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G6PD Orchestrates Genome-Wide DNA Methylation and Gene Expression in the Vascular Wall

International journal of molecular sciences, 2023-12, Vol.24 (23), p.16727

2023

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G6PD is a critical enabler of hypoxia-induced accumulation of macrophages and platelets in mice lungs and contributor to lung inflammation

Vascular pharmacology, 2022-06, Vol.144, p.106976-106976, Article 106976

2022

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Glucose-6-phosphate dehydrogenase and MEG3 controls hypoxia-induced expression of serum response factor (SRF) and SRF-dependent genes in pulmonary smooth muscle cell

Journal of smooth muscle research = Nihon Heikatsukin Gakkai kikanshi, 2022, Vol.58, p.34

2022

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Hypoxia-induced glucose-6-phosphate dehydrogenase overexpression and -activation in pulmonary artery smooth muscle cells: implication in pulmonary hypertension

American journal of physiology. Lung cellular and molecular physiology, 2015-02, Vol.308 (3), p.L287-L300

2015

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Hallmarks of Pulmonary Hypertension: Mesenchymal and Inflammatory Cell Metabolic Reprogramming

Antioxidants & redox signaling, 2018-01, Vol.28 (3), p.230

2018

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Inhibition of Glucose-6-Phosphate Dehydrogenase Activity Attenuates Right Ventricle Pressure and Hypertrophy Elicited by VEGFR Inhibitor + Hypoxia

The Journal of pharmacology and experimental therapeutics, 2021-05, Vol.377 (2), p.284

2021

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MicroRNA-140 is elevated and mitofusin-1 is downregulated in the right ventricle of the Sugen5416/hypoxia/normoxia model of pulmonary arterial hypertension

American journal of physiology. Heart and circulatory physiology, 2016-09, Vol.311 (3), p.H689-H698

2016

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Increased Reactive Oxygen Species, Metabolic Maladaptation, and Autophagy Contribute to Pulmonary Arterial Hypertension–Induced Ventricular Hypertrophy and Diastolic Heart Failure

Hypertension (Dallas, Tex. 1979), 2014-12, Vol.64 (6), p.1266-1274

2014

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Superoxide production by NAD(P)H oxidase and mitochondria is increased in genetically obese and hyperglycemic rat heart and aorta before the development of cardiac dysfunction. The role of glucose-6-phosphate dehydrogenase-derived NADPH

American journal of physiology. Heart and circulatory physiology, 2009-07, Vol.297 (1), p.H153-H162

2009

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Hypoxic activation of glucose-6-phosphate dehydrogenase controls the expression of genes involved in the pathogenesis of pulmonary hypertension through the regulation of DNA methylation

American journal of physiology. Lung cellular and molecular physiology, 2020-04, Vol.318 (4), p.L773-L786

2020

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Impact of glucose-6-phosphate dehydrogenase deficiency on the pathophysiology of cardiovascular disease

American journal of physiology. Heart and circulatory physiology, 2013-02, Vol.304 (4), p.H491-H500

2013

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Vascular smooth muscle cell contractile protein expression is increased through protein kinase G-dependent and -independent pathways by glucose-6-phosphate dehydrogenase inhibition and deficiency

American journal of physiology. Heart and circulatory physiology, 2016-10, Vol.311 (4), p.H904-H912

2016

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Poly(ethylene glycol)-cholesterol inhibits L-type Ca2+ channel currents and augments voltage-dependent inactivation in A7r5 cells

PloS one, 2014-09, Vol.9 (9), p.e107049-e107049

2014

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Oxidant and redox signaling in vascular oxygen sensing mechanisms: basic concepts, current controversies, and potential importance of cytosolic NADPH

American journal of physiology. Lung cellular and molecular physiology, 2005-08, Vol.289 (2), p.L159-L173

2005

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Contractile protein expression is upregulated by reactive oxygen species in aorta of Goto-Kakizaki rat

American journal of physiology. Heart and circulatory physiology, 2014-01, Vol.306 (2), p.H214-H224

2014

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Oxidant and redox signaling in vascular oxygen sensing: implications for systemic and pulmonary hypertension

Antioxidants & redox signaling, 2008-06, Vol.10 (6), p.1137

2008

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Differences in the expression of DNA methyltransferases and demethylases in leukocytes and the severity of pulmonary arterial hypertension between ethnic groups

Physiological reports, 2022-05, Vol.10 (10), p.e15282-n/a

2022

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Glucose-6-phosphate dehydrogenase and NADPH redox regulates cardiac myocyte L-type calcium channel activity and myocardial contractile function

PloS one, 2012-10, Vol.7 (10), p.e45365

2012

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Survivin regulates intracellular stiffness and extracellular matrix production in vascular smooth muscle cells

APL bioengineering, 2023-12, Vol.7 (4), p.046104-046104

2023

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Glucose-6-phosphate dehydrogenase increases Ca 2+ currents by interacting with Ca v 1.2 and reducing intrinsic inactivation of the L-type calcium channel

American journal of physiology. Heart and circulatory physiology, 2020-07, Vol.319 (1), p.H144-H158

2020

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Glucose-6-phosphate dehydrogenase increases Ca2+ currents by interacting with Cav1.2 and reducing intrinsic inactivation of the L-type calcium channel

American journal of physiology. Heart and circulatory physiology, 2020-07, Vol.319 (1), p.H144-H158

2020

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Upregulation of Glucose-6-Phosphate Dehydrogenase and NAD(P)H Oxidase Activity Increases Oxidative Stress in Failing Human Heart

Journal of cardiac failure, 2007-08, Vol.13 (6), p.497-506

2007

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Deletion Of G‐Protein‐Coupled Receptor 75 Prevents Hypoxia‐Induced Pulmonary Vasoconstriction and Hypertension

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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G6PD activity contributes to the regulation of histone acetylation and gene expression in smooth muscle cells and to the pathogenesis of vascular diseases

American journal of physiology. Heart and circulatory physiology, 2021-03, Vol.320 (3), p.H999-H1016

2021

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The sources of oxidative stress in the vessel wall

Kidney international, 2005-05, Vol.67 (5), p.1659-1661

2005

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G6PD Orchestrates Genome-Wide DNA Methylation and Gene Expression in the Vascular Wall

G6PD is a critical enabler of hypoxia-induced accumulation of macrophages and platelets in mice lungs and contributor to lung inflammation

Glucose-6-phosphate dehydrogenase and MEG3 controls hypoxia-induced expression of serum response factor (SRF) and SRF-dependent genes in pulmonary smooth muscle cell

Hypoxia-induced glucose-6-phosphate dehydrogenase overexpression and -activation in pulmonary artery smooth muscle cells: implication in pulmonary hypertension

Hallmarks of Pulmonary Hypertension: Mesenchymal and Inflammatory Cell Metabolic Reprogramming

Inhibition of Glucose-6-Phosphate Dehydrogenase Activity Attenuates Right Ventricle Pressure and Hypertrophy Elicited by VEGFR Inhibitor + Hypoxia

MicroRNA-140 is elevated and mitofusin-1 is downregulated in the right ventricle of the Sugen5416/hypoxia/normoxia model of pulmonary arterial hypertension

Increased Reactive Oxygen Species, Metabolic Maladaptation, and Autophagy Contribute to Pulmonary Arterial Hypertension–Induced Ventricular Hypertrophy and Diastolic Heart Failure

Superoxide production by NAD(P)H oxidase and mitochondria is increased in genetically obese and hyperglycemic rat heart and aorta before the development of cardiac dysfunction. The role of glucose-6-phosphate dehydrogenase-derived NADPH

Hypoxic activation of glucose-6-phosphate dehydrogenase controls the expression of genes involved in the pathogenesis of pulmonary hypertension through the regulation of DNA methylation

Impact of glucose-6-phosphate dehydrogenase deficiency on the pathophysiology of cardiovascular disease

Vascular smooth muscle cell contractile protein expression is increased through protein kinase G-dependent and -independent pathways by glucose-6-phosphate dehydrogenase inhibition and deficiency

Poly(ethylene glycol)-cholesterol inhibits L-type Ca2+ channel currents and augments voltage-dependent inactivation in A7r5 cells

Oxidant and redox signaling in vascular oxygen sensing mechanisms: basic concepts, current controversies, and potential importance of cytosolic NADPH

Contractile protein expression is upregulated by reactive oxygen species in aorta of Goto-Kakizaki rat

Oxidant and redox signaling in vascular oxygen sensing: implications for systemic and pulmonary hypertension

Differences in the expression of DNA methyltransferases and demethylases in leukocytes and the severity of pulmonary arterial hypertension between ethnic groups

Glucose-6-phosphate dehydrogenase and NADPH redox regulates cardiac myocyte L-type calcium channel activity and myocardial contractile function

Survivin regulates intracellular stiffness and extracellular matrix production in vascular smooth muscle cells

Glucose-6-phosphate dehydrogenase increases Ca 2+ currents by interacting with Ca v 1.2 and reducing intrinsic inactivation of the L-type calcium channel

Glucose-6-phosphate dehydrogenase increases Ca2+ currents by interacting with Cav1.2 and reducing intrinsic inactivation of the L-type calcium channel

Upregulation of Glucose-6-Phosphate Dehydrogenase and NAD(P)H Oxidase Activity Increases Oxidative Stress in Failing Human Heart

Deletion Of G‐Protein‐Coupled Receptor 75 Prevents Hypoxia‐Induced Pulmonary Vasoconstriction and Hypertension

G6PD activity contributes to the regulation of histone acetylation and gene expression in smooth muscle cells and to the pathogenesis of vascular diseases

The sources of oxidative stress in the vessel wall

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