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Connective tissue growth factor: a fibrogenic master switch in fibrotic liver diseases

Liver international, 2008-09, Vol.28 (8), p.1065-1079

2008

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Serum resistin levels in critically ill patients are associated with inflammation, organ dysfunction and metabolism and may predict survival of non-septic patients

Critical care (London, England), 2009-01, Vol.13 (3), p.R95-R95

2009

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Monitoring fibrogenic progression in the liver

Clinica chimica acta, 2014-06, Vol.433, p.111-122

2014

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Biomarkers of liver fibrosis: Clinical translation of molecular pathogenesis or based on liver-dependent malfunction tests

Clinica chimica acta, 2007-06, Vol.381 (2), p.107-113

2007

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Non-invasive biomarkers for monitoring the fibrogenic process in liver： A short survey

World journal of gastroenterology : WJG, 2009-05, Vol.15 (20), p.2433-2440

2009

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Differential effects of TGF-β on connective tissue growth factor (CTGF/CCN2) expression in hepatic stellate cells and hepatocytes

Journal of hepatology, 2007-11, Vol.47 (5), p.699-710

2007

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Pharmacological application of caffeine inhibits TGF-β-stimulated connective tissue growth factor expression in hepatocytes via PPARγ and SMAD2/3-dependent pathways

Journal of hepatology, 2008-11, Vol.49 (5), p.758-767

2008

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Less Smad2 is good for you! A scientific update on coffee's liver benefits

Hepatology (Baltimore, Md.), 2009-09, Vol.50 (3), p.970-978

Gressner, Olav A.

2009

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Evolving concepts of liver fibrogenesis provide new diagnostic and therapeutic options

Comparative hepatology, 2007-07, Vol.6 (1), p.7-7, Article 7

2007

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Connective tissue growth factor is a Smad2 regulated amplifier of transforming growth factor β actions in hepatocytes—But without modulating bone morphogenetic protein 7 signaling

Hepatology (Baltimore, Md.), 2009-06, Vol.49 (6), p.2021-2030

2009

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Activation of hepatic stellate cells is associated with cytokine expression in thioacetamide-induced hepatic fibrosis in mice

Laboratory investigation, 2008-11, Vol.88 (11), p.1192-1203

2008

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BMP-7 as antagonist of organ fibrosis

Frontiers in bioscience (Landmark. Print), 2009-06, Vol.14 (13), p.4992-5012

2009

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In the Search of the Magic Bullet

Gastroenterology (New York, N.Y. 1943), 2010-11, Vol.139 (5), p.1453-1457

Gressner, Olav A

2010

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Changing the pathogenetic roadmap of liver fibrosis? Where did it start; where will it go?

Journal of gastroenterology and hepatology, 2008-07, Vol.23 (7pt1), p.1024-1035

2008

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Connective tissue growth factor (CTGF/CCN2) in serum is an indicator of fibrogenic progression and malignant transformation in patients with chronic hepatitis B infection

Clinica chimica acta, 2013-06, Vol.421, p.126-131

2013

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Oral application of 1,7-dimethylxanthine (paraxanthine) attenuates the formation of experimental cholestatic liver fibrosis

Hepatology research, 2011-11, Vol.41 (11), p.1094-1109

2011

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Connective Tissue Growth Factor in Serum as a New Candidate Test for Assessment of Hepatic Fibrosis

Clinical chemistry (Baltimore, Md.), 2006-09, Vol.52 (9), p.1815-1817

2006

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Connective tissue growth factor reacts as an IL-6/STAT3-regulated hepatic negative acute phase protein

World journal of gastroenterology : WJG, 2011-01, Vol.17 (2), p.151-163

2011

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Activation of TGF‐β within cultured hepatocytes and in liver injury leads to intracrine signaling with expression of connective tissue growth factor

Journal of cellular and molecular medicine, 2008-12, Vol.12 (6b), p.2717-2730

2008

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Identification of paraxanthine as the most potent caffeine-derived inhibitor of connective tissue growth factor expression in liver parenchymal cells

Liver international, 2009-07, Vol.29 (6), p.886-897

2009

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Gc-globulin (vitamin D binding protein) is synthesized and secreted by hepatocytes and internalized by hepatic stellate cells through Ca(2+)-dependent interaction with the megalin/gp330 receptor

Clinica chimica acta, 2008-04, Vol.390 (1-2), p.28-37

2008

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Impact of quality control accepted inter-laboratory variations on calculated Fibrotest/Actitest scores for the non-invasive biochemical assessment of liver fibrosis

Clinica chimica acta, 2009-11, Vol.409 (1), p.90-95

2009

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Intracrine signalling of activin A in hepatocytes upregulates connective tissue growth factor (CTGF/CCN2) expression

Liver international, 2008-10, Vol.28 (9), p.1207-1216

2008

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Increased serum xylosyltransferase activity in patients with liver fibrosis

Clinica chimica acta, 2009-11, Vol.409 (1), p.123-126

2009

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Retraction notice to ”Intraperitoneal application of caffeine prevents D-galactosamine induced hepatic expression of connective tissue growth factor (CTGF/CCN2) in the rat” [JHEPAT 50 (2009) 1053–1055]

Journal of hepatology, 2010-09, Vol.53 (3), p.590-590

2010

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Connective tissue growth factor: a fibrogenic master switch in fibrotic liver diseases

Serum resistin levels in critically ill patients are associated with inflammation, organ dysfunction and metabolism and may predict survival of non-septic patients

Monitoring fibrogenic progression in the liver

Biomarkers of liver fibrosis: Clinical translation of molecular pathogenesis or based on liver-dependent malfunction tests

Non-invasive biomarkers for monitoring the fibrogenic process in liver： A short survey

Differential effects of TGF-β on connective tissue growth factor (CTGF/CCN2) expression in hepatic stellate cells and hepatocytes

Pharmacological application of caffeine inhibits TGF-β-stimulated connective tissue growth factor expression in hepatocytes via PPARγ and SMAD2/3-dependent pathways

Less Smad2 is good for you! A scientific update on coffee's liver benefits

Evolving concepts of liver fibrogenesis provide new diagnostic and therapeutic options

Connective tissue growth factor is a Smad2 regulated amplifier of transforming growth factor β actions in hepatocytes—But without modulating bone morphogenetic protein 7 signaling

Activation of hepatic stellate cells is associated with cytokine expression in thioacetamide-induced hepatic fibrosis in mice

BMP-7 as antagonist of organ fibrosis

In the Search of the Magic Bullet

Changing the pathogenetic roadmap of liver fibrosis? Where did it start; where will it go?

Connective tissue growth factor (CTGF/CCN2) in serum is an indicator of fibrogenic progression and malignant transformation in patients with chronic hepatitis B infection

Oral application of 1,7-dimethylxanthine (paraxanthine) attenuates the formation of experimental cholestatic liver fibrosis

Connective Tissue Growth Factor in Serum as a New Candidate Test for Assessment of Hepatic Fibrosis

Connective tissue growth factor reacts as an IL-6/STAT3-regulated hepatic negative acute phase protein

Activation of TGF‐β within cultured hepatocytes and in liver injury leads to intracrine signaling with expression of connective tissue growth factor

Identification of paraxanthine as the most potent caffeine-derived inhibitor of connective tissue growth factor expression in liver parenchymal cells

Gc-globulin (vitamin D binding protein) is synthesized and secreted by hepatocytes and internalized by hepatic stellate cells through Ca(2+)-dependent interaction with the megalin/gp330 receptor

Impact of quality control accepted inter-laboratory variations on calculated Fibrotest/Actitest scores for the non-invasive biochemical assessment of liver fibrosis

Intracrine signalling of activin A in hepatocytes upregulates connective tissue growth factor (CTGF/CCN2) expression

Increased serum xylosyltransferase activity in patients with liver fibrosis

Retraction notice to ”Intraperitoneal application of caffeine prevents D-galactosamine induced hepatic expression of connective tissue growth factor (CTGF/CCN2) in the rat” [JHEPAT 50 (2009) 1053–1055]

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