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Metabolism and function of hepatitis B virus cccDNA: Implications for the development of cccDNA-targeting antiviral therapeutics

Antiviral research, 2015-10, Vol.122, p.91-100

2015

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DNA Polymerase alpha is essential for intracellular amplification of hepatitis B virus covalently closed circular DNA

PLoS pathogens, 2019-04, Vol.15 (4), p.e1007742-e1007742

2019

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Present and future therapies of hepatitis B: From discovery to cure

Hepatology (Baltimore, Md.), 2015-12, Vol.62 (6), p.1893-1908

2015

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HBV core protein allosteric modulators differentially alter cccDNA biosynthesis from de novo infection and intracellular amplification pathways

PLoS pathogens, 2017-09, Vol.13 (9), p.e1006658-e1006658

2017

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Have the Starting Lineup of Five for Hepatitis B Virus Covalently Closed Circular DNA Synthesis Been Identified?

Hepatology (Baltimore, Md.), 2020-09, Vol.72 (3), p.1142-1144

2020

Volltextzugriff (PDF)

Inhibition of hepatitis B virus replication by the host zinc finger antiviral protein

PLoS pathogens, 2013-07, Vol.9 (7), p.e1003494-e1003494

2013

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Broad and Differential Animal Angiotensin-Converting Enzyme 2 Receptor Usage by SARS-CoV-2

Journal of virology, 2020-08, Vol.94 (18)

2020

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Cellular DNA Topoisomerases Are Required for the Synthesis of Hepatitis B Virus Covalently Closed Circular DNA

Journal of virology, 2019-06, Vol.93 (11)

2019

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Protein phosphatase 1 catalyzes HBV core protein dephosphorylation and is co-packaged with viral pregenomic RNA into nucleocapsids

PLoS pathogens, 2020-07, Vol.16 (7), p.e1008669-e1008669

2020

Volltextzugriff (PDF)

Targeting the multifunctional HBV core protein as a potential cure for chronic hepatitis B

Antiviral research, 2020-10, Vol.182, p.104917-104917, Article 104917

2020

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Interferon induction of IFITM proteins promotes infection by human coronavirus OC43

Proceedings of the National Academy of Sciences - PNAS, 2014-05, Vol.111 (18), p.6756-6761

2014

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IFITM Genes, Variants, and Their Roles in the Control and Pathogenesis of Viral Infections

Frontiers in microbiology, 2019-01, Vol.9, p.3228-3228

2019

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Hepatitis B Virus Virions Produced Under Nucleos(t)ide Analogue Treatment Are Mainly Not Infectious Because of Irreversible DNA Chain Termination

Hepatology (Baltimore, Md.), 2020-02, Vol.71 (2), p.463-476

2020

Volltextzugriff (PDF)

DNA Polymerase κ Is a Key Cellular Factor for the Formation of Covalently Closed Circular DNA of Hepatitis B Virus

PLoS pathogens, 2016-10, Vol.12 (10), p.e1005893-e1005893

2016

Volltextzugriff (PDF)

Identification of Residues Controlling Restriction versus Enhancing Activities of IFITM Proteins on Entry of Human Coronaviruses

Journal of virology, 2018-03, Vol.92 (6)

2018

Volltextzugriff (PDF)

Mechanism of interferon alpha therapy for chronic hepatitis B and potential approaches to improve its therapeutic efficacy

Antiviral research, 2024-01, Vol.221, p.105782-105782, Article 105782

2024

Volltextzugriff (PDF)

The Interferon-Inducible Protein Tetherin Inhibits Hepatitis B Virus Virion Secretion

Journal of virology, 2015-09, Vol.89 (18), p.9200-9212

2015

Volltextzugriff (PDF)

A research agenda for curing chronic hepatitis B virus infection

Hepatology (Baltimore, Md.), 2018-03, Vol.67 (3), p.1127-1131

2018

Volltextzugriff (PDF)

Hepatitis B Virus Core Protein Dephosphorylation Occurs during Pregenomic RNA Encapsidation

Journal of virology, 2018-07, Vol.92 (13)

2018

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Amino acid residues at core protein dimer-dimer interface modulate multiple steps of hepatitis B virus replication and HBeAg biogenesis

PLoS pathogens, 2021-11, Vol.17 (11), p.e1010057-e1010057

2021

Volltextzugriff (PDF)

Do hepatitis B virus surface antigens have any role in viral carcinogenesis?

Hepatology (Baltimore, Md.), 2018-09, Vol.68 (3), p.801-803

2018

Volltextzugriff (PDF)

Antiviral therapies targeting host ER alpha-glucosidases: Current status and future directions

Antiviral research, 2013-09, Vol.99 (3), p.251-260

2013

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Sulfamoylbenzamide Derivatives Inhibit the Assembly of Hepatitis B Virus Nucleocapsids

Journal of Virology, 2013-06, Vol.87 (12), p.6931-6942

2013

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Identification of Disubstituted Sulfonamide Compounds as Specific Inhibitors of Hepatitis B Virus Covalently Closed Circular DNA Formation

Antimicrobial Agents and Chemotherapy, 2012-08, Vol.56 (8), p.4277-4288

2012

Volltextzugriff (PDF)

Alpha-interferon suppresses hepadnavirus transcription by altering epigenetic modification of cccDNA minichromosomes

PLoS pathogens, 2013-09, Vol.9 (9), p.e1003613

2013

Volltextzugriff (PDF)

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Metabolism and function of hepatitis B virus cccDNA: Implications for the development of cccDNA-targeting antiviral therapeutics

DNA Polymerase alpha is essential for intracellular amplification of hepatitis B virus covalently closed circular DNA

Present and future therapies of hepatitis B: From discovery to cure

HBV core protein allosteric modulators differentially alter cccDNA biosynthesis from de novo infection and intracellular amplification pathways

Have the Starting Lineup of Five for Hepatitis B Virus Covalently Closed Circular DNA Synthesis Been Identified?

Inhibition of hepatitis B virus replication by the host zinc finger antiviral protein

Broad and Differential Animal Angiotensin-Converting Enzyme 2 Receptor Usage by SARS-CoV-2

Cellular DNA Topoisomerases Are Required for the Synthesis of Hepatitis B Virus Covalently Closed Circular DNA

Protein phosphatase 1 catalyzes HBV core protein dephosphorylation and is co-packaged with viral pregenomic RNA into nucleocapsids

Targeting the multifunctional HBV core protein as a potential cure for chronic hepatitis B

Interferon induction of IFITM proteins promotes infection by human coronavirus OC43

IFITM Genes, Variants, and Their Roles in the Control and Pathogenesis of Viral Infections

Hepatitis B Virus Virions Produced Under Nucleos(t)ide Analogue Treatment Are Mainly Not Infectious Because of Irreversible DNA Chain Termination

DNA Polymerase κ Is a Key Cellular Factor for the Formation of Covalently Closed Circular DNA of Hepatitis B Virus

Identification of Residues Controlling Restriction versus Enhancing Activities of IFITM Proteins on Entry of Human Coronaviruses

Mechanism of interferon alpha therapy for chronic hepatitis B and potential approaches to improve its therapeutic efficacy

The Interferon-Inducible Protein Tetherin Inhibits Hepatitis B Virus Virion Secretion

A research agenda for curing chronic hepatitis B virus infection

Hepatitis B Virus Core Protein Dephosphorylation Occurs during Pregenomic RNA Encapsidation

Amino acid residues at core protein dimer-dimer interface modulate multiple steps of hepatitis B virus replication and HBeAg biogenesis

Do hepatitis B virus surface antigens have any role in viral carcinogenesis?

Antiviral therapies targeting host ER alpha-glucosidases: Current status and future directions

Sulfamoylbenzamide Derivatives Inhibit the Assembly of Hepatitis B Virus Nucleocapsids

Identification of Disubstituted Sulfonamide Compounds as Specific Inhibitors of Hepatitis B Virus Covalently Closed Circular DNA Formation

Alpha-interferon suppresses hepadnavirus transcription by altering epigenetic modification of cccDNA minichromosomes

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