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The anti-cancer drugs curaxins target spatial genome organization

Nature communications, 2019-03, Vol.10 (1), p.1441-11, Article 1441

2019

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Facilitates Chromatin Transcription Complex Is an “Accelerator” of Tumor Transformation and Potential Marker and Target of Aggressive Cancers

Cell reports (Cambridge), 2013-07, Vol.4 (1), p.159-173

2013

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Chromatin Stability as a Target for Cancer Treatment

BioEssays, 2019-01, Vol.41 (1), p.e1800141-n/a

Gurova, Katerina V.

2019

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Complex mutual regulation of facilitates chromatin transcription (FACT) subunits on both mRNA and protein levels in human cells

Cell cycle (Georgetown, Tex.), 2013-08, Vol.12 (15), p.2423-2434

2013

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Small Molecules That Reactivate p53 in Renal Cell Carcinoma Reveal a NF-κB-Dependent Mechanism of p53 Suppression in Tumors

Proceedings of the National Academy of Sciences - PNAS, 2005-11, Vol.102 (48), p.17448-17453

2005

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Uncovering the fine print of the CreERT2-LoxP system while generating a conditional knockout mouse model of Ssrp1 gene

PloS one, 2018, Vol.13 (6), p.e0199785-e0199785

2018

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The Combination of Curaxin CBL0137 and Histone Deacetylase Inhibitor Panobinostat Delays KMT2A-Rearranged Leukemia Progression

Frontiers in oncology, 2022-05, Vol.12, p.863329-863329

2022

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Mechanism of FACT removal from transcribed genes by anticancer drugs curaxins

Science advances, 2018-11, Vol.4 (11), p.eaav2131

2018

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Small-Molecule Multidrug Resistance―Associated Protein 1 Inhibitor Reversan Increases the Therapeutic Index of Chemotherapy in Mouse Models of Neuroblastoma

Cancer research (Chicago, Ill.), 2009-08, Vol.69 (16), p.6573-6580

2009

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Prevention of Chromatin Destabilization by FACT Is Crucial for Malignant Transformation

iScience, 2020-06, Vol.23 (6), p.101177-101177, Article 101177

2020

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The 3D Genome as a Target for Anticancer Therapy

Trends in molecular medicine, 2020-02, Vol.26 (2), p.141-149

2020

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Inhibition of Encephalomyocarditis Virus and Poliovirus Replication by Quinacrine: Implications for the Design and Discovery of Novel Antiviral Drugs

Journal of Virology, 2010-09, Vol.84 (18), p.9390-9397

2010

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Anti-malaria drug blocks proteotoxic stress response: Anti-cancer implications

Cell cycle (Georgetown, Tex.), 2009-12, Vol.8 (23), p.3960-3970

2009

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Role of Chromatin Damage and Chromatin Trapping of FACT in Mediating the Anticancer Cytotoxicity of DNA-Binding Small-Molecule Drugs

Cancer research (Chicago, Ill.), 2018-03, Vol.78 (6), p.1431-1443

2018

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p53 pathway in renal cell carcinoma is repressed by a dominant mechanism

Cancer research (Chicago, Ill.), 2004-03, Vol.64 (6), p.1951-1958

2004

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Separable Cell Cycle Arrest and Immune Response Elicited through Pharmacological CDK4/6 and MEK Inhibition in RASmut Disease Models

Molecular cancer therapeutics, 2024-09, p.OF1-OF14

2024

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Alkaloid-rich fraction of Ervatamia coronaria sensitizes colorectal cancer through modulating AMPK and mTOR signalling pathways

Journal of ethnopharmacology, 2022-01, Vol.283, p.114666-114666, Article 114666

2022

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FACT inhibitor CBL0137, administered in an optimized schedule, potentiates radiation therapy for glioblastoma by suppressing DNA damage repair

Journal of neuro-oncology, 2024-09

2024

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Inhibition of the FACT Complex Reduces Transcription from the Human Cytomegalovirus Major Immediate Early Promoter in Models of Lytic and Latent Replication

Journal of virology, 2016-04, Vol.90 (8), p.4249-4253

2016

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Anticancer drug candidate CBL0137, which inhibits histone chaperone FACT, is efficacious in preclinical orthotopic models of temozolomide-responsive and -resistant glioblastoma

Neuro-oncology (Charlottesville, Va.), 2017-02, Vol.19 (2), p.186-196

2017

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Inflammation and p53: A Tale of Two Stresses

Genes & cancer, 2011-04, Vol.2 (4), p.503-516

2011

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Curaxins: anticancer compounds that simultaneously suppress NF-κB and activate p53 by targeting FACT

Science translational medicine, 2011-08, Vol.3 (95), p.95ra74

2011

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Pharmacological Targeting of the Histone Chaperone Complex FACT Preferentially Eliminates Glioblastoma Stem Cells and Prolongs Survival in Preclinical Models

Cancer research (Chicago, Ill.), 2016-04, Vol.76 (8), p.2432-2442

2016

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Dual Targeting of Chromatin Stability By The Curaxin CBL0137 and Histone Deacetylase Inhibitor Panobinostat Shows Significant Preclinical Efficacy in Neuroblastoma

Clinical cancer research, 2021-08, Vol.27 (15), p.4338-4352

2021

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The anti-cancer drugs curaxins target spatial genome organization

Facilitates Chromatin Transcription Complex Is an “Accelerator” of Tumor Transformation and Potential Marker and Target of Aggressive Cancers

Therapeutic targeting of the MYC signal by inhibition of histone chaperone FACT in neuroblastoma

Chromatin Stability as a Target for Cancer Treatment

Complex mutual regulation of facilitates chromatin transcription (FACT) subunits on both mRNA and protein levels in human cells

Small Molecules That Reactivate p53 in Renal Cell Carcinoma Reveal a NF-κB-Dependent Mechanism of p53 Suppression in Tumors

Uncovering the fine print of the CreERT2-LoxP system while generating a conditional knockout mouse model of Ssrp1 gene

The Combination of Curaxin CBL0137 and Histone Deacetylase Inhibitor Panobinostat Delays KMT2A-Rearranged Leukemia Progression

Mechanism of FACT removal from transcribed genes by anticancer drugs curaxins

Small-Molecule Multidrug Resistance―Associated Protein 1 Inhibitor Reversan Increases the Therapeutic Index of Chemotherapy in Mouse Models of Neuroblastoma

Prevention of Chromatin Destabilization by FACT Is Crucial for Malignant Transformation

The 3D Genome as a Target for Anticancer Therapy

Inhibition of Encephalomyocarditis Virus and Poliovirus Replication by Quinacrine: Implications for the Design and Discovery of Novel Antiviral Drugs

Anti-malaria drug blocks proteotoxic stress response: Anti-cancer implications

Role of Chromatin Damage and Chromatin Trapping of FACT in Mediating the Anticancer Cytotoxicity of DNA-Binding Small-Molecule Drugs

p53 pathway in renal cell carcinoma is repressed by a dominant mechanism

Separable Cell Cycle Arrest and Immune Response Elicited through Pharmacological CDK4/6 and MEK Inhibition in RASmut Disease Models

Alkaloid-rich fraction of Ervatamia coronaria sensitizes colorectal cancer through modulating AMPK and mTOR signalling pathways

FACT inhibitor CBL0137, administered in an optimized schedule, potentiates radiation therapy for glioblastoma by suppressing DNA damage repair

Inhibition of the FACT Complex Reduces Transcription from the Human Cytomegalovirus Major Immediate Early Promoter in Models of Lytic and Latent Replication

Anticancer drug candidate CBL0137, which inhibits histone chaperone FACT, is efficacious in preclinical orthotopic models of temozolomide-responsive and -resistant glioblastoma

Inflammation and p53: A Tale of Two Stresses

Curaxins: anticancer compounds that simultaneously suppress NF-κB and activate p53 by targeting FACT

Pharmacological Targeting of the Histone Chaperone Complex FACT Preferentially Eliminates Glioblastoma Stem Cells and Prolongs Survival in Preclinical Models

Dual Targeting of Chromatin Stability By The Curaxin CBL0137 and Histone Deacetylase Inhibitor Panobinostat Shows Significant Preclinical Efficacy in Neuroblastoma

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