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Oral uricase eliminates blood uric acid in the hyperuricemic pig model

PloS one, 2017-06, Vol.12 (6), p.e0179195-e0179195

2017

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Oral Treatment With an Engineered Uricase, ALLN-346, Reduces Hyperuricemia, and Uricosuria in Urate Oxidase-Deficient Mice

Frontiers in medicine, 2020-11, Vol.7, p.569215-569215

2020

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Energy Metabolism in Uncoupling Protein 3 Gene Knockout Mice

The Journal of biological chemistry, 2000-05, Vol.275 (21), p.16258-16266

2000

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UCP3: An Uncoupling Protein Homologue Expressed Preferentially and Abundantly in Skeletal Muscle and Brown Adipose Tissue

Biochemical and biophysical research communications, 1997-06, Vol.235 (1), p.79-82

1997

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Kidney Transplant Outcomes for Patients with Enteric Oxalosis: PO2533

Journal of the American Society of Nephrology, 2020-10, Vol.31 (10S), p.768-768

2020

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Safety and Efficacy of Reloxaliase in Enteric Hyperoxaluria (EH): An Aggregate Review of Completed Studies: PO0418

Journal of the American Society of Nephrology, 2020-10, Vol.31 (10S), p.177-177

2020

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Mo1321 - Development of an ex vivo Pancreas Using a Lipase-Bead Complex: Improving Bioavailability of Fat from Enteral Formulas and Nutritional Drinks in Patients with Exocrine Pancreatic Insufficiency Due to Chronic Pancreatitis or Pancreatectomy

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S678-S678

2017

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Absorption and Safety With Sustained Use of RELiZORB Evaluation (ASSURE) Study in Patients With Cystic Fibrosis Receiving Enteral Feeding

Journal of pediatric gastroenterology and nutrition, 2018-10, Vol.67 (4), p.527-532

2018

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Increased Fat Absorption From Enteral Formula Through an In‐line Digestive Cartridge in Patients With Cystic Fibrosis

Journal of pediatric gastroenterology and nutrition, 2017-07, Vol.65 (1), p.97-101

2017

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Pharmacodynamic (PD) Profiling of Reloxaliase in Patients with Severe Hyperoxaluria: TH-OR19

Journal of the American Society of Nephrology, 2020-10, Vol.31 (10S), p.6-6

2020

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Pilot study of reloxaliase in patients with severe enteric hyperoxaluria and hyperoxalemia

Nephrology, dialysis, transplantation, 2021-04, Vol.36 (5), p.945-948

2021

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Development of an ex vivo Pancreas Using a Lipase-Bead Complex: Improving Bioavailability of Fat from Enteral Formulas and Nutritional Drinks in Patients with Exocrine Pancreatic Insufficiency Due to Chronic Pancreatitis or Pancreatectomy

Gastroenterology (New York, N.Y. 1943), 2017-04, Vol.152 (5), p.S678-S678

2017

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Differential regulation of uncoupling protein gene homologues in multiple tissues of hibernating ground squirrels

American journal of physiology. Regulatory, integrative and comparative physiology, 1998-10, Vol.275 (4), p.1232-R1238

1998

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Regulation of skeletal muscle UCP-2 and UCP-3 gene expression by exercise and denervation

American journal of physiology: endocrinology and metabolism, 1999-01, Vol.276 (1), p.E217-E221

1999

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ALLN-177, oral enzyme therapy for hyperoxaluria

International urology and nephrology, 2019-04, Vol.51 (4), p.601-608

2019

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A Double-Blind, Placebo Controlled, Randomized Phase 1 Cross-Over Study with ALLN-177, an Orally Administered Oxalate Degrading Enzyme

American journal of nephrology, 2016-01, Vol.44 (2), p.150-158

2016

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PD31-11 ALLN-177 REDUCES HYPEROXALURIA IN A PORCINE MODEL OF SECONDARY HYPEROXALURIA (2°HO) INDUCED BY A HUMAN-LIKE OXALATE RICH DIET

The Journal of urology, 2016-04, Vol.195 (4), p.e721-e721

2016

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Uncoupling Protein-2 Negatively Regulates Insulin Secretion and Is a Major Link between Obesity, β Cell Dysfunction, and Type 2 Diabetes

Cell, 2001-06, Vol.105 (6), p.745-755

2001

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A piglet with surgically induced exocrine pancreatic insufficiency as an animal model of newborns to study fat digestion

British journal of nutrition, 2014-12, Vol.112 (12), p.2060-2067

2014

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Liprotamase, a Non-porcine Enzyme Therapy, Restores Growth, and Normalizes Lipid Absorption in Pigs with Exocrine Pancreatic Insufficiency (EPI): 136

The American journal of gastroenterology, 2010-10, Vol.105, p.S51

2010

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1025 Liprotamase, a Microbial Enzyme Therapy, Reveresed Impaired Growth in a Model of Cystic Fibrosis: Pigs With Total Exocrine Pancreatic Insufficiency (EPI Pigs)

Gastroenterology (New York, N.Y. 1943), 2010, Vol.138 (5), p.S-149-S-149

2010

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Beta-3 adrenergic receptor agonists cause an increase in gastrointestinal transit time in wild-type mice, but not in mice lacking the beta-3 adrenergic receptor

The Journal of pharmacology and experimental therapeutics, 1998-11, Vol.287 (2), p.720

1998

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The human uncoupling protein-3 gene. Genomic structure, chromosomal localization, and genetic basis for short and long form transcripts

The Journal of biological chemistry, 1997-10, Vol.272 (41), p.25433-25436

1997

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Hyperoxaluria Is Reduced and Nephrocalcinosis Prevented with an Oxalate-Degrading Enzyme in Mice with Hyperoxaluria

American journal of nephrology, 2009-01, Vol.29 (2), p.86-93

2009

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Expression of Human α2-Adrenergic Receptors in Adipose Tissue of β3-Adrenergic Receptor-deficient Mice Promotes Diet-induced Obesity

The Journal of biological chemistry, 2000-11, Vol.275 (44), p.34797-34802

2000

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Oral uricase eliminates blood uric acid in the hyperuricemic pig model

Oral Treatment With an Engineered Uricase, ALLN-346, Reduces Hyperuricemia, and Uricosuria in Urate Oxidase-Deficient Mice

Energy Metabolism in Uncoupling Protein 3 Gene Knockout Mice

UCP3: An Uncoupling Protein Homologue Expressed Preferentially and Abundantly in Skeletal Muscle and Brown Adipose Tissue

Kidney Transplant Outcomes for Patients with Enteric Oxalosis: PO2533

Safety and Efficacy of Reloxaliase in Enteric Hyperoxaluria (EH): An Aggregate Review of Completed Studies: PO0418

Mo1321 - Development of an ex vivo Pancreas Using a Lipase-Bead Complex: Improving Bioavailability of Fat from Enteral Formulas and Nutritional Drinks in Patients with Exocrine Pancreatic Insufficiency Due to Chronic Pancreatitis or Pancreatectomy

Absorption and Safety With Sustained Use of RELiZORB Evaluation (ASSURE) Study in Patients With Cystic Fibrosis Receiving Enteral Feeding

Increased Fat Absorption From Enteral Formula Through an In‐line Digestive Cartridge in Patients With Cystic Fibrosis

Pharmacodynamic (PD) Profiling of Reloxaliase in Patients with Severe Hyperoxaluria: TH-OR19

Pilot study of reloxaliase in patients with severe enteric hyperoxaluria and hyperoxalemia

Development of an ex vivo Pancreas Using a Lipase-Bead Complex: Improving Bioavailability of Fat from Enteral Formulas and Nutritional Drinks in Patients with Exocrine Pancreatic Insufficiency Due to Chronic Pancreatitis or Pancreatectomy

Differential regulation of uncoupling protein gene homologues in multiple tissues of hibernating ground squirrels

Regulation of skeletal muscle UCP-2 and UCP-3 gene expression by exercise and denervation

ALLN-177, oral enzyme therapy for hyperoxaluria

A Double-Blind, Placebo Controlled, Randomized Phase 1 Cross-Over Study with ALLN-177, an Orally Administered Oxalate Degrading Enzyme

PD31-11 ALLN-177 REDUCES HYPEROXALURIA IN A PORCINE MODEL OF SECONDARY HYPEROXALURIA (2°HO) INDUCED BY A HUMAN-LIKE OXALATE RICH DIET

Uncoupling Protein-2 Negatively Regulates Insulin Secretion and Is a Major Link between Obesity, β Cell Dysfunction, and Type 2 Diabetes

A piglet with surgically induced exocrine pancreatic insufficiency as an animal model of newborns to study fat digestion

Liprotamase, a Non-porcine Enzyme Therapy, Restores Growth, and Normalizes Lipid Absorption in Pigs with Exocrine Pancreatic Insufficiency (EPI): 136

1025 Liprotamase, a Microbial Enzyme Therapy, Reveresed Impaired Growth in a Model of Cystic Fibrosis: Pigs With Total Exocrine Pancreatic Insufficiency (EPI Pigs)

Beta-3 adrenergic receptor agonists cause an increase in gastrointestinal transit time in wild-type mice, but not in mice lacking the beta-3 adrenergic receptor

The human uncoupling protein-3 gene. Genomic structure, chromosomal localization, and genetic basis for short and long form transcripts

Hyperoxaluria Is Reduced and Nephrocalcinosis Prevented with an Oxalate-Degrading Enzyme in Mice with Hyperoxaluria

Expression of Human α2-Adrenergic Receptors in Adipose Tissue of β3-Adrenergic Receptor-deficient Mice Promotes Diet-induced Obesity

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