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Pregnancy Enhances Sustained Ca²⁺ Bursts and Endothelial Nitric Oxide Synthase Activation in Ovine Uterine Artery Endothelial Cells Through Increased Connexin 43 Function

Biology of reproduction, 2010, Vol.82 (1), p.66-75

2010

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Vascular endothelial growth factor acts through novel, pregnancy-enhanced receptor signalling pathways to stimulate endothelial nitric oxide synthase activity in uterine artery endothelial cells

Biochemical journal, 2009-01, Vol.417 (2), p.501-511

2009

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Differential control of uterine artery endothelial monolayer integrity by TNF and VEGF is achieved through multiple mechanisms operating inside and outside the cell – Relevance to preeclampsia

Molecular and cellular endocrinology, 2021-08, Vol.534, p.111368-111368, Article 111368

2021

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Adenoviral transduction of EGFR into pregnancy-adapted uterine artery endothelial cells remaps growth factor induction of endothelial dysfunction

Molecular and cellular endocrinology, 2020-01, Vol.499, p.110590-110590, Article 110590

2020

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TNF-alpha inhibits pregnancy-adapted Ca2+ signaling in uterine artery endothelial cells

Molecular and cellular endocrinology, 2019-05, Vol.488, p.14-24

2019

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Phosphorylation of Ser-279/282 and Tyr-265 positions on Cx43 as possible mediators of VEGF-165 inhibition of pregnancy-adapted Ca2+ burst function in ovine uterine artery endothelial cells

Molecular and cellular endocrinology, 2015-09, Vol.412, p.73-84

2015

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Altered VEGF-stimulated Ca2+ signaling in part underlies pregnancy-adapted eNOS activity in UAEC

Journal of endocrinology, 2014-10, Vol.223 (1), p.1-11

2014

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The loss of sustained Ca(2+) signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy

American journal of physiology. Heart and circulatory physiology, 2013-10, Vol.305 (7), p.H969-H979

2013

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Pregnancy Enhances Sustained Ca2+ Bursts and Endothelial Nitric Oxide Synthase Activation in Ovine Uterine Artery Endothelial Cells Through Increased Connexin 43 Function1

Biology of reproduction, 2010-01, Vol.82 (1), p.66-75

2010

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Changes in Ca2+ Signaling and Nitric Oxide Output by Human Umbilical Vein Endothelium in Diabetic and Gestational Diabetic Pregnancies

Biology of reproduction, 2015-09, Vol.93 (3), p.60-60

2015

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Pregnancy programming and preeclampsia: identifying a human endothelial model to study pregnancy-adapted endothelial function and endothelial adaptive failure in preeclamptic subjects

Advances in experimental medicine and biology, 2014-01, Vol.814, p.27

2014

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Changes in Ca2+ Signaling and Nitric Oxide Output by Human Umbilical Vein Endothelium in Diabetic and Gestational Diabetic Pregnancies1

Biology of reproduction, 2015-09, Vol.93 (3)

2015

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The loss of sustained Ca2+ signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy

American journal of physiology. Heart and circulatory physiology, 2013-10, Vol.305 (7), p.H969-H979

2013

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Interaction of ethanol with retinol and retinoic acid in RAR β and GAP-43 expression

Neurotoxicology and teratology, 2000-11, Vol.22 (6), p.829-836

2000

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Effect of retinoic acid and ethanol on retinoic acid receptor β and glial fibrillary acidic protein mRNA expression in human astrocytoma cells

Neuroscience letters, 2000-11, Vol.294 (2), p.73-76

2000

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Prenatal ethanol consumption increases retinol and cellular retinol-binding protein expression in the rat fetal snout

Biology of the neonate, 2001-01, Vol.80 (2), p.152-157

2001

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TNF-alpha inhibits pregnancy-adapted Ca 2+ signaling in uterine artery endothelial cells

Molecular and cellular endocrinology, 2019-05, Vol.488, p.14

2019

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Retinoic acid and dexamethasone affect RAR-beta and surfactant protein C mRNA in the MLE lung cell line

American journal of physiology. Lung cellular and molecular physiology, 1998-01, Vol.274 (1), p.1-L7

1998

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The loss of sustained Ca^sup 2+^ signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy

American journal of physiology. Heart and circulatory physiology, 2013-10, Vol.305 (7), p.H969

2013

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The loss of sustained Ca 2+ signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy

American journal of physiology. Heart and circulatory physiology, 2013-10, Vol.305 (7), p.H969-H979

2013

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Effect of maternal/fetal vitamin A deficiency on fetal rat lung surfactant protein expression and the response to prenatal dexamethasone

Pediatric research, 1998-02, Vol.43 (2), p.178-183

1998

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Pregnancy-Enhanced Endothelial Nitric Oxide Synthase (eNOS) Activation in Uterine Artery Endothelial Cells Shows Altered Sensitivity to Ca2+, U0126, and Wortmannin But Not LY294002—Evidence that Pregnancy Adaptation of eNOS Activation Occurs at Multiple Levels of Cell Signaling

Endocrinology (Philadelphia), 2006-05, Vol.147 (5), p.2442-2457

2006

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VEGF acts through novel, pregnancy-enhanced receptor signaling pathways to stimulate eNOS activity in uterine artery endothelial cells

Biochemical journal, 2009-01, Vol.417 (2), p.501-511

2009

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The effect of maternal ethanol ingestion on fetal vitamin A in the rat

Pediatric research, 1990-09, Vol.28 (3), p.186-189

1990

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The Interaction of Ethanol and Vitamin A as a Potential Mechanism for the Pathogenesis of Fetal Alcohol Syndrome

Alcoholism, clinical and experimental research, 1998-10, Vol.22 (7), p.1544-1556

1998

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Pregnancy Enhances Sustained Ca²⁺ Bursts and Endothelial Nitric Oxide Synthase Activation in Ovine Uterine Artery Endothelial Cells Through Increased Connexin 43 Function

Vascular endothelial growth factor acts through novel, pregnancy-enhanced receptor signalling pathways to stimulate endothelial nitric oxide synthase activity in uterine artery endothelial cells

Differential control of uterine artery endothelial monolayer integrity by TNF and VEGF is achieved through multiple mechanisms operating inside and outside the cell – Relevance to preeclampsia

Adenoviral transduction of EGFR into pregnancy-adapted uterine artery endothelial cells remaps growth factor induction of endothelial dysfunction

TNF-alpha inhibits pregnancy-adapted Ca2+ signaling in uterine artery endothelial cells

Phosphorylation of Ser-279/282 and Tyr-265 positions on Cx43 as possible mediators of VEGF-165 inhibition of pregnancy-adapted Ca2+ burst function in ovine uterine artery endothelial cells

Altered VEGF-stimulated Ca2+ signaling in part underlies pregnancy-adapted eNOS activity in UAEC

The loss of sustained Ca(2+) signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy

Pregnancy Enhances Sustained Ca2+ Bursts and Endothelial Nitric Oxide Synthase Activation in Ovine Uterine Artery Endothelial Cells Through Increased Connexin 43 Function1

Changes in Ca2+ Signaling and Nitric Oxide Output by Human Umbilical Vein Endothelium in Diabetic and Gestational Diabetic Pregnancies

Pregnancy programming and preeclampsia: identifying a human endothelial model to study pregnancy-adapted endothelial function and endothelial adaptive failure in preeclamptic subjects

Changes in Ca2+ Signaling and Nitric Oxide Output by Human Umbilical Vein Endothelium in Diabetic and Gestational Diabetic Pregnancies1

The loss of sustained Ca2+ signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy

Interaction of ethanol with retinol and retinoic acid in RAR β and GAP-43 expression

Effect of retinoic acid and ethanol on retinoic acid receptor β and glial fibrillary acidic protein mRNA expression in human astrocytoma cells

Prenatal ethanol consumption increases retinol and cellular retinol-binding protein expression in the rat fetal snout

TNF-alpha inhibits pregnancy-adapted Ca 2+ signaling in uterine artery endothelial cells

Retinoic acid and dexamethasone affect RAR-beta and surfactant protein C mRNA in the MLE lung cell line

The loss of sustained Ca^sup 2+^ signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy

The loss of sustained Ca 2+ signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy

Effect of maternal/fetal vitamin A deficiency on fetal rat lung surfactant protein expression and the response to prenatal dexamethasone

Pregnancy-Enhanced Endothelial Nitric Oxide Synthase (eNOS) Activation in Uterine Artery Endothelial Cells Shows Altered Sensitivity to Ca2+, U0126, and Wortmannin But Not LY294002—Evidence that Pregnancy Adaptation of eNOS Activation Occurs at Multiple Levels of Cell Signaling

VEGF acts through novel, pregnancy-enhanced receptor signaling pathways to stimulate eNOS activity in uterine artery endothelial cells

The effect of maternal ethanol ingestion on fetal vitamin A in the rat

The Interaction of Ethanol and Vitamin A as a Potential Mechanism for the Pathogenesis of Fetal Alcohol Syndrome

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