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Correction: PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kB, MAPkinase and p53 pathways

Leukemia, 2022, Vol.36 (1), p.296-296

2022

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A selective inhibitor of the p110δ isoform of PI 3-kinase inhibits AML cell proliferation and survival and increases the cytotoxic effects of VP16

Oncogene, 2006-10, Vol.25 (50), p.6648-6659

2006

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PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF- Kappa B, MAPkinase and p53 pathways

Leukemia, 2005-04, Vol.19 (4), p.586-594

2005

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Treatment-related fertility impairment in long-term female childhood, adolescent and young adult cancer survivors: investigating dose-effect relationships in a European case-control study (PanCareLIFE)

Human reproduction (Oxford), 2021-05, Vol.36 (6), p.1561-1573

2021

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Second allogeneic bone marrow transplants from unrelated donors for graft failure following initial unrelated donor bone marrow transplantation

Bone marrow transplantation (Basingstoke), 1998-04, Vol.21 (7), p.687-690

1998

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Minimal residual disease status before allogeneic bone marrow transplantation is an important determinant of successful outcome for children and adolescents with acute lymphoblastic leukemia

Blood, 1998-12, Vol.92 (11), p.4072-4079

1998

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Constitutive activation of the Wnt/β-catenin signalling pathway in acute myeloid leukaemia

Oncogene, 2005-03, Vol.24 (14), p.2410-2420

2005

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Minimal residual disease status as a predictor of relapse after allogeneic bone marrow transplantation for children with acute lymphoblastic leukaemia

British journal of haematology, 1998-08, Vol.102 (3), p.860-871

1998

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A selective inhibitor of the p110 isoform of PI 3-kinase inhibits AML cell proliferation and survival and increases the cytotoxic effects of VP16

Oncogene, 2006-10, Vol.25 (50), p.6648-6659

2006

Volltextzugriff (PDF)

A selective inhibitor of the p110d isoform of PI 3-kinase inhibits AML cell proliferation and survival and increases the cytotoxic effects of VP16

Oncogene, 2006-10, Vol.25 (50), p.6648-6659

2006

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A selective inhibitor of the p110delta isoform of PI 3-kinase inhibits AML cell proliferation and survival and increases the cytotoxic effects of VP16

Oncogene, 2006-10, Vol.25 (50), p.6648-6659

2006

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PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB, Mapkinase and p53 pathways

Leukemia, 2005-04, Vol.19 (4), p.586

2005

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PI3-kinase Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kB, MAPkinase and p53 pathways

Leukemia, 2005-04, Vol.19 (4), p.586-594

2005

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P13-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-κB, mapkinase and p53 pathways

Leukemia, 2005, Vol.19 (4), p.586-594

2005

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Lack of effect of the human GM-CSF analog E21R on the survival of primary human acute myeloid leukemia cells

Blood, 2004-04, Vol.103 (8), p.3230-3232

2004

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PanCareLIFE: The scientific basis for a European project to improve long-term care regarding fertility, ototoxicity and health-related quality of life after cancer occurring among children and adolescents

European journal of cancer (1990), 2018-11, Vol.103, p.227-237

2018

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G541(P) Improving the efficiency of procedure lists in paediatric haematology-oncology - a quality improvement project

Archives of disease in childhood, 2015-04, Vol.100 (Suppl 3), p.A239-A240

2015

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Correction: PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kB, MAPkinase and p53 pathways

A selective inhibitor of the p110δ isoform of PI 3-kinase inhibits AML cell proliferation and survival and increases the cytotoxic effects of VP16

PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF- Kappa B, MAPkinase and p53 pathways

Treatment-related fertility impairment in long-term female childhood, adolescent and young adult cancer survivors: investigating dose-effect relationships in a European case-control study (PanCareLIFE)

Second allogeneic bone marrow transplants from unrelated donors for graft failure following initial unrelated donor bone marrow transplantation

Minimal residual disease status before allogeneic bone marrow transplantation is an important determinant of successful outcome for children and adolescents with acute lymphoblastic leukemia

Constitutive activation of the Wnt/β-catenin signalling pathway in acute myeloid leukaemia

Minimal residual disease status as a predictor of relapse after allogeneic bone marrow transplantation for children with acute lymphoblastic leukaemia

A selective inhibitor of the p110 isoform of PI 3-kinase inhibits AML cell proliferation and survival and increases the cytotoxic effects of VP16

A selective inhibitor of the p110d isoform of PI 3-kinase inhibits AML cell proliferation and survival and increases the cytotoxic effects of VP16

A selective inhibitor of the p110delta isoform of PI 3-kinase inhibits AML cell proliferation and survival and increases the cytotoxic effects of VP16

PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB, Mapkinase and p53 pathways

PI3-kinase Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kB, MAPkinase and p53 pathways

P13-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-κB, mapkinase and p53 pathways

Lack of effect of the human GM-CSF analog E21R on the survival of primary human acute myeloid leukemia cells

PanCareLIFE: The scientific basis for a European project to improve long-term care regarding fertility, ototoxicity and health-related quality of life after cancer occurring among children and adolescents

G541(P) Improving the efficiency of procedure lists in paediatric haematology-oncology - a quality improvement project

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