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Control of pro-angiogenic cytokine mRNA half-life in cancer: the role of AU-rich elements and associated proteins

Journal of interferon & cytokine research, 2014-04, Vol.34 (4), p.242-254

2014

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A synonymous polymorphism of the Tristetraprolin (TTP) gene, an AU-rich mRNA-binding protein, affects translation efficiency and response to Herceptin treatment in breast cancer patients

Human molecular genetics, 2011-12, Vol.20 (23), p.4556-4568

2011

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Contribution of rare and common variants determine complex diseases—Hirschsprung disease as a model

Developmental biology, 2013-10, Vol.382 (1), p.320-329

2013

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Differential Contributions of Rare and Common, Coding and Noncoding Ret Mutations to Multifactorial Hirschsprung Disease Liability

American journal of human genetics, 2010-07, Vol.87 (1), p.60-74

2010

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Regulation of the mRNA half-life in breast cancer

World journal of clinical oncology, 2014-08, Vol.5 (3), p.323-334

2014

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Constitutive ERK activity induces downregulation of tristetraprolin, a major protein controlling interleukin8/CXCL8 mRNA stability in melanoma cells

American Journal of Physiology: Cell Physiology, 2011-09, Vol.301 (3), p.C609-C618

2011

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Genetic and epigenetic factors affect RET gene expression in breast cancer cell lines and influence survival in patients

Oncotarget, 2016-05, Vol.7 (18), p.26465-26479

2016

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A prospective observational study of associated anomalies in Hirschsprung's disease

Orphanet journal of rare diseases, 2013-11, Vol.8 (1), p.184-184

2013

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Induction of RET dependent and independent pro-inflammatory programs in human peripheral blood mononuclear cells from Hirschsprung patients

PloS one, 2013-03, Vol.8 (3), p.e59066-e59066

2013

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The involvement of the RET variant G691S in medullary thyroid carcinoma enlightened by a meta‐analysis study

International journal of cancer, 2013-06, Vol.132 (12), p.2808-2819

2013

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Functional Loss of Semaphorin 3C and/or Semaphorin 3D and Their Epistatic Interaction with Ret Are Critical to Hirschsprung Disease Liability

American journal of human genetics, 2015-04, Vol.96 (4), p.581-596

2015

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Expression Variability and Function of the RET Gene in Adult Peripheral Blood Mononuclear Cells

Journal of cellular physiology, 2014-12, Vol.229 (12), p.2027-2037

2014

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The involvement of the RET variant G691S in medullary thyroid carcinoma: Conflicting results of meta‐analyses need to be reconciled

International journal of cancer, 2013-10, Vol.133 (7), p.1760-1761

2013

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A common variant located in the 3′UTR of the RET gene is associated with protection from Hirschsprung disease

Human mutation, 2007-02, Vol.28 (2), p.168-176

2007

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Molecular mechanisms of RET-induced Hirschsprung pathogenesis

Annals of medicine (Helsinki), 2006-01, Vol.38 (1), p.11-19

2006

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CONSTITUTIVE ERK ACTIVITY INDUCES DOWN-REGULATION OF TRISTETRAPROLIN, A MAJOR PROTEIN CONTROLLING INTERLEUKIN8/CXCL8 mRNA STABILITY IN MELANOMA CELLS

American Journal of Physiology: Cell Physiology, 2011-05

2011

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Constitutive ERK activity induces downregulation of tristetraprolin, a major protein controlling interleukin8/CXCL8 mRNA stability in melanoma cells

American Journal of Physiology: Cell Physiology, 2011, Vol.70 (3)

2011

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A common haplotype at the 5′ end of the RET proto-oncogene, overrepresented in Hirschsprung patients, is associated with reduced gene expression

Human mutation, 2005-02, Vol.25 (2), p.189-195

2005

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Allele-Specific Expression at the RET Locus in Blood and Gut Tissue of Individuals Carrying Risk Alleles for Hirschsprung Disease

Human mutation, 2013-05, Vol.34 (5), p.754-762

2013

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Allele-Specific Expression at the RET Locus in Blood and Gut Tissue of Individuals Carrying Risk Alleles for Hirschsprung Disease

Human mutation, 2013-07, Vol.34 (7), p.1047-1047

2013

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Complex pathogenesis of Hirschsprung's disease in a patient with hydrocephalus, vesico-ureteral reflux and a balanced translocation t(3;17)(p12;q11)

European journal of human genetics : EJHG, 2009-04, Vol.17 (4), p.483-490

2009

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Double Heterozygosity for a RET Substitution Interfering with Splicing and an EDNRB Missense Mutation in Hirschsprung Disease

American journal of human genetics, 1999-04, Vol.64 (4), p.1216-1221

1999

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A Rare Haplotype of the RET Proto-Oncogene Is a Risk-Modifying Allele in Hirschsprung Disease

American journal of human genetics, 2002-10, Vol.71 (4), p.969-974

2002

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A common variant located in the 3′UTR of theRET gene is associated with protection from Hirschsprung disease

Human mutation, 2007-02, Vol.28 (2), p.168-176

2007

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Control of pro-angiogenic cytokine mRNA half-life in cancer: the role of AU-rich elements and associated proteins

A synonymous polymorphism of the Tristetraprolin (TTP) gene, an AU-rich mRNA-binding protein, affects translation efficiency and response to Herceptin treatment in breast cancer patients

Contribution of rare and common variants determine complex diseases—Hirschsprung disease as a model

Differential Contributions of Rare and Common, Coding and Noncoding Ret Mutations to Multifactorial Hirschsprung Disease Liability

Regulation of the mRNA half-life in breast cancer

Constitutive ERK activity induces downregulation of tristetraprolin, a major protein controlling interleukin8/CXCL8 mRNA stability in melanoma cells

Genetic and epigenetic factors affect RET gene expression in breast cancer cell lines and influence survival in patients

A prospective observational study of associated anomalies in Hirschsprung's disease

Induction of RET dependent and independent pro-inflammatory programs in human peripheral blood mononuclear cells from Hirschsprung patients

Correction: Induction of RET Dependent and Independent Pro-Inflammatory Programs in Human Peripheral Blood Mononuclear Cells from Hirschsprung Patients

The involvement of the RET variant G691S in medullary thyroid carcinoma enlightened by a meta‐analysis study

Functional Loss of Semaphorin 3C and/or Semaphorin 3D and Their Epistatic Interaction with Ret Are Critical to Hirschsprung Disease Liability

Expression Variability and Function of the RET Gene in Adult Peripheral Blood Mononuclear Cells

The involvement of the RET variant G691S in medullary thyroid carcinoma: Conflicting results of meta‐analyses need to be reconciled

A common variant located in the 3′UTR of the RET gene is associated with protection from Hirschsprung disease

Molecular mechanisms of RET-induced Hirschsprung pathogenesis

CONSTITUTIVE ERK ACTIVITY INDUCES DOWN-REGULATION OF TRISTETRAPROLIN, A MAJOR PROTEIN CONTROLLING INTERLEUKIN8/CXCL8 mRNA STABILITY IN MELANOMA CELLS

Constitutive ERK activity induces downregulation of tristetraprolin, a major protein controlling interleukin8/CXCL8 mRNA stability in melanoma cells

A common haplotype at the 5′ end of the RET proto-oncogene, overrepresented in Hirschsprung patients, is associated with reduced gene expression

Allele-Specific Expression at the RET Locus in Blood and Gut Tissue of Individuals Carrying Risk Alleles for Hirschsprung Disease

Allele-Specific Expression at the RET Locus in Blood and Gut Tissue of Individuals Carrying Risk Alleles for Hirschsprung Disease

Complex pathogenesis of Hirschsprung's disease in a patient with hydrocephalus, vesico-ureteral reflux and a balanced translocation t(3;17)(p12;q11)

Double Heterozygosity for a RET Substitution Interfering with Splicing and an EDNRB Missense Mutation in Hirschsprung Disease

A Rare Haplotype of the RET Proto-Oncogene Is a Risk-Modifying Allele in Hirschsprung Disease

A common variant located in the 3′UTR of theRET gene is associated with protection from Hirschsprung disease

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