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Historical Perspective of the Characterization of Conotoxins Targeting Voltage-Gated Sodium Channels

Marine drugs, 2023-03, Vol.21 (4), p.209

Groome, James R

2023

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Roles for Countercharge in the Voltage Sensor Domain of Ion Channels

Frontiers in pharmacology, 2020-02, Vol.11, p.160-160

2020

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Nav1.4 mutations cause hypokalaemic periodic paralysis by disrupting IIIS4 movement during recovery

Brain (London, England : 1878), 2014-04, Vol.137 (Pt 4), p.998-1008

2014

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Gating defects in K1126I, a putative hypokalemic periodic paralysis mutation in hNav1.4

Biophysical journal, 2023-02, Vol.122 (3), p.103a-103a

Groome, James R.

2023

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S1-S3 counter charges in the voltage sensor module of a mammalian sodium channel regulate fast inactivation

The Journal of general physiology, 2013-05, Vol.141 (5), p.601-618

2013

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NaV1.4 DI-S4 periodic paralysis mutation R222W enhances inactivation and promotes leak current to attenuate action potentials and depolarize muscle fibers

Scientific reports, 2018-07, Vol.8 (1), p.1-13, Article 10372

2018

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Domain III S4 in closed-state fast inactivation: Insights from a periodic paralysis mutation

Channels (Austin, Tex.), 2014-09, Vol.8 (5), p.467-471

2014

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The voltage sensor module in sodium channels

Handbook of experimental pharmacology, 2014, Vol.221, p.7

Groome, James R

2014

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Correction: S1-S3 counter charges in the voltage sensor module of a mammalian sodium channel regulate fast inactivation

The Journal of general physiology, 2015-12, Vol.146 (6), p.541-546

2015

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Transient compartment-like syndrome and normokalaemic periodic paralysis due to a Cav1.1 mutation

Brain (London, England : 1878), 2013-12, Vol.136 (12), p.3775-3786

2013

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Ribbon α-Conotoxin KTM Exhibits Potent Inhibition of Nicotinic Acetylcholine Receptors

Marine drugs, 2019-11, Vol.17 (12), p.669

2019

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Transient compartment-like syndrome and normokalaemic periodic paralysis due to a Ca(v)1.1 mutation

Brain (London, England : 1878), 2013-12, Vol.136 (Pt 12), p.3775-3786

2013

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Temperature-sensitive defects in paramyotonia congenita mutants R1448C and T1313M

Muscle & nerve, 2004-09, Vol.30 (3), p.277-288

2004

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Open- and closed-state fast inactivation in sodium channels: Differential effects of a site-3 anemone toxin

Channels (Austin, Tex.), 2011-01, Vol.5 (1), p.65-78

2011

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Characterizing gating defects of SCN2A countercharge mutations in epilepsy syndromes

Biophysical journal, 2022-02, Vol.121 (3), p.96a-96a

2022

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A negative charge in transmembrane segment 1 of domain II of the cockroach sodium channel is critical for channel gating and action of pyrethroid insecticides

Toxicology and applied pharmacology, 2010-08, Vol.247 (1), p.53-59

2010

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Differential effects of homologous S4 mutations in human skeletal muscle sodium channels on deactivation gating from open and inactivated states

The Journal of physiology, 1999-05, Vol.516 (3), p.687-698

1999

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Role for Fast Inactivation in Domain I of Voltage Gated Sodium Channels

Biophysical journal, 2017-02, Vol.112 (3), p.103a-103a

2017

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Domain I Countercharges Limit Slow Inactivation in hNav1.4 Sodium Channels

Biophysical journal, 2019-02, Vol.116 (3), p.390a-390a

2019

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Voltage Sensor Domains and Closed-State Inactivation in Sodium Channels

Biophysical journal, 2015-01, Vol.108 (2), p.572a-572a

Groome, James R.

2015

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Charge Immobilization of Skeletal Muscle Na + Channels: Role of Residues in the Inactivation Linker

Biophysical journal, 2007-09, Vol.93 (5), p.1519-1533

2007

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Negative Countercharges and S4 Interaction in Domain IV of Nav1.4

Biophysical journal, 2014-01, Vol.106 (2), p.132a-132a

Groome, James R.

2014

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Cysteine Mutagenesis to Probe Site-Specific Interactions in the Voltage Sensor Module of hNaV1.4

Biophysical journal, 2020-02, Vol.118 (3), p.575a-575a

2020

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Markov and Eyring Modeling Predict Differential Contributions of Homologous Mutations in Hypokalemic Periodic Paralysis to Skeletal Muscle Fiber Excitability

Biophysical journal, 2021-02, Vol.120 (3), p.154a-154a

2021

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Differential effects of paramyotonia congenita mutations F1473S and F1705I on sodium channel gating

Channels (Austin, Tex.), 2008-01, Vol.2 (1), p.39-50

2008

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Historical Perspective of the Characterization of Conotoxins Targeting Voltage-Gated Sodium Channels

Roles for Countercharge in the Voltage Sensor Domain of Ion Channels

Nav1.4 mutations cause hypokalaemic periodic paralysis by disrupting IIIS4 movement during recovery

Gating defects in K1126I, a putative hypokalemic periodic paralysis mutation in hNav1.4

S1-S3 counter charges in the voltage sensor module of a mammalian sodium channel regulate fast inactivation

NaV1.4 DI-S4 periodic paralysis mutation R222W enhances inactivation and promotes leak current to attenuate action potentials and depolarize muscle fibers

Domain III S4 in closed-state fast inactivation: Insights from a periodic paralysis mutation

The voltage sensor module in sodium channels

Correction: S1-S3 counter charges in the voltage sensor module of a mammalian sodium channel regulate fast inactivation

Transient compartment-like syndrome and normokalaemic periodic paralysis due to a Cav1.1 mutation

Ribbon α-Conotoxin KTM Exhibits Potent Inhibition of Nicotinic Acetylcholine Receptors

Transient compartment-like syndrome and normokalaemic periodic paralysis due to a Ca(v)1.1 mutation

Temperature-sensitive defects in paramyotonia congenita mutants R1448C and T1313M

Open- and closed-state fast inactivation in sodium channels: Differential effects of a site-3 anemone toxin

Characterizing gating defects of SCN2A countercharge mutations in epilepsy syndromes

A negative charge in transmembrane segment 1 of domain II of the cockroach sodium channel is critical for channel gating and action of pyrethroid insecticides

Differential effects of homologous S4 mutations in human skeletal muscle sodium channels on deactivation gating from open and inactivated states

Role for Fast Inactivation in Domain I of Voltage Gated Sodium Channels

Domain I Countercharges Limit Slow Inactivation in hNav1.4 Sodium Channels

Voltage Sensor Domains and Closed-State Inactivation in Sodium Channels

Charge Immobilization of Skeletal Muscle Na + Channels: Role of Residues in the Inactivation Linker

Negative Countercharges and S4 Interaction in Domain IV of Nav1.4

Cysteine Mutagenesis to Probe Site-Specific Interactions in the Voltage Sensor Module of hNaV1.4

Markov and Eyring Modeling Predict Differential Contributions of Homologous Mutations in Hypokalemic Periodic Paralysis to Skeletal Muscle Fiber Excitability

Differential effects of paramyotonia congenita mutations F1473S and F1705I on sodium channel gating

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