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Evidence that Ca2+-release-activated Ca2+ channels in rat hepatocytes are required for the maintenance of hormone-induced Ca2+ oscillations

Biochemical journal, 2003-03, Vol.370 (Pt 2), p.695-702

2003

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Maintenance of the filamentous actin cytoskeleton is necessary for the activation of store-operated Ca2+ channels, but not other types of plasma-membrane Ca2+ channels, in rat hepatocytes

Biochemical journal, 2002-04, Vol.363 (Pt 1), p.117-126

2002

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Inositol trisphosphate analogues selective for types I and II inositol trisphosphate receptors exert differential effects on vasopressin-stimulated Ca2+ inflow and Ca2+ release from intracellular stores in rat hepatocytes

Biochemical journal, 2004-07, Vol.381 (Pt 2), p.519-526

2004

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Evidence for the involvement of a small subregion of the endoplasmic reticulum in the inositol trisphosphate receptor-induced activation of Ca2+ inflow in rat hepatocytes

Biochemical journal, 1999-07, Vol.341 ( Pt 2) (2), p.401-408

1999

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Evidence that 2-aminoethyl diphenylborate is a novel inhibitor of store-operated Ca2+ channels in liver cells, and acts through a mechanism which does not involve inositol trisphosphate receptors

Biochemical journal, 2001-03, Vol.354 (Pt 2), p.285-290

2001

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Regulation of F-actin and Endoplasmic Reticulum Organization by the Trimeric G-protein Gi2 in Rat Hepatocytes

The Journal of biological chemistry, 2000-07, Vol.275 (29), p.22229-22237

2000

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An Evaluation of Strategies Available for the Identification of GTP-Binding Proteins Required in Intracellular Signalling Pathways

Cellular Signalling, 1997-05, Vol.9 (3), p.207-218

1997

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Induction of cholestasis in the perfused rat liver by 2-aminoethyl diphenylborate, an inhibitor of the hepatocyte plasma membrane Ca2+ channels

Journal of gastroenterology and hepatology, 2004-10, Vol.19 (10), p.1128-1134

2004

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Induction of cholestasis in the perfused rat liver by 2‐aminoethyl diphenylborate, an inhibitor of the hepatocyte plasma membrane Ca 2+ channels

Journal of gastroenterology and hepatology, 2004-10, Vol.19 (10), p.1128-1134

2004

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Intracellular mitochondrial membrane potential as an indicator of hepatocyte energy metabolism: Further evidence for thermodynamic control of metabolism

Biochimica et biophysica acta, 1988-12, Vol.936 (3), p.294-306

1988

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Maintenance of the filamentous actin cytoskeleton is necessary for the activation of store-operated Ca2+ channels, but not other types of plasma-membrane Ca2+ channels, in rat hepatocytes

Biochemical journal, 2002-04, Vol.363 (1), p.117-126

2002

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Evidence that 2-aminoethyl diphenylborate is a novel inhibitor of store-operated Ca2+ channels in liver cells, and acts through a mechanism which does not involve inositol trisphosphate receptors

Biochemical journal, 2001-03, Vol.354 (2), p.285-290

2001

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Store-operated Ca2+ inflow in Reuber hepatoma cells is inhibited by voltage-operated Ca2+ channel antagonists and, in contrast to freshly isolated hepatocytes, does not require a pertussis toxin-sensitive trimeric GTP-binding protein

Biochimica et biophysica acta. Molecular cell research, 2000-06, Vol.1497 (1), p.11-26

2000

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Store-activated Ca2+ inflow in Xenopus laevis oocytes: inhibition by primaquine and evaluation of the role of membrane fusion

Biochemical journal, 1996-11, Vol.319 ( Pt 3) (3), p.755-760

1996

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Protein kinase A regulates the disposition of Ca2+ which enters the cytoplasmic space through store-activated Ca2+ channels in rat hepatocytes by diverting inflowing Ca2+ to mitochondria

Biochemical journal, 1998-03, Vol.330 ( Pt 3) (3), p.1179-1187

1998

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Store-operated Ca 2+ inflow in Reuber hepatoma cells is inhibited by voltage-operated Ca 2+ channel antagonists and, in contrast to freshly isolated hepatocytes, does not require a pertussis toxin-sensitive trimeric GTP-binding protein

Biochimica et biophysica acta. Molecular cell research, 2000, Vol.1497 (1), p.11-26

2000

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Evidence that a low-molecular-mass GTP-binding protein is required for store-activated Ca2+ inflow in hepatocytes

Biochemical journal, 1997-12, Vol.328 ( Pt 2) (2), p.463-471

1997

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Evidence for the involvement of a small subregion of the endoplasmic reticulum in the inositol trisphosphate receptor-induced activation of Ca2+ inflow in rat hepatocytes

Biochemical journal, 1999-07, Vol.341 (2), p.401-408

1999

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Stimulation by thyroid hormone of coupled respiration and of respiration apparently not coupled to the synthesis of ATP in rat hepatocytes

The Journal of biological chemistry, 1992-05, Vol.267 (13), p.8903-8908

1992

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On the thyroid hormone-induced increase in respiratory capacity of isolated rat hepatocytes

Biochimica et biophysica acta. Bioenergetics, 1991-12, Vol.1098 (1), p.61-67

1991

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The administration of triiodothyronine to rats results in a lowering of the mitochondrial membrane potential in isolated hepatocytes

Biochimica et biophysica acta, 1991-12, Vol.1133 (1), p.89-94

1991

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Effects of thyroid status on glucose cycling by isolated rat hepatocytes

Metabolism, clinical and experimental, 1996, Vol.45 (1), p.101-108

1996

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The characterization of perfluorosuccinate as an inhibitor of gluconeogenesis in isolated rat hepatocytes

Biochemical pharmacology, 1989-09, Vol.38 (17), p.2867-2872

1989

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Operation and energy dependence of the reducing-equivalent shuttles during lactate metabolism by isolated hepatocytes

Biochimica et biophysica acta, 1992-09, Vol.1136 (3), p.223-230

1992

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Reducing-equivalent transfer to the mitochondria during gluconeogenesis and ureogenesis in hepatocytes from rats of different thyroid status

Biochimica et biophysica acta, 1992-10, Vol.1137 (1), p.34-38

1992

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Evidence that Ca2+-release-activated Ca2+ channels in rat hepatocytes are required for the maintenance of hormone-induced Ca2+ oscillations

Maintenance of the filamentous actin cytoskeleton is necessary for the activation of store-operated Ca2+ channels, but not other types of plasma-membrane Ca2+ channels, in rat hepatocytes

Inositol trisphosphate analogues selective for types I and II inositol trisphosphate receptors exert differential effects on vasopressin-stimulated Ca2+ inflow and Ca2+ release from intracellular stores in rat hepatocytes

Evidence for the involvement of a small subregion of the endoplasmic reticulum in the inositol trisphosphate receptor-induced activation of Ca2+ inflow in rat hepatocytes

Evidence that 2-aminoethyl diphenylborate is a novel inhibitor of store-operated Ca2+ channels in liver cells, and acts through a mechanism which does not involve inositol trisphosphate receptors

Regulation of F-actin and Endoplasmic Reticulum Organization by the Trimeric G-protein Gi2 in Rat Hepatocytes

An Evaluation of Strategies Available for the Identification of GTP-Binding Proteins Required in Intracellular Signalling Pathways

Induction of cholestasis in the perfused rat liver by 2-aminoethyl diphenylborate, an inhibitor of the hepatocyte plasma membrane Ca2+ channels

Induction of cholestasis in the perfused rat liver by 2‐aminoethyl diphenylborate, an inhibitor of the hepatocyte plasma membrane Ca 2+ channels

Intracellular mitochondrial membrane potential as an indicator of hepatocyte energy metabolism: Further evidence for thermodynamic control of metabolism

Maintenance of the filamentous actin cytoskeleton is necessary for the activation of store-operated Ca2+ channels, but not other types of plasma-membrane Ca2+ channels, in rat hepatocytes

Evidence that 2-aminoethyl diphenylborate is a novel inhibitor of store-operated Ca2+ channels in liver cells, and acts through a mechanism which does not involve inositol trisphosphate receptors

Store-operated Ca2+ inflow in Reuber hepatoma cells is inhibited by voltage-operated Ca2+ channel antagonists and, in contrast to freshly isolated hepatocytes, does not require a pertussis toxin-sensitive trimeric GTP-binding protein

Store-activated Ca2+ inflow in Xenopus laevis oocytes: inhibition by primaquine and evaluation of the role of membrane fusion

Protein kinase A regulates the disposition of Ca2+ which enters the cytoplasmic space through store-activated Ca2+ channels in rat hepatocytes by diverting inflowing Ca2+ to mitochondria

Store-operated Ca 2+ inflow in Reuber hepatoma cells is inhibited by voltage-operated Ca 2+ channel antagonists and, in contrast to freshly isolated hepatocytes, does not require a pertussis toxin-sensitive trimeric GTP-binding protein

Evidence that a low-molecular-mass GTP-binding protein is required for store-activated Ca2+ inflow in hepatocytes

Evidence for the involvement of a small subregion of the endoplasmic reticulum in the inositol trisphosphate receptor-induced activation of Ca2+ inflow in rat hepatocytes

Stimulation by thyroid hormone of coupled respiration and of respiration apparently not coupled to the synthesis of ATP in rat hepatocytes

On the thyroid hormone-induced increase in respiratory capacity of isolated rat hepatocytes

The administration of triiodothyronine to rats results in a lowering of the mitochondrial membrane potential in isolated hepatocytes

Effects of thyroid status on glucose cycling by isolated rat hepatocytes

The characterization of perfluorosuccinate as an inhibitor of gluconeogenesis in isolated rat hepatocytes

Operation and energy dependence of the reducing-equivalent shuttles during lactate metabolism by isolated hepatocytes

Reducing-equivalent transfer to the mitochondria during gluconeogenesis and ureogenesis in hepatocytes from rats of different thyroid status

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