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Connexin hemichannels mediate glutathione transport and protect lens fiber cells from oxidative stress

Journal of cell science, 2018-03, Vol.131 (6), p.jcs212506-jcs212506

2018

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Connexin 43 hemichannels regulate mitochondrial ATP generation, mobilization, and mitochondrial homeostasis against oxidative stress

eLife, 2022-11, Vol.11

2022

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Connexin hemichannels with prostaglandin release in anabolic function of bone to mechanical loading

eLife, 2022-02, Vol.11

2022

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Mechanotransduction via the coordinated actions of integrins, PI3K signaling and Connexin hemichannels

Bone research, 2021-02, Vol.9 (1), p.8-8

2021

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Regulation of Connexin Gap Junctions and Hemichannels by Calcium and Calcium Binding Protein Calmodulin

International journal of molecular sciences, 2020-11, Vol.21 (21), p.8194

2020

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Coupling Effect of Water and Proteoglycans on the In Situ Toughness of Bone

Journal of bone and mineral research, 2016-05, Vol.31 (5), p.1026-1029

2016

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Mechanical stress-activated integrin α5β1 induces opening of connexin 43 hemichannels

Proceedings of the National Academy of Sciences - PNAS, 2012-02, Vol.109 (9), p.3359-3364

2012

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Direct Regulation of Osteocytic Connexin 43 Hemichannels through AKT Kinase Activated by Mechanical Stimulation

The Journal of biological chemistry, 2014-04, Vol.289 (15), p.10582-10591

2014

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Connexin hemichannels regulate redox potential via metabolite exchange and protect lens against cellular oxidative damage

Redox biology, 2021-10, Vol.46, p.102102, Article 102102

2021

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Connexin 43 Channels Are Essential for Normal Bone Structure and Osteocyte Viability

Journal of bone and mineral research, 2015-03, Vol.30 (3), p.436-448

2015

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Connexin 43 Hemichannels Regulate Osteoblast to Osteocyte Differentiation

Frontiers in cell and developmental biology, 2022-05, Vol.10, p.892229-892229

2022

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Development of a potent embryonic chick lens model for studying congenital cataracts in vivo

Communications biology, 2021-03, Vol.4 (1), p.325-325, Article 325

2021

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Mechanical strain opens connexin 43 hemichannels in osteocytes: a novel mechanism for the release of prostaglandin

Molecular biology of the cell, 2005-07, Vol.16 (7), p.3100-3106

2005

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Mechanosensitive piezo1 calcium channel activates connexin 43 hemichannels through PI3K signaling pathway in bone

Cell & bioscience, 2022-12, Vol.12 (1), p.191-191, Article 191

2022

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Gap junction- and hemichannel-independent actions of connexins

Biochimica et biophysica acta, 2005-06, Vol.1711 (2), p.208-214

2005

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Beyond the Channels: Adhesion Functions of Aquaporin 0 and Connexin 50 in Lens Development

Frontiers in cell and developmental biology, 2022-04, Vol.10, p.866980-866980

2022

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Adaptation of Connexin 43-Hemichannel Prostaglandin Release to Mechanical Loading

The Journal of biological chemistry, 2008-09, Vol.283 (39), p.26374-26382

2008

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Connexin Gap Junctions and Hemichannels in Modulating Lens Redox Homeostasis and Oxidative Stress in Cataractogenesis

Antioxidants, 2021-08, Vol.10 (9), p.1374

2021

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Connexin 50 Functions as an Adhesive Molecule and Promotes Lens Cell Differentiation

Scientific reports, 2017-07, Vol.7 (1), p.5298-12, Article 5298

2017

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Connexin 43 hemichannels protect bone loss during estrogen deficiency

Bone research, 2019-04, Vol.7 (1), p.11-11

2019

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Protein kinase A activation alleviates cataract formation via increased gap junction intercellular communication

iScience, 2023-03, Vol.26 (3), p.106114-106114, Article 106114

2023

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ATP Inhibits Breast Cancer Migration and Bone Metastasis through Down-Regulation of CXCR4 and Purinergic Receptor P2Y11

Cancers, 2021-08, Vol.13 (17), p.4293

2021

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Protocol for altering connexin hemichannel function in primary chicken lens fiber cells using high-titer retroviral RCAS(A) infection

STAR protocols, 2023-12, Vol.4 (4), p.102564-102564, Article 102564

2023

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Cataract-causing mutation of human connexin 46 impairs gap junction, but increases hemichannel function and cell death

PloS one, 2013-09, Vol.8 (9), p.e74732-e74732

2013

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Data on Connexin 43 hemichannels regulation of cellular redox in lens

Data in brief, 2021-12, Vol.39, p.107572-107572, Article 107572

2021

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Connexin hemichannels mediate glutathione transport and protect lens fiber cells from oxidative stress

Connexin 43 hemichannels regulate mitochondrial ATP generation, mobilization, and mitochondrial homeostasis against oxidative stress

Connexin hemichannels with prostaglandin release in anabolic function of bone to mechanical loading

Mechanotransduction via the coordinated actions of integrins, PI3K signaling and Connexin hemichannels

Regulation of Connexin Gap Junctions and Hemichannels by Calcium and Calcium Binding Protein Calmodulin

Coupling Effect of Water and Proteoglycans on the In Situ Toughness of Bone

Mechanical stress-activated integrin α5β1 induces opening of connexin 43 hemichannels

Direct Regulation of Osteocytic Connexin 43 Hemichannels through AKT Kinase Activated by Mechanical Stimulation

Connexin hemichannels regulate redox potential via metabolite exchange and protect lens against cellular oxidative damage

Connexin 43 Channels Are Essential for Normal Bone Structure and Osteocyte Viability

Connexin 43 Hemichannels Regulate Osteoblast to Osteocyte Differentiation

Development of a potent embryonic chick lens model for studying congenital cataracts in vivo

Mechanical strain opens connexin 43 hemichannels in osteocytes: a novel mechanism for the release of prostaglandin

Mechanosensitive piezo1 calcium channel activates connexin 43 hemichannels through PI3K signaling pathway in bone

Gap junction- and hemichannel-independent actions of connexins

Beyond the Channels: Adhesion Functions of Aquaporin 0 and Connexin 50 in Lens Development

Adaptation of Connexin 43-Hemichannel Prostaglandin Release to Mechanical Loading

Connexin Gap Junctions and Hemichannels in Modulating Lens Redox Homeostasis and Oxidative Stress in Cataractogenesis

Connexin 50 Functions as an Adhesive Molecule and Promotes Lens Cell Differentiation

Connexin 43 hemichannels protect bone loss during estrogen deficiency

Protein kinase A activation alleviates cataract formation via increased gap junction intercellular communication

ATP Inhibits Breast Cancer Migration and Bone Metastasis through Down-Regulation of CXCR4 and Purinergic Receptor P2Y11

Protocol for altering connexin hemichannel function in primary chicken lens fiber cells using high-titer retroviral RCAS(A) infection

Cataract-causing mutation of human connexin 46 impairs gap junction, but increases hemichannel function and cell death

Data on Connexin 43 hemichannels regulation of cellular redox in lens

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