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IKKβ/NF-κB Activation Causes Severe Muscle Wasting in Mice

Cell, 2004-10, Vol.119 (2), p.285-298

2004

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Sepsis stimulates release of myofilaments in skeletal muscle by a calcium‐dependent mechanism

The FASEB journal, 1999-08, Vol.13 (11), p.1435-1443

1999

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Catabolic Response to Stress and Injury: Implications for Regulation

World journal of surgery, 2000-12, Vol.24 (12), p.1452-1459

Hasselgren, Per‐Olof

2000

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Energy-ubiquitin-dependent muscle proteolysis during sepsis in rats is regulated by glucocorticoids

The Journal of clinical investigation, 1996-01, Vol.97 (2), p.339-348

1996

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Muscle cachexia: current concepts of intracellular mechanisms and molecular regulation

Annals of surgery, 2001-01, Vol.233 (1), p.9-17

2001

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Intracellular regulation of protein degradation during sepsis is different in fast- and slow-twitch muscle

American journal of physiology. Regulatory, integrative and comparative physiology, 1997-03, Vol.272 (3), p.849-R856

1997

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Linkage of aerobic glycolysis to sodium-potassium transport in rat skeletal muscle. Implications for increased muscle lactate production in sepsis

The Journal of clinical investigation, 1996-11, Vol.98 (10), p.2388-2397

1996

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Bilateral synchronous breast cancer and HER-2/Neu overexpression

Breast cancer research and treatment, 2002-04, Vol.72 (3), p.195-201

2002

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The ubiquitin-proteasome pathway : Review of a novel intracellular mechanism of muscle protein breakdown during sepsis and other catabolic conditions

Annals of surgery, 1997-03, Vol.225 (3), p.307-316

1997

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Ciliary neurotrophic factor is catabolic and shares with IL-6 the capacity to induce an acute phase response

American journal of physiology. Regulatory, integrative and comparative physiology, 1996-07, Vol.271 (1), p.185-R190

1996

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Burn injury stimulates multiple proteolytic pathways in skeletal muscle, including the ubiquitin-energy-dependent pathway

Journal of the American College of Surgeons, 1995-02, Vol.180 (2), p.161-170

1995

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Dexamethasone stimulates proteasome- and calcium-dependent proteolysis in cultured L6 myotubes

Shock (Augusta, Ga.), 1998-10, Vol.10 (4), p.298-306

1998

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Intestinal permeability is reduced and IL-10 levels are increased in septic IL-6 knockout mice

American journal of physiology. Regulatory, integrative and comparative physiology, 2001-09, Vol.281 (3), p.1013-R1023

2001

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Influence of burn injury on protein metabolism in different types of skeletal muscle and the role of glucocorticoids

Journal of the American College of Surgeons, 1995, Vol.180 (1), p.33-42

1995

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The expression of genes in the ubiquitin-proteasome proteolytic pathway is increased in skeletal muscle from patients with cancer

Surgery, 1999-10, Vol.126 (4), p.744-750

1999

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Induction of the stress response increases interleukin-6 production in the intestinal mucosa of endotoxaemic mice

Clinical science (1979), 2000-12, Vol.99 (6), p.489

2000

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Muscle protein metabolism during sepsis

Biochemical Society transactions, 1995-11, Vol.23 (4), p.1019-1025

Hasselgren, P O

1995

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The molecular regulation of protein breakdown following burn injury is different in fast- and slow-twitch skeletal muscle

International journal of molecular medicine, 1998-01, Vol.1 (1), p.163

1998

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Energy-ubiquitin-dependent muscle proteolysis during sepsis in rats is regulated by glucocorticoids

The Journal of clinical investigation, 1996-01, Vol.97 (2), p.339-348

1996

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Effect of the glucocorticoid receptor antagonist RU 38486 on muscle protein breakdown in sepsis

Surgery, 1991-04, Vol.109 (4), p.468-473

1991

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Sepsis stimulates nonlysosomal, energy-dependent proteolysis and increases ubiquitin mRNA levels in rat skeletal muscle

The Journal of clinical investigation, 1994-12, Vol.94 (6), p.2255-2264

1994

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Effect of Glutamine on Protein Synthesis in Isolated Intestinal Epithelial Cells

JPEN. Journal of parenteral and enteral nutrition, 1993-07, Vol.17 (4), p.307-314

1993

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Proteasome blockers inhibit protein breakdown in skeletal muscle after burn injury in rats

Clinical science (1979), 1998-08, Vol.95 (2), p.225

1998

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Sepsis is associated with increased mRNAs of the ubiquitin-proteasome proteolytic pathway in human skeletal muscle

The Journal of clinical investigation, 1997-01, Vol.99 (2), p.163-168

1997

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Endotoxemia and IL-1β Stimulate Mucosal IL-6 Production in Different Parts of the Gastrointestinal Tract

The Journal of surgical research, 1998-04, Vol.76 (1), p.27-31

1998

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IKKβ/NF-κB Activation Causes Severe Muscle Wasting in Mice

Sepsis stimulates release of myofilaments in skeletal muscle by a calcium‐dependent mechanism

Catabolic Response to Stress and Injury: Implications for Regulation

Energy-ubiquitin-dependent muscle proteolysis during sepsis in rats is regulated by glucocorticoids

Muscle cachexia: current concepts of intracellular mechanisms and molecular regulation

Intracellular regulation of protein degradation during sepsis is different in fast- and slow-twitch muscle

Linkage of aerobic glycolysis to sodium-potassium transport in rat skeletal muscle. Implications for increased muscle lactate production in sepsis

Bilateral synchronous breast cancer and HER-2/Neu overexpression

The ubiquitin-proteasome pathway : Review of a novel intracellular mechanism of muscle protein breakdown during sepsis and other catabolic conditions

Ciliary neurotrophic factor is catabolic and shares with IL-6 the capacity to induce an acute phase response

Burn injury stimulates multiple proteolytic pathways in skeletal muscle, including the ubiquitin-energy-dependent pathway

Dexamethasone stimulates proteasome- and calcium-dependent proteolysis in cultured L6 myotubes

Intestinal permeability is reduced and IL-10 levels are increased in septic IL-6 knockout mice

Influence of burn injury on protein metabolism in different types of skeletal muscle and the role of glucocorticoids

The expression of genes in the ubiquitin-proteasome proteolytic pathway is increased in skeletal muscle from patients with cancer

Induction of the stress response increases interleukin-6 production in the intestinal mucosa of endotoxaemic mice

Muscle protein metabolism during sepsis

The molecular regulation of protein breakdown following burn injury is different in fast- and slow-twitch skeletal muscle

Energy-ubiquitin-dependent muscle proteolysis during sepsis in rats is regulated by glucocorticoids

Effect of the glucocorticoid receptor antagonist RU 38486 on muscle protein breakdown in sepsis

Sepsis stimulates nonlysosomal, energy-dependent proteolysis and increases ubiquitin mRNA levels in rat skeletal muscle

Effect of Glutamine on Protein Synthesis in Isolated Intestinal Epithelial Cells

Proteasome blockers inhibit protein breakdown in skeletal muscle after burn injury in rats

Sepsis is associated with increased mRNAs of the ubiquitin-proteasome proteolytic pathway in human skeletal muscle

Endotoxemia and IL-1β Stimulate Mucosal IL-6 Production in Different Parts of the Gastrointestinal Tract

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