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SFRP4 promotes autophagy and blunts FSH responsiveness through inhibition of AKT signaling in ovarian granulosa cells

Cell communication and signaling, 2024-08, Vol.22 (1), p.396-17, Article 396

2024

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IGF1R Derived PI3K/AKT Signaling Maintains Growth in a Subset of Human T-Cell Acute Lymphoblastic Leukemias

PloS one, 2016-08, Vol.11 (8), p.e0161158-e0161158

2016

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Synthetic modeling reveals HOXB genes are critical for the initiation and maintenance of human leukemia

Nature communications, 2019-07, Vol.10 (1), p.2913-15, Article 2913

2019

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Inactivation of the Kinase Domain of CDK10 Prevents Tumor Growth in a Preclinical Model of Colorectal Cancer, and Is Accompanied by Downregulation of Bcl-2

Molecular cancer therapeutics, 2017-10, Vol.16 (10), p.2292-2303

2017

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3075 – DNMT3A LIMITS MYELOID SIGNALING RESPONSES IN T-CELL PROGENITORS DURING NORMAL AND LEUKEMIC DEVELOPMENT

Experimental hematology, 2024-08, Vol.137, p.104397, Article 104397

2024

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DNMT3A Limits Myeloid Signaling Responses in Committed T Cells during Normal and Leukemic Development

Blood, 2022-11, Vol.140 (Supplement 1), p.5914-5914

2022

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MYCN Is Required in Immature T-ALL and Bears Epigenetic Marks Associated with Cell Identity

Blood, 2022-11, Vol.140 (Supplement 1), p.8752-8753

2022

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2036 – INFLAMMAGING AND T-ALL: ONCOGENE-CYTOKINE INTERACTIONS AND THEIR ROLE IN LEUKEMOGENESIS

Experimental hematology, 2019-08, Vol.76, p.e1-e1

2019

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High-level IGF1R expression is required for leukemia-initiating cell activity in T-ALL and is supported by Notch signaling

The Journal of experimental medicine, 2011-08, Vol.208 (9), p.1809-1822

2011

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Epigenetic Restoration of Fetal-like IGF1 Signaling Inhibits Leukemia Stem Cell Activity

Cell stem cell, 2018-11, Vol.23 (5), p.714-726.e7

2018

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Insulin-like growth factor (IGF) signaling in T-cell acute lymphoblastic leukemia

Advances in biological regulation, 2019-12, Vol.74, p.100652-100652, Article 100652

2019

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RUNX1 promotes cell growth in human T-cell acute lymphoblastic leukemia by transcriptional regulation of key target genes

Experimental hematology, 2018-08, Vol.64, p.84-96

2018

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Ultrasensitive Detection of NOTCH1 c.7544_7545delCT Mutations in Chronic Lymphocytic Leukemia by Droplet Digital PCR Reveals High Frequency of Subclonal Mutations and Predicts Clinical Outcome in Cases with Trisomy 12

The Journal of molecular diagnostics : JMD, 2020-04, Vol.22 (4), p.571-578

2020

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Notch-mediated repression of miR-223 contributes to IGF1R regulation in T-ALL

Leukemia research, 2012-07, Vol.36 (7), p.905-911

2012

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Synthetic De Novo Modeling of Human T-Cell Acute Lymphoblastic Leukemia Reveals HOXB Genes Drive Expansion of Leukemia Cells-of-Origin and Established Tumor Clones

Blood, 2018-11, Vol.132 (Supplement 1), p.1322-1322

2018

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Acute T-cell leukemias remain dependent on Notch signaling despite PTEN and INK4A/ARF loss

Blood, 2010-02, Vol.115 (6), p.1175-1184

2010

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Establishment of paternal methylation imprint at the H19/Igf2 imprinting control region

Science advances, 2023-09, Vol.9 (36), p.eadi2050-eadi2050

2023

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Notch Signaling Represses Mir-223 in T-Cell Acute Lymphoblastic Leukemia

Blood, 2011-11, Vol.118 (21), p.4630-4630

2011

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RUNX1 Is Required For Maintenance Of Established T-ALL Blasts

Blood, 2013-11, Vol.122 (21), p.3742-3742

2013

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Collaboration Between RUNX and NOTCH Pathways in T-Cell Acute Lymphoblastic Leukemia

Blood, 2012-11, Vol.120 (21), p.1279-1279

2012

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Defined, Serum-Free Conditions for in Vitro culture of Primary Human T-ALL Blasts

Blood, 2012-11, Vol.120 (21), p.3537-3537

2012

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High-Level IGF1R Expression Is Required for Leukemia Stem Cell Activity In T-ALL and Is Supported by Notch Signaling

Blood, 2010-11, Vol.116 (21), p.3649-3649

2010

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High-level IGF1R expression is required for leukemia-initiating cell activity in T-ALL and is supported by Notch signaling

The Journal of cell biology, 2011-08, Vol.194 (3), p.i8-i8

2011

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Acute T-Cell Leukemias Remain Dependent On Notch Signaling Despite PTEN and INK4A/ARF Loss

Blood, 2009-11, Vol.114 (22), p.8-8

2009

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IGF Signaling Is Critical for Growth and Survival of T-Cell Acute Lymphoblastic Leukemia Cells and Is Potentiated by Notch Upregulation of IGF1R

Blood, 2008-11, Vol.112 (11), p.3811-3811

2008

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SFRP4 promotes autophagy and blunts FSH responsiveness through inhibition of AKT signaling in ovarian granulosa cells

IGF1R Derived PI3K/AKT Signaling Maintains Growth in a Subset of Human T-Cell Acute Lymphoblastic Leukemias

Synthetic modeling reveals HOXB genes are critical for the initiation and maintenance of human leukemia

Inactivation of the Kinase Domain of CDK10 Prevents Tumor Growth in a Preclinical Model of Colorectal Cancer, and Is Accompanied by Downregulation of Bcl-2

3075 – DNMT3A LIMITS MYELOID SIGNALING RESPONSES IN T-CELL PROGENITORS DURING NORMAL AND LEUKEMIC DEVELOPMENT

DNMT3A Limits Myeloid Signaling Responses in Committed T Cells during Normal and Leukemic Development

MYCN Is Required in Immature T-ALL and Bears Epigenetic Marks Associated with Cell Identity

2036 – INFLAMMAGING AND T-ALL: ONCOGENE-CYTOKINE INTERACTIONS AND THEIR ROLE IN LEUKEMOGENESIS

High-level IGF1R expression is required for leukemia-initiating cell activity in T-ALL and is supported by Notch signaling

Epigenetic Restoration of Fetal-like IGF1 Signaling Inhibits Leukemia Stem Cell Activity

Insulin-like growth factor (IGF) signaling in T-cell acute lymphoblastic leukemia

RUNX1 promotes cell growth in human T-cell acute lymphoblastic leukemia by transcriptional regulation of key target genes

Ultrasensitive Detection of NOTCH1 c.7544_7545delCT Mutations in Chronic Lymphocytic Leukemia by Droplet Digital PCR Reveals High Frequency of Subclonal Mutations and Predicts Clinical Outcome in Cases with Trisomy 12

Notch-mediated repression of miR-223 contributes to IGF1R regulation in T-ALL

Synthetic De Novo Modeling of Human T-Cell Acute Lymphoblastic Leukemia Reveals HOXB Genes Drive Expansion of Leukemia Cells-of-Origin and Established Tumor Clones

Acute T-cell leukemias remain dependent on Notch signaling despite PTEN and INK4A/ARF loss

Establishment of paternal methylation imprint at the H19/Igf2 imprinting control region

Notch Signaling Represses Mir-223 in T-Cell Acute Lymphoblastic Leukemia

RUNX1 Is Required For Maintenance Of Established T-ALL Blasts

Collaboration Between RUNX and NOTCH Pathways in T-Cell Acute Lymphoblastic Leukemia

Defined, Serum-Free Conditions for in Vitro culture of Primary Human T-ALL Blasts

High-Level IGF1R Expression Is Required for Leukemia Stem Cell Activity In T-ALL and Is Supported by Notch Signaling

High-level IGF1R expression is required for leukemia-initiating cell activity in T-ALL and is supported by Notch signaling

Acute T-Cell Leukemias Remain Dependent On Notch Signaling Despite PTEN and INK4A/ARF Loss

IGF Signaling Is Critical for Growth and Survival of T-Cell Acute Lymphoblastic Leukemia Cells and Is Potentiated by Notch Upregulation of IGF1R

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