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PRRT2 Is a Key Component of the Ca2+-Dependent Neurotransmitter Release Machinery

Cell reports (Cambridge), 2016-04, Vol.15 (1), p.117-131

2016

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Nonsense-mediated mRNA decay and loss-of-function of the protein underlie the X-linked epilepsy associated with the W356× mutation in synapsin I

PloS one, 2013-06, Vol.8 (6), p.e67724-e67724

2013

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APache Is an AP2-Interacting Protein Involved in Synaptic Vesicle Trafficking and Neuronal Development

Cell reports (Cambridge), 2017-12, Vol.21 (12), p.3596-3611

2017

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How Do Synaptic Vesicles "Know" Which Pool They Belong to?

The Journal of neuroscience, 2017-03, Vol.37 (9), p.2276-2278

Guarnieri, Fabrizia C

2017

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Apache is a neuronal player in autophagy required for retrograde axonal transport of autophagosomes

Cellular and molecular life sciences : CMLS, 2024-12, Vol.81 (1), p.416, Article 416

2024

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PRRT2 Is a Key Component of the Ca(2+)-Dependent Neurotransmitter Release Machinery

Cell reports (Cambridge), 2016-04, Vol.15 (1), p.117-131

2016

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A novel SYN1 missense mutation in non-syndromic X-linked intellectual disability affects synaptic vesicle life cycle, clustering and mobility

Human molecular genetics, 2017-12, Vol.26 (23), p.4699-4714

2017

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eEF2K/eEF2 Pathway Controls the Excitation/Inhibition Balance and Susceptibility to Epileptic Seizures

Cerebral cortex (New York, N.Y. 1991), 2017-03, Vol.27 (3), p.2226-2248

2017

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Synapsins contribute to the dynamic spatial organization of synaptic vesicles in an activity-dependent manner

The Journal of neuroscience, 2012-08, Vol.32 (35), p.12214-12227

2012

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Nonsense-Mediated mRNA Decay and Loss-of-Function of the Protein Underlie the X-Linked Epilepsy Associated with the W356x Mutation in Synapsin I

PloS one, 2013-06, Vol.8 (6), p.e67724

2013

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Synapsin I deletion reduces neuronal damage and ameliorates clinical progression of experimental autoimmune encephalomyelitis

Brain, behavior, and immunity, 2018-02, Vol.68, p.197-210

2018

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PRRT2 Is a Key Component of the Ca 2+ -Dependent Neurotransmitter Release Machinery

Cell reports (Cambridge), 2016-04, Vol.15 (1), p.117-131

2016

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PRRT2 Is a Key Component of the Ca super(2+)-Dependent Neurotransmitter Release Machinery

Cell reports (Cambridge), 2016-04, Vol.15 (1), p.117-131

2016

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Nonsense-Mediated mRNA Decay and Loss-of-Function of the Protein Underlie the X-Linked Epilepsy Associated with the W356 Mutation in Synapsin I. e67724

PloS one, 2013-06, Vol.8 (6)

2013

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Synapsins and Synaptic Vesicle Storage

Presynaptic Terminals, 2015, p.295-326

2015

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PRRT2 Is a Key Component of the Ca2+-Dependent Neurotransmitter Release Machinery

Nonsense-mediated mRNA decay and loss-of-function of the protein underlie the X-linked epilepsy associated with the W356× mutation in synapsin I

APache Is an AP2-Interacting Protein Involved in Synaptic Vesicle Trafficking and Neuronal Development

How Do Synaptic Vesicles "Know" Which Pool They Belong to?

Apache is a neuronal player in autophagy required for retrograde axonal transport of autophagosomes

PRRT2 Is a Key Component of the Ca(2+)-Dependent Neurotransmitter Release Machinery

A novel SYN1 missense mutation in non-syndromic X-linked intellectual disability affects synaptic vesicle life cycle, clustering and mobility

eEF2K/eEF2 Pathway Controls the Excitation/Inhibition Balance and Susceptibility to Epileptic Seizures

Synapsins contribute to the dynamic spatial organization of synaptic vesicles in an activity-dependent manner

Nonsense-Mediated mRNA Decay and Loss-of-Function of the Protein Underlie the X-Linked Epilepsy Associated with the W356x Mutation in Synapsin I

Synapsin I deletion reduces neuronal damage and ameliorates clinical progression of experimental autoimmune encephalomyelitis

PRRT2 Is a Key Component of the Ca 2+ -Dependent Neurotransmitter Release Machinery

PRRT2 Is a Key Component of the Ca super(2+)-Dependent Neurotransmitter Release Machinery

Nonsense-Mediated mRNA Decay and Loss-of-Function of the Protein Underlie the X-Linked Epilepsy Associated with the W356 Mutation in Synapsin I. e67724

Synapsins and Synaptic Vesicle Storage

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