.7 (3), p.388-393

1995

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Changes in peptidyl-prolyl cis/trans isomerase activity and fk506 binding protein expression following neuroprotection by fk506 in the ischemic rat brain

Neuroscience, 2003-09, Vol.120 (4), p.1037-1048

2003

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JNK and p38 stresskinases--degenerative effectors of signal-transduction-cascades in the nervous system

Progress in neurobiology, 2000-05, Vol.61 (1), p.45

2000

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Tachykinin receptor inhibition and c-Fos expression in the rat brain following formalin-induced pain

Neuroscience, 1999-12, Vol.95 (3), p.813-820

1999

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Cannabinoide reduzieren den Opioidverbrauch bei älteren Schmerzpatienten: Eine retrospektive Dreijahresauswertung einer ambulanten kassenärztlichen Praxis

Schmerz (Berlin, Germany), 2022-04, Vol.37 (1), p.29-37

2022

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Expression of activating transcription factor-2, serum response factor and cAMP/Ca response element binding protein in the adult rat brain following generalized seizures, nerve fibre lesion and ultraviolet irradiation

Neuroscience, 1997-11, Vol.81 (1), p.199-212

1997

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FKBP ligands as novel therapeutics for neurological disorders

Mini reviews in medicinal chemistry, 2001-11, Vol.1 (4), p.377

2001

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The immunophilin ligand FK506, but not GPI-1046, protects against neuronal death and inhibits c-Jun expression in the substantia nigra pars compacta following transection of the rat medial forebrain bundle

Neuroscience, 1999-12, Vol.95 (3), p.753-762

1999

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Context-specific inhibition of JNKs: overcoming the dilemma of protection and damage

Cannabinoids reduce opioid use in older patients with pain : Retrospective three-year analysis of data from a general practice

Specific activities of individual c-Jun N-terminal kinases in the brain

AP-1 proteins in the adult brain : facts and fiction about effectors of neuroprotection and neurodegeneration: AP-1

c-Jun N-terminal kinases trigger both degeneration and neurite outgrowth in primary hippocampal and cortical neurons

Inducible and constitutive transcription factors in the mammalian nervous system: control of gene expression by Jun, Fos and Krox, and CREB/ATF proteins

The c-Jun N-terminal kinase (JNK) inhibitor XG-102 enhances the neuroprotection of hyperbaric oxygen after cerebral ischaemia in adult rats

Memory retrieval after contextual fear conditioning induces c‐Fos and JunB expression in CA1 hippocampus

Induction of immediate early gene expression by high-frequency stimulation of the subthalamic nucleus in rats

Specific temporal and spatial distribution of JUN, FOS, and KROX-24 proteins in spinal neurons following noxious transsynaptic stimulation

Neuroprotection by Oxygen in Acute Transient Focal Cerebral Ischemia Is Dose Dependent and Shows Superiority of Hyperbaric Oxygenation

Activity and expression of JNK1, p38 and ERK kinases, c-Jun N-terminal phosphorylation, and c- jun promoter binding in the adult rat brain following kainate-induced seizures

OpenVigil FDA - Inspection of U.S. American Adverse Drug Events Pharmacovigilance Data and Novel Clinical Applications

Normal values of quantitative T2′ in a spontaneously hypertensive stroke prone rat stem at 3 T

Normal values of quantitative T2′ in a spontaneously hypertensive stroke prone rat stem at 3T

Depletion of the AP-1 repressor JDP2 induces cell death similar to apoptosis

JNK2 mediates TNF-induced cell death in mouse embryonic fibroblasts via regulation of both caspase and cathepsin protease pathways

Block of c-Fos and JunB Expression by Antisense Oligonucleotides Inhibits Light-induced-Phase Shifts of the Mammalian Circadian Clock

Changes in peptidyl-prolyl cis/trans isomerase activity and fk506 binding protein expression following neuroprotection by fk506 in the ischemic rat brain

JNK and p38 stresskinases--degenerative effectors of signal-transduction-cascades in the nervous system

Tachykinin receptor inhibition and c-Fos expression in the rat brain following formalin-induced pain

Cannabinoide reduzieren den Opioidverbrauch bei älteren Schmerzpatienten: Eine retrospektive Dreijahresauswertung einer ambulanten kassenärztlichen Praxis

Expression of activating transcription factor-2, serum response factor and cAMP/Ca response element binding protein in the adult rat brain following generalized seizures, nerve fibre lesion and ultraviolet irradiation

FKBP ligands as novel therapeutics for neurological disorders

The immunophilin ligand FK506, but not GPI-1046, protects against neuronal death and inhibits c-Jun expression in the substantia nigra pars compacta following transection of the rat medial forebrain bundle

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