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Brain aging and neurodegeneration: from a mitochondrial point of view

Journal of neurochemistry, 2017-11, Vol.143 (4), p.418-431

2017

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Impairments in Brain Bioenergetics in Aging and Tau Pathology: A Chicken and Egg Situation?

Cells (Basel, Switzerland), 2021-09, Vol.10 (10), p.2531

Grimm, Amandine

2021

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Insights into Disease-Associated Tau Impact on Mitochondria

International journal of molecular sciences, 2020-09, Vol.21 (17), p.6344

2020

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Mitochondrial dysfunction: the missing link between aging and sporadic Alzheimer’s disease

Biogerontology, 2016-04, Vol.17 (2), p.281-296

2016

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Mitochondria- and Oxidative Stress-Targeting Substances in Cognitive Decline-Related Disorders: From Molecular Mechanisms to Clinical Evidence

Oxidative medicine and cellular longevity, 2019-01, Vol.2019 (2019), p.1-26

2019

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Circadian Control of DRP1 Activity Regulates Mitochondrial Dynamics and Bioenergetics

Cell metabolism, 2018-03, Vol.27 (3), p.657-666.e5

2018

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Mitochondria, Estrogen and Female Brain Aging

Frontiers in aging neuroscience, 2018-04, Vol.10, p.124-124

2018

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Ginkgo biloba extract increases neurite outgrowth and activates the Akt/mTOR pathway

PloS one, 2019-12, Vol.14 (12), p.e0225761-e0225761

2019

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Spermidine Rescues Bioenergetic and Mitophagy Deficits Induced by Disease-Associated Tau Protein

International journal of molecular sciences, 2023-03, Vol.24 (6), p.5297

2023

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Dietary Mitophagy Enhancer: A Strategy for Healthy Brain Aging?

Antioxidants, 2020-10, Vol.9 (10), p.932

2020

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Mitochondria Transfer in Brain Injury and Disease

Cells (Basel, Switzerland), 2022-11, Vol.11 (22), p.3603

2022

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Mitochondria modulatory effects of new TSPO ligands in a cellular model of tauopathies

Journal of neuroendocrinology, 2020-01, Vol.32 (1), p.e12796-n/a

2020

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TSPO Ligands Boost Mitochondrial Function and Pregnenolone Synthesis

Journal of Alzheimer's disease, 2019-01, Vol.72 (4), p.1045-1058

2019

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From Young to Old: Mimicking Neuronal Aging in Directly Converted Neurons from Young Donors

Cells (Basel, Switzerland), 2024-08, Vol.13 (15), p.1260

2024

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Local Oxidative Damage in the Soma and Dendrites Quarantines Neuronal Mitochondria at the Site of Insult

iScience, 2018-08, Vol.6, p.114-127

2018

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Transmission-selective muscle pathology induced by the active propagation of mutant huntingtin across the human neuromuscular synapse

Frontiers in molecular neuroscience, 2024-01, Vol.16, p.1287510-1287510

2024

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Genetically Engineered Triple MAPT -Mutant Human-Induced Pluripotent Stem Cells (N279K, P301L, and E10+16 Mutations) Exhibit Impairments in Mitochondrial Bioenergetics and Dynamics

Cells (Basel, Switzerland), 2023-05, Vol.12 (10), p.1385

2023

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Random errors in protein synthesis activate an age-dependent program of muscle atrophy in mice

Communications biology, 2021-06, Vol.4 (1), p.703-703, Article 703

2021

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Inhibition of the mitochondrial enzyme ABAD restores the amyloid-β-mediated deregulation of estradiol

PloS one, 2011-12, Vol.6 (12), p.e28887

2011

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Silencing of the ER and Integrative Stress Responses in the Liver of Mice with Error-Prone Translation

Cells (Basel, Switzerland), 2021-10, Vol.10 (11), p.2856

2021

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Clock-Controlled Mitochondrial Dynamics Correlates with Cyclic Pregnenolone Synthesis

Cells (Basel, Switzerland), 2020-10, Vol.9 (10), p.2323

2020

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Alzheimer's amyloid-β peptide disturbs P2X7 receptor-mediated circadian oscillations of intracellular calcium

Folia neuropathologica, 2016-01, Vol.54 (4), p.360-368

2016

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Sex differences in Alzheimer’s disease: metabolic reprogramming and therapeutic intervention

Trends in endocrinology and metabolism, 2021-12, Vol.32 (12), p.963-979

2021

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Gelsemium low doses protect against serum deprivation-induced stress on mitochondria in neuronal cells

Journal of ethnopharmacology, 2025-01, Vol.336, p.118714, Article 118714

2025

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Link between the unfolded protein response and dysregulation of mitochondrial bioenergetics in Alzheimer’s disease

Cellular and molecular life sciences : CMLS, 2019-04, Vol.76 (7), p.1419-1431

2019

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Brain aging and neurodegeneration: from a mitochondrial point of view

Impairments in Brain Bioenergetics in Aging and Tau Pathology: A Chicken and Egg Situation?

Insights into Disease-Associated Tau Impact on Mitochondria

Mitochondrial dysfunction: the missing link between aging and sporadic Alzheimer’s disease

Mitochondria- and Oxidative Stress-Targeting Substances in Cognitive Decline-Related Disorders: From Molecular Mechanisms to Clinical Evidence

Circadian Control of DRP1 Activity Regulates Mitochondrial Dynamics and Bioenergetics

Mitochondria, Estrogen and Female Brain Aging

Ginkgo biloba extract increases neurite outgrowth and activates the Akt/mTOR pathway

Spermidine Rescues Bioenergetic and Mitophagy Deficits Induced by Disease-Associated Tau Protein

Dietary Mitophagy Enhancer: A Strategy for Healthy Brain Aging?

Mitochondria Transfer in Brain Injury and Disease

Mitochondria modulatory effects of new TSPO ligands in a cellular model of tauopathies

TSPO Ligands Boost Mitochondrial Function and Pregnenolone Synthesis

From Young to Old: Mimicking Neuronal Aging in Directly Converted Neurons from Young Donors

Local Oxidative Damage in the Soma and Dendrites Quarantines Neuronal Mitochondria at the Site of Insult

Transmission-selective muscle pathology induced by the active propagation of mutant huntingtin across the human neuromuscular synapse

Genetically Engineered Triple MAPT -Mutant Human-Induced Pluripotent Stem Cells (N279K, P301L, and E10+16 Mutations) Exhibit Impairments in Mitochondrial Bioenergetics and Dynamics

Random errors in protein synthesis activate an age-dependent program of muscle atrophy in mice

Inhibition of the mitochondrial enzyme ABAD restores the amyloid-β-mediated deregulation of estradiol

Silencing of the ER and Integrative Stress Responses in the Liver of Mice with Error-Prone Translation

Clock-Controlled Mitochondrial Dynamics Correlates with Cyclic Pregnenolone Synthesis

Alzheimer's amyloid-β peptide disturbs P2X7 receptor-mediated circadian oscillations of intracellular calcium

Sex differences in Alzheimer’s disease: metabolic reprogramming and therapeutic intervention

Gelsemium low doses protect against serum deprivation-induced stress on mitochondria in neuronal cells

Link between the unfolded protein response and dysregulation of mitochondrial bioenergetics in Alzheimer’s disease

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