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Intragenic origins due to short G1 phases underlie oncogene-induced DNA replication stress

Nature (London), 2018-03, Vol.555 (7694), p.112-116

2018

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DNA Replication Stress as a Hallmark of Cancer

Annual review of pathology, 2015-01, Vol.10 (1), p.425-448

2015

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Genomic instability--an evolving hallmark of cancer

Nature reviews. Molecular cell biology, 2010-03, Vol.11 (3), p.220

2010

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Emerging common themes in regulation of PIKKs and PI3Ks

The EMBO journal, 2009-10, Vol.28 (20), p.3067-3073

2009

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Oncogene-Induced DNA Damage Model for Cancer Development

Science (American Association for the Advancement of Science), 2008-03, Vol.319 (5868), p.1352-1355

2008

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High-resolution mapping of mitotic DNA synthesis regions and common fragile sites in the human genome through direct sequencing

Cell research, 2020-11, Vol.30 (11), p.997-1008

2020

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Break-Induced Replication Repair of Damaged Forks Induces Genomic Duplications in Human Cells

Science (American Association for the Advancement of Science), 2014-01, Vol.343 (6166), p.88-91

2014

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Mammalian RAD52 Functions in Break-Induced Replication Repair of Collapsed DNA Replication Forks

Molecular cell, 2016-12, Vol.64 (6), p.1127-1134

2016

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Ultraviolet-B-mediated induction of protein-protein interactions in mammalian cells

Nature communications, 2013, Vol.4 (1), p.1779, Article 1779

2013

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RECQL4 is not critical for firing of human DNA replication origins

Scientific reports, 2024-04, Vol.14 (1), p.7708-7708, Article 7708

2024

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Impaired liver regeneration in aged mice can be rescued by silencing Hippo core kinases MST1 and MST2

EMBO molecular medicine, 2017-01, Vol.9 (1), p.46-60

2017

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Homozygous substitution of threonine 191 by proline in polymerase η causes Xeroderma pigmentosum variant

Scientific reports, 2024-01, Vol.14 (1), p.1117-1117, Article 1117

2024

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R-spondin 1 and noggin facilitate expansion of resident stem cells from non-damaged gallbladders

EMBO reports, 2016-05, Vol.17 (5), p.769-779

2016

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Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions

Nature, 2005-04, Vol.434 (7035), p.907-913

2005

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An Oligomerized 53BP1 Tudor Domain Suffices for Recognition of DNA Double-Strand Breaks

Molecular and Cellular Biology, 2009-02, Vol.29 (4), p.1050-1058

2009

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Reversal of the DNA-Binding-Induced Loop L1 Conformational Switch in an Engineered Human p53 Protein

Journal of molecular biology, 2014-02, Vol.426 (4), p.936-944

2014

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A transcription-based mechanism for oncogenic β-catenin-induced lethality in BRCA1/2-deficient cells

Nature communications, 2021-08, Vol.12 (1), p.4919-4919, Article 4919

2021

Volltextzugriff (PDF)

POLD3 Is Haploinsufficient for DNA Replication in Mice

Molecular cell, 2016-09, Vol.63 (5), p.877-883

2016

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Methylated lysine 79 of histone H3 targets 53BP1 to DNA double-strand breaks

Nature, 2004-11, Vol.432 (7015), p.406-411

2004

Volltextzugriff (PDF)

Crystal structure of a multidomain human p53 tetramer bound to the natural CDKN1A (p21) p53-response element

Molecular cancer research, 2011-11, Vol.9 (11), p.1493-1499

2011

Volltextzugriff (PDF)

An induced fit mechanism regulates p53 DNA binding kinetics to confer sequence specificity

The EMBO journal, 2011-06, Vol.30 (11), p.2167-2176

2011

Volltextzugriff (PDF)

A Model to Investigate Single-Strand DNA Responses in G1 Human Cells via a Telomere-Targeted, Nuclease-Deficient CRISPR-Cas9 System

PloS one, 2017-01, Vol.12 (1), p.e0169126-e0169126

2017

Volltextzugriff (PDF)

p53 Binding Protein 1 (53BP1) Is an Early Participant in the Cellular Response to DNA Double-Strand Breaks

The Journal of cell biology, 2000-12, Vol.151 (7), p.1381-1390

2000

Volltextzugriff (PDF)

Non-covalent SARS-CoV-2 Mpro inhibitors developed from in silico screen hits

Scientific reports, 2022-02, Vol.12 (1), p.2505-2505, Article 2505

2022

Volltextzugriff (PDF)

Deregulated Overexpression of hCdt1 and hCdc6 Promotes Malignant Behavior

Cancer research (Chicago, Ill.), 2007-11, Vol.67 (22), p.10899-10909

2007

Volltextzugriff (PDF)

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Intragenic origins due to short G1 phases underlie oncogene-induced DNA replication stress

DNA Replication Stress as a Hallmark of Cancer

Genomic instability--an evolving hallmark of cancer

Emerging common themes in regulation of PIKKs and PI3Ks

Oncogene-Induced DNA Damage Model for Cancer Development

High-resolution mapping of mitotic DNA synthesis regions and common fragile sites in the human genome through direct sequencing

Break-Induced Replication Repair of Damaged Forks Induces Genomic Duplications in Human Cells

Mammalian RAD52 Functions in Break-Induced Replication Repair of Collapsed DNA Replication Forks

Ultraviolet-B-mediated induction of protein-protein interactions in mammalian cells

RECQL4 is not critical for firing of human DNA replication origins

Impaired liver regeneration in aged mice can be rescued by silencing Hippo core kinases MST1 and MST2

Homozygous substitution of threonine 191 by proline in polymerase η causes Xeroderma pigmentosum variant

R-spondin 1 and noggin facilitate expansion of resident stem cells from non-damaged gallbladders

Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions

An Oligomerized 53BP1 Tudor Domain Suffices for Recognition of DNA Double-Strand Breaks

Reversal of the DNA-Binding-Induced Loop L1 Conformational Switch in an Engineered Human p53 Protein

A transcription-based mechanism for oncogenic β-catenin-induced lethality in BRCA1/2-deficient cells

POLD3 Is Haploinsufficient for DNA Replication in Mice

Methylated lysine 79 of histone H3 targets 53BP1 to DNA double-strand breaks

Crystal structure of a multidomain human p53 tetramer bound to the natural CDKN1A (p21) p53-response element

An induced fit mechanism regulates p53 DNA binding kinetics to confer sequence specificity

A Model to Investigate Single-Strand DNA Responses in G1 Human Cells via a Telomere-Targeted, Nuclease-Deficient CRISPR-Cas9 System

p53 Binding Protein 1 (53BP1) Is an Early Participant in the Cellular Response to DNA Double-Strand Breaks

Non-covalent SARS-CoV-2 Mpro inhibitors developed from in silico screen hits

Deregulated Overexpression of hCdt1 and hCdc6 Promotes Malignant Behavior

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