UB Paderborn / Katalog / Suche / in CDI

1 – 25 von 44

Sortiert nach

The loss of Mcl-1 expression in human polymorphonuclear leukocytes promotes apoptosis

Journal of leukocyte biology, 2000-07, Vol.68 (1), p.158-166

2000

Volltextzugriff (PDF)

Sepsis-induced SOCS-3 expression is immunologically restricted to phagocytes

Journal of leukocyte biology, 2003-11, Vol.74 (5), p.916-922

2003

Volltextzugriff (PDF)

α-Defensin 1 (Human Neutrophil Protein 1) as an Antichemotactic Agent for Human Polymorphonuclear Leukocytes

Antimicrobial Agents and Chemotherapy, 2003-08, Vol.47 (8), p.2666-2668

2003

Volltextzugriff (PDF)

Suppression of PMN apoptosis by hypoxia is dependent on Mcl-1 and MAPK activity

Surgery, 2000-08, Vol.128 (2), p.171-177

2000

Volltextzugriff (PDF)

CXCR2 inhibition suppresses hemorrhage-induced priming for acute lung injury in mice

Journal of leukocyte biology, 2004-07, Vol.76 (1), p.58-64

2004

Volltextzugriff (PDF)

Regulation of IL-8RA (CXCR1) expression in polymorphonuclear leukocytes by hypoxia/reoxygenation

Journal of leukocyte biology, 1999-02, Vol.65 (2), p.171-178

1999

Volltextzugriff (PDF)

IL-1β stimulation induces paracrine regulation of PMN function and apoptosis

Shock (Augusta, Ga.), 1999-11, Vol.12 (5), p.373-381

1999

Volltextzugriff (PDF)

Shock-Induced Neutrophil Mediated Priming for Acute Lung Injury in Mice: Divergent Effects of TLR-4 and TLR-4/FasL Deficiency

The American journal of pathology, 2002-12, Vol.161 (6), p.2283-2294

2002

Volltextzugriff (PDF)

Human thymic epithelial cells produce TGF-beta 3 and express TGF-beta receptors

International immunology, 1995-10, Vol.7 (10), p.1681-1690

1995

Volltextzugriff (PDF)

Pathological aspects of apoptosis in severe sepsis and shock?

The international journal of biochemistry & cell biology, 2003, Vol.35 (1), p.7-15

2003

Volltextzugriff (PDF)

MECHANISMS OF INDUCTION OF SUPPRESSOR OF CYTOKINE SIGNALING (SOCS) UP-REGULATION FOLLOWING TRAUMA AND SEPSIS: 173

Shock (Augusta, Ga.), 2004-03, Vol.21 (Supplement), p.44

2004

Volltextzugriff (PDF)

Expression of cardiac muscle markers in rat myocyte cell lines

Molecular and cellular biochemistry, 1996-04, Vol.157 (1-2), p.87-91

1996

Volltextzugriff (PDF)

Mechanisms of immune resolution

Critical care medicine, 2003-08, Vol.31 (8 Suppl), p.S558-S571

2003

Volltextzugriff (PDF)

Differential effects of macrophage inflammatory chemokine-2 and keratinocyte-derived chemokine on hemorrhage-induced neutrophil priming for lung inflammation: Assessment by adoptive cells transfer in mice

Shock (Augusta, Ga.), 2003-04, Vol.19 (4), p.358-365

2003

Volltextzugriff (PDF)

FACTORS RESPONSIBLE FOR SUPPRESSOR OF CYTOKINE SIGNALING (SOCS) -3 UPREGULATION IN POLYMICROBIAL SEPSIS IS SITE-SPECIFIC: 4

Shock (Augusta, Ga.), 2003-06, Vol.19 (Supplement), p.2

2003

Volltextzugriff (PDF)

Myd88 DEFICIENCY ABROGATES ASPECTS OF SPLENIC LYMPHOID IMMUNE DYSFUNCTION SEEN IN RESPONSE TO TISSUE INJURY: 38

Shock (Augusta, Ga.), 2003-06, Vol.19 (Supplement), p.13

2003

Volltextzugriff (PDF)

Coordinate TGF-beta receptor gene expression during rat heart development

Cellular & molecular biology research, 1994, Vol.40 (2), p.93

1994

Volltextzugriff (PDF)

SUPPRESSOR OF CYTOKINE SIGNALING (SOCS) -3 EXPRESSION IS UPREGULATED IN POLYMICROBIAL SEPSIS: 24

Shock (Augusta, Ga.), 2002-06, Vol.17 (Supplement), p.8

2002

Volltextzugriff (PDF)

REQUIREMENT FOR PI3K AND MEK-I IN THE PARACRINE SUPPRESSION OF NEUTROPHIL (PMN) CHEMOTAXIS BY TNF-α-STIMULATED PMN: 222

Shock (Augusta, Ga.), 2001-06, Vol.15 (Supplement), p.74-75

2001

Volltextzugriff (PDF)

DIVERGENT EFFECTS OF SEPSIS ON DENDRITIC CELLS: 75

Shock (Augusta, Ga.), 2004-03, Vol.21 (Supplement), p.19

2004

Volltextzugriff (PDF)

Divergent roles of murine neutrophil chemokines in hemorrhage induced priming for acute lung injury

Cytokine (Philadelphia, Pa.), 2005-08, Vol.31 (3), p.169-179

2005

Volltextzugriff (PDF)

PMN DEPLETION ATTENUATES LUNG INJURY RESULTANT FROM COMBINED INSULTS OF HEMORRHAGE FOLLOWED BY SEPSIS INDEPENDENT OF ICAM-1 EXPRESSION: 197

Shock (Augusta, Ga.), 2003-06, Vol.19 (Supplement), p.67

2003

Volltextzugriff (PDF)

DIFFERENTIAL EFFECTS OF MIP-2 AND KC ON HEMORRHAGE INDUCED NEUTROPHIL PRIMING FOR LUNG INFLAMMATION: 62

Shock (Augusta, Ga.), 2002-06, Vol.17 (Supplement), p.21

2002

Volltextzugriff (PDF)

NEUTROPHIL SEQUESTRATION IN LUNG IS A TRANSIENT EVENT DURING POLYMICROBIAL SEPSIS: 88

Shock (Augusta, Ga.), 2002-06, Vol.17 (Supplement), p.30

2002

Volltextzugriff (PDF)

IL-1beta stimulation induces paracrine regulation of PMN function and apoptosis

Shock (Augusta, Ga.), 1999-11, Vol.12 (5), p.373-381

1999

Volltextzugriff (PDF)

1 – 25 von 44

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(41)
Mit Volltextzugriff
(43)

Publikationsform

Artikel
(43)
Tagungsberichte
(1)

Sprache

Englisch
(44)
Japanisch
(2)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Zeitschrift / Serie

5 weitere anzeigen...

Weniger anzeigen...

Menü

Weitere Dienste

Einstellungen

The loss of Mcl-1 expression in human polymorphonuclear leukocytes promotes apoptosis

Sepsis-induced SOCS-3 expression is immunologically restricted to phagocytes

α-Defensin 1 (Human Neutrophil Protein 1) as an Antichemotactic Agent for Human Polymorphonuclear Leukocytes

Suppression of PMN apoptosis by hypoxia is dependent on Mcl-1 and MAPK activity

CXCR2 inhibition suppresses hemorrhage-induced priming for acute lung injury in mice

Regulation of IL-8RA (CXCR1) expression in polymorphonuclear leukocytes by hypoxia/reoxygenation

IL-1β stimulation induces paracrine regulation of PMN function and apoptosis

Shock-Induced Neutrophil Mediated Priming for Acute Lung Injury in Mice: Divergent Effects of TLR-4 and TLR-4/FasL Deficiency

Human thymic epithelial cells produce TGF-beta 3 and express TGF-beta receptors

Pathological aspects of apoptosis in severe sepsis and shock?

MECHANISMS OF INDUCTION OF SUPPRESSOR OF CYTOKINE SIGNALING (SOCS) UP-REGULATION FOLLOWING TRAUMA AND SEPSIS: 173

Expression of cardiac muscle markers in rat myocyte cell lines

Mechanisms of immune resolution

Differential effects of macrophage inflammatory chemokine-2 and keratinocyte-derived chemokine on hemorrhage-induced neutrophil priming for lung inflammation: Assessment by adoptive cells transfer in mice

FACTORS RESPONSIBLE FOR SUPPRESSOR OF CYTOKINE SIGNALING (SOCS) -3 UPREGULATION IN POLYMICROBIAL SEPSIS IS SITE-SPECIFIC: 4

Myd88 DEFICIENCY ABROGATES ASPECTS OF SPLENIC LYMPHOID IMMUNE DYSFUNCTION SEEN IN RESPONSE TO TISSUE INJURY: 38

Coordinate TGF-beta receptor gene expression during rat heart development

SUPPRESSOR OF CYTOKINE SIGNALING (SOCS) -3 EXPRESSION IS UPREGULATED IN POLYMICROBIAL SEPSIS: 24

REQUIREMENT FOR PI3K AND MEK-I IN THE PARACRINE SUPPRESSION OF NEUTROPHIL (PMN) CHEMOTAXIS BY TNF-α-STIMULATED PMN: 222

DIVERGENT EFFECTS OF SEPSIS ON DENDRITIC CELLS: 75

Divergent roles of murine neutrophil chemokines in hemorrhage induced priming for acute lung injury

PMN DEPLETION ATTENUATES LUNG INJURY RESULTANT FROM COMBINED INSULTS OF HEMORRHAGE FOLLOWED BY SEPSIS INDEPENDENT OF ICAM-1 EXPRESSION: 197

DIFFERENTIAL EFFECTS OF MIP-2 AND KC ON HEMORRHAGE INDUCED NEUTROPHIL PRIMING FOR LUNG INFLAMMATION: 62

NEUTROPHIL SEQUESTRATION IN LUNG IS A TRANSIENT EVENT DURING POLYMICROBIAL SEPSIS: 88

IL-1beta stimulation induces paracrine regulation of PMN function and apoptosis

Suchergebnisse filtern