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361 Antagomir to MicroRNA-21 Reverses the Loss of Negative TGF- Signaling From Inappropriately Decreased Smad7 Expression in Crohn's Disease, and Decreases Excess Collagen, CTGF, IGF-I and Fibrosis in TNBS-Induced Colitis

Gastroenterology (New York, N.Y. 1943), 2012-05, Vol.142 (5), p.S-85-S-85

2012

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Activation of the umami taste receptor (T1R1/T1R3) initiates the peristaltic reflex and pellet propulsion in the distal colon

American journal of physiology: Gastrointestinal and liver physiology, 2014-12, Vol.307 (11), p.G1100-G1107

2014

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Amelioration of excess collagen IαI, fibrosis, and smooth muscle growth in TNBS‐induced colitis in IGF‐I(+/−) mice

Inflammatory bowel diseases, 2011-03, Vol.17 (3), p.711-719

2011

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Artificial light at night (ALAN) pollution alters bat lunar chronobiology: insights from broad-scale long-term acoustic monitoring

Ecological processes, 2024-02, Vol.13 (1), p.13-12, Article 13

2024

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Cilengitide Blocks αVβ3 Integrin-Mediated, Rgd-Dependent Activation of Latent TGF-β1 and Attenuates Fibrosis in TNBS-Induced Colitis and Human Muscle of Stricturing Crohn's Disease

Gastroenterology (New York, N.Y. 1943), 2011, Vol.140 (5), p.S-109-S-109

2011

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Coexpression of 5-HT 2A and 5-HT 4 receptors coupled to distinct signaling pathways in human intestinal muscle cells

Gastroenterology (New York, N.Y. 1943), 1995, Vol.109 (6), p.1791-1800

1995

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Coexpression of 5-HT2A and 5-HT4 receptors coupled to distinct signaling pathways in human intestinal muscle cells

Gastroenterology (New York, N.Y. 1943), 1995-12, Vol.109 (6), p.1791-1800

1995

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Collagen production by human smooth muscle cells isolated during intestinal organogenesis

Anatomy and embryology, 1992-05, Vol.185 (6), p.517-527

1992

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Cytokine-induced iNOS and ERK1/2 inhibit adenylyl cyclase type 5/6 activity and stimulate phosphodiesterase 4D5 activity in intestinal longitudinal smooth muscle

American Journal of Physiology: Cell Physiology, 2014-08, Vol.307 (4), p.C402-C411

2014

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Diurnal roost selection of northern long-eared bats on the southern edge of their range

Forest ecology and management, 2021-09, Vol.496, p.119410, Article 119410

2021

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Dynamic occupancy in a peripheral population of Myotis septentrionalis during disease outbreak

Landscape ecology, 2024-09, Vol.39 (9), p.173, Article 173

2024

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Early treatment of white‐nose syndrome is necessary to stop population decline

The Journal of applied ecology, 2022-10, Vol.59 (10), p.2531-2541

2022

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Endogenous IGF-I and [alpha]^sub v^[beta]^sub 3^ integrin ligands regulate increased smooth muscle growth in TNBS-induced colitis

American journal of physiology: Gastrointestinal and liver physiology, 2009-06, Vol.296 (6), p.G1230

2009

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Endogenous IGF-I and alpha v beta3 integrin ligands regulate increased smooth muscle growth in TNBS-induced colitis

American journal of physiology: Gastrointestinal and liver physiology, 2009-06, Vol.296 (6), p.G1230

2009

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Endogenous IGF-I and alphaVbeta3 integrin ligands regulate increased smooth muscle hyperplasia in stricturing Crohn's disease

Gastroenterology (New York, N.Y. 1943), 2010-01, Vol.138 (1), p.285-293

2010

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Endogenous IGF-I and α v β 3 integrin ligands regulate increased smooth muscle growth in TNBS-induced colitis

American journal of physiology: Gastrointestinal and liver physiology, 2009-06, Vol.296 (6), p.G1230-G1237

2009

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Endogenous IGF-I and αvβ3 integrin ligands regulate increased smooth muscle growth in TNBS-induced colitis

American journal of physiology: Gastrointestinal and liver physiology, 2009-06, Vol.296 (6), p.G1230-G1237

2009

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Endogenous IGF-I and αvβ3 integrin ligands regulate increased smooth muscle growth in TNBS-induced colitis

American journal of physiology: Gastrointestinal and liver physiology, 2009, Vol.59 (6)

2009

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Endogenous IGF-I and αVβ3 Integrin Ligands Regulate Increased Smooth Muscle Hyperplasia in Stricturing Crohn's Disease

Gastroenterology (New York, N.Y. 1943), 2010, Vol.138 (1), p.285-293

2010

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Endogenous IGFBP‐3 regulates excess collagen expression in intestinal smooth muscle cells of Crohn's disease strictures

Inflammatory bowel diseases, 2011-01, Vol.17 (1), p.193-201

2011

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Endoplasmic Reticulum Stress in Subepithelial Myofibroblasts Increases the TGF-[beta]1 Activity That Regulates Fibrosis in Crohn's Disease

Inflammatory bowel diseases, 2020-06, Vol.26 (6), p.809

2020

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Endoplasmic Reticulum Stress in Subepithelial Myofibroblasts Increases the TGF-β1 Activity That Regulates Fibrosis in Crohn’s Disease

Inflammatory bowel diseases, 2020-05, Vol.26 (6), p.809-819

2020

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Frequent arousals from winter torpor in Rafinesque's big-eared bat (Corynorhinus rafinesquii)

PloS one, 2012-11, Vol.7 (11), p.e49754-e49754

2012

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Genes and environment in neonatal intraventricular hemorrhage

Seminars in perinatology, 2015-12, Vol.39 (8), p.592-603

2015

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Hypercontractility of intestinal longitudinal smooth muscle induced by cytokines is mediated by the nuclear factor-κB/AMP-activated kinase/myosin light chain kinase pathway

The Journal of pharmacology and experimental therapeutics, 2014-07, Vol.350 (1), p.89-98

2014

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361 Antagomir to MicroRNA-21 Reverses the Loss of Negative TGF- Signaling From Inappropriately Decreased Smad7 Expression in Crohn's Disease, and Decreases Excess Collagen, CTGF, IGF-I and Fibrosis in TNBS-Induced Colitis

Activation of the umami taste receptor (T1R1/T1R3) initiates the peristaltic reflex and pellet propulsion in the distal colon

Amelioration of excess collagen IαI, fibrosis, and smooth muscle growth in TNBS‐induced colitis in IGF‐I(+/−) mice

Artificial light at night (ALAN) pollution alters bat lunar chronobiology: insights from broad-scale long-term acoustic monitoring

Cilengitide Blocks αVβ3 Integrin-Mediated, Rgd-Dependent Activation of Latent TGF-β1 and Attenuates Fibrosis in TNBS-Induced Colitis and Human Muscle of Stricturing Crohn's Disease

Coexpression of 5-HT 2A and 5-HT 4 receptors coupled to distinct signaling pathways in human intestinal muscle cells

Coexpression of 5-HT2A and 5-HT4 receptors coupled to distinct signaling pathways in human intestinal muscle cells

Collagen production by human smooth muscle cells isolated during intestinal organogenesis

Cytokine-induced iNOS and ERK1/2 inhibit adenylyl cyclase type 5/6 activity and stimulate phosphodiesterase 4D5 activity in intestinal longitudinal smooth muscle

Diurnal roost selection of northern long-eared bats on the southern edge of their range

Dynamic occupancy in a peripheral population of Myotis septentrionalis during disease outbreak

Early treatment of white‐nose syndrome is necessary to stop population decline

Endogenous IGF-I and [alpha]^sub v^[beta]^sub 3^ integrin ligands regulate increased smooth muscle growth in TNBS-induced colitis

Endogenous IGF-I and alpha v beta3 integrin ligands regulate increased smooth muscle growth in TNBS-induced colitis

Endogenous IGF-I and alphaVbeta3 integrin ligands regulate increased smooth muscle hyperplasia in stricturing Crohn's disease

Endogenous IGF-I and α v β 3 integrin ligands regulate increased smooth muscle growth in TNBS-induced colitis

Endogenous IGF-I and αvβ3 integrin ligands regulate increased smooth muscle growth in TNBS-induced colitis

Endogenous IGF-I and αvβ3 integrin ligands regulate increased smooth muscle growth in TNBS-induced colitis

Endogenous IGF-I and αVβ3 Integrin Ligands Regulate Increased Smooth Muscle Hyperplasia in Stricturing Crohn's Disease

Endogenous IGFBP‐3 regulates excess collagen expression in intestinal smooth muscle cells of Crohn's disease strictures

Endoplasmic Reticulum Stress in Subepithelial Myofibroblasts Increases the TGF-[beta]1 Activity That Regulates Fibrosis in Crohn's Disease

Endoplasmic Reticulum Stress in Subepithelial Myofibroblasts Increases the TGF-β1 Activity That Regulates Fibrosis in Crohn’s Disease

Frequent arousals from winter torpor in Rafinesque's big-eared bat (Corynorhinus rafinesquii)

Genes and environment in neonatal intraventricular hemorrhage

Hypercontractility of intestinal longitudinal smooth muscle induced by cytokines is mediated by the nuclear factor-κB/AMP-activated kinase/myosin light chain kinase pathway

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