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Pre-plaque conformational changes in Alzheimer’s disease-linked Aβ and APP

Nature communications, 2017-03, Vol.8 (1), p.14726-14726, Article 14726

2017

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Early-life stress elicits peripheral and brain immune activation differently in wild type and 5xFAD mice in a sex-specific manner

Journal of neuroinflammation, 2022-06, Vol.19 (1), p.151-15, Article 151

2022

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Maternal separation differentially modulates early pathology by sex in 5xFAD Alzheimer’s disease-transgenic mice

Brain, behavior, & immunity. Health, 2023-10, Vol.32, p.100663-100663, Article 100663

2023

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Oligomerization of Alzheimer's beta-amyloid within processes and synapses of cultured neurons and brain

The Journal of neuroscience, 2004-04, Vol.24 (14), p.3592-3599

2004

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Chaperones Increase Association of tau Protein with Microtubules

Proceedings of the National Academy of Sciences - PNAS, 2003-01, Vol.100 (2), p.721-726

2003

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Stimulation of beta-amyloid precursor protein trafficking by insulin reduces intraneuronal beta-amyloid and requires mitogen-activated protein kinase signaling

The Journal of neuroscience, 2001-04, Vol.21 (8), p.2561-2570

2001

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Generation and Regulation of β‐Amyloid Peptide Variants by Neurons

Journal of neurochemistry, 1998-11, Vol.71 (5), p.1920-1925

1998

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Testosterone Reduces Neuronal Secretion of Alzheimer's β -Amyloid Peptides

Proceedings of the National Academy of Sciences - PNAS, 2000-02, Vol.97 (3), p.1202-1205

2000

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Estrogen reduces neuronal generation of Alzheimer β-amyloid peptides

Nature Medicine, 1998-04, Vol.4 (4), p.447-451

1998

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Endoplasmic Reticulum and Trans-Golgi Network Generate Distinct Populations of Alzheimer β -Amyloid Peptides

Proceedings of the National Academy of Sciences - PNAS, 1999-01, Vol.96 (2), p.742-747

1999

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Increased apolipoprotein E epsilon 4 in epilepsy with senile plaques

Annals of neurology, 1997-03, Vol.41 (3), p.402-404

1997

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ADAM10 and BACE1 are localized to synaptic vesicles

Journal of neurochemistry, 2015-11, Vol.135 (3), p.606-615

2015

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Cerebral A beta deposition precedes reduced cerebrospinal fluid and serum A beta 42/A beta 40 ratios in the App(NL-F/NL-F) knock-in mouse model of Alzheimer's disease

Alzheimer's research & therapy, 2023-03, Vol.15 (1)

2023

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Regulation of NMDA receptor trafficking by amyloid-β

Nature neuroscience, 2005-08, Vol.8 (8), p.1051-1058

2005

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Metal Chelator Decreases Alzheimer β-Amyloid Plaques

Neuron, 2001-06, Vol.30 (3), p.641-642

2001

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Intraneuronal Aβ42 Accumulation in Human Brain

The American journal of pathology, 2000, Vol.156 (1), p.15-20

2000

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Evidence for Phosphorylation and Oligomeric Assembly of Presenilin 1

Proceedings of the National Academy of Sciences - PNAS, 1997-05, Vol.94 (10), p.5090-5094

1997

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Myotonia in colchicine myoneuropathy

Muscle & nerve, 1996-07, Vol.19 (7), p.870-875

1996

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beta-Amyloid Accumulation Impairs Multivesicular Body Sorting by Inhibiting the Ubiquitin-Proteasome System

The Journal of neuroscience, 2006-04, Vol.26 (16), p.4277-4288

2006

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Beta-amyloid accumulation in APP mutant neurons reduces PSD-95 and GluR1 in synapses

Neurobiology of disease, 2005-11, Vol.20 (2), p.187-198

2005

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Intraneuronal Aβ accumulation and origin of plaques in Alzheimer's disease

Neurobiology of aging, 2005-10, Vol.26 (9), p.1235-1244

2005

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APP depletion alters selective pre- and post-synaptic proteins

Molecular and cellular neuroscience, 2019-03, Vol.95, p.86-95

2019

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Correction: Lesion of the subiculum reduces the spread of amyloid beta pathology to interconnected brain regions in a mouse model of Alzheimer's disease

Acta neuropathologica communications, 2024-02, Vol.12 (1), p.26-26, Article 26

2024

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Alzheimer beta-amyloid peptides: normal and abnormal localization

Histology and histopathology, 2002-01, Vol.17 (1), p.239

2002

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Apolipoprotein E, Aβ-Amyloid, and the Molecular Pathology of Alzheimer's Disease Therapeutic Implications

Annals of the New York Academy of Sciences, 1996-12, Vol.802 (1), p.42-49

1996

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Pre-plaque conformational changes in Alzheimer’s disease-linked Aβ and APP

Early-life stress elicits peripheral and brain immune activation differently in wild type and 5xFAD mice in a sex-specific manner

Maternal separation differentially modulates early pathology by sex in 5xFAD Alzheimer’s disease-transgenic mice

Oligomerization of Alzheimer's beta-amyloid within processes and synapses of cultured neurons and brain

Chaperones Increase Association of tau Protein with Microtubules

Stimulation of beta-amyloid precursor protein trafficking by insulin reduces intraneuronal beta-amyloid and requires mitogen-activated protein kinase signaling

Generation and Regulation of β‐Amyloid Peptide Variants by Neurons

Testosterone Reduces Neuronal Secretion of Alzheimer's β -Amyloid Peptides

Estrogen reduces neuronal generation of Alzheimer β-amyloid peptides

Endoplasmic Reticulum and Trans-Golgi Network Generate Distinct Populations of Alzheimer β -Amyloid Peptides

Increased apolipoprotein E epsilon 4 in epilepsy with senile plaques

ADAM10 and BACE1 are localized to synaptic vesicles

Cerebral A beta deposition precedes reduced cerebrospinal fluid and serum A beta 42/A beta 40 ratios in the App(NL-F/NL-F) knock-in mouse model of Alzheimer's disease

Regulation of NMDA receptor trafficking by amyloid-β

Metal Chelator Decreases Alzheimer β-Amyloid Plaques

Intraneuronal Aβ42 Accumulation in Human Brain

Evidence for Phosphorylation and Oligomeric Assembly of Presenilin 1

Myotonia in colchicine myoneuropathy

beta-Amyloid Accumulation Impairs Multivesicular Body Sorting by Inhibiting the Ubiquitin-Proteasome System

Beta-amyloid accumulation in APP mutant neurons reduces PSD-95 and GluR1 in synapses

Intraneuronal Aβ accumulation and origin of plaques in Alzheimer's disease

APP depletion alters selective pre- and post-synaptic proteins

Correction: Lesion of the subiculum reduces the spread of amyloid beta pathology to interconnected brain regions in a mouse model of Alzheimer's disease

Alzheimer beta-amyloid peptides: normal and abnormal localization

Apolipoprotein E, Aβ-Amyloid, and the Molecular Pathology of Alzheimer's Disease Therapeutic Implications

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