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Biophysical characterization of the Varroa destructor NaV1 sodium channel and its affinity for τ‐fluvalinate insecticide

The FASEB journal, 2017-07, Vol.31 (7), p.3066-3071

2017

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Biophysical characterization of the Varroa destructor Na V 1 sodium channel and its affinity for τ-fluvalinate insecticide

The FASEB journal, 2017-07, Vol.31 (7), p.3066-3071

2017

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Metaflumizone inhibits the honeybee NaV1 channel by targeting recovery from slow inactivation

FEBS letters, 2017-12, Vol.591 (23), p.3842-3849

2017

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Gating pore currents and the resting state of Naᵥ1.4 voltage sensor domains

Proceedings of the National Academy of Sciences - PNAS, 2012-11, Vol.109 (47), p.19250-19255

2012

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Metaflumizone inhibits the honeybee Na V 1 channel by targeting recovery from slow inactivation

FEBS letters, 2017-12, Vol.591 (23), p.3842-3849

2017

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Biophysics, pathophysiology, and pharmacology of ion channel gating pores

Frontiers in pharmacology, 2014-04, Vol.5, p.53-53

2014

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A proton leak current through the cardiac sodium channel is linked to mixed arrhythmia and the dilated cardiomyopathy phenotype

PloS one, 2012-05, Vol.7 (5), p.e38331

2012

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Molecular biology and biophysical properties of ion channel gating pores

Quarterly reviews of biophysics, 2014-11, Vol.47 (4), p.364-388

2014

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Biophysical characterization of the honeybee DSC1 orthologue reveals a novel voltage-dependent Ca2+ channel subfamily: CaV4

The Journal of general physiology, 2016-08, Vol.148 (2), p.133-145

2016

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Correction: A Proton Leak Current through the Cardiac Sodium Channel Is Linked to Mixed Arrhythmia and the Dilated Cardiomyopathy Phenotype

PloS one, 2012-06, Vol.7 (6)

2012

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Novel OSNR Measurement Techniques Based on Optical Spectrum Analysis and Their Application to Coherent-Detection Systems

Journal of lightwave technology, 2019-01, Vol.37 (2), p.562-570

2019

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Gating pore currents are defects in common with two Nav1.5 mutations in patients with mixed arrhythmias and dilated cardiomyopathy

The Journal of general physiology, 2015-02, Vol.145 (2), p.93-106

2015

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A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy

Scientific reports, 2018-09, Vol.8 (1), p.13804-12, Article 13804

2018

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Metaflumizone inhibits the honeybee NaV 1 channel by targeting recovery from slow inactivation

FEBS letters, 2017-12, Vol.591 (23), p.3842-3849

2017

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Nav1.5 mutations linked to dilated cardiomyopathy phenotypes: Is the gating pore current the missing link?

Channels (Austin, Tex.), 2014, Vol.8 (1), p.90-94

2014

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Honeybee CaV4 has distinct permeation, inactivation, and pharmacology from homologous NaV channels

The Journal of general physiology, 2024-05, Vol.156 (5)

2024

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Gating pore currents, a new pathological mechanism underlying cardiac arrhythmias associated with dilated cardiomyopathy

Channels (Austin, Tex.), 2015-05, Vol.9 (3), p.139-144

2015

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Substitutions of the S4DIV R2 residue (R1451) in NaV1.4 lead to complex forms of paramyotonia congenita and periodic paralyses

Scientific reports, 2018-02, Vol.8 (1), p.1-13, Article 2041

2018

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Biophysical Characterization of the Honeybee's DSC1 Ortholog Highlights a New Voltage Dependant Calcium Channel Subfamily

Biophysical journal, 2016-02, Vol.110 (3), p.34a-34a

2016

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Abstract 15933: Scn5a Gating Pore Current Causes Cardiac Arrhythmias Associated With Dilated Cardiomyopathy

Circulation (New York, N.Y.), 2015-11, Vol.132 (Suppl_3 Suppl 3), p.A15933-A15933

2015

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Characterization of the honeybee AmNaV1 channel and tools to assess the toxicity of insecticides

Scientific reports, 2015-07, Vol.5 (1), p.12475, Article 12475

2015

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Biophysical Characterization of Two NaV1.4 Mutations Making a Clinical Overlap between the Myotonia-Hyperkalemic and Hypokalemic Periodic Paralysis Clusters of Disorders

Biophysical journal, 2018-02, Vol.114 (3), p.632a-632a

2018

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Mutations in the Voltage Sensors of Domains I and II of Nav1.5 that are Associated with Arrhythmias and Dilated Cardiomyopathy Generate Gating Pore Currents

Frontiers in pharmacology, 2015, Vol.6, p.301-301

2015

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Gating Pore Currents are Common Defects of Two Nav1.5 Mutations in Patients with Mixed Arrhythmias and Dilated Cardiomyopathy

Biophysical journal, 2015-01, Vol.108 (2), p.572a-572a

2015

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Biophysical Characterization of Two Nav1.4 Mutations Identified in Patients with Cold-Induced Myotonia and Periodic Paralysis

Biophysical journal, 2016-02, Vol.110 (3), p.438a-438a

2016

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Biophysical characterization of the Varroa destructor NaV1 sodium channel and its affinity for τ‐fluvalinate insecticide

Biophysical characterization of the Varroa destructor Na V 1 sodium channel and its affinity for τ-fluvalinate insecticide

Metaflumizone inhibits the honeybee NaV1 channel by targeting recovery from slow inactivation

Gating pore currents and the resting state of Naᵥ1.4 voltage sensor domains

Metaflumizone inhibits the honeybee Na V 1 channel by targeting recovery from slow inactivation

Biophysics, pathophysiology, and pharmacology of ion channel gating pores

A proton leak current through the cardiac sodium channel is linked to mixed arrhythmia and the dilated cardiomyopathy phenotype

Molecular biology and biophysical properties of ion channel gating pores

Biophysical characterization of the honeybee DSC1 orthologue reveals a novel voltage-dependent Ca2+ channel subfamily: CaV4

Correction: A Proton Leak Current through the Cardiac Sodium Channel Is Linked to Mixed Arrhythmia and the Dilated Cardiomyopathy Phenotype

Novel OSNR Measurement Techniques Based on Optical Spectrum Analysis and Their Application to Coherent-Detection Systems

Gating pore currents are defects in common with two Nav1.5 mutations in patients with mixed arrhythmias and dilated cardiomyopathy

A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy

Metaflumizone inhibits the honeybee NaV 1 channel by targeting recovery from slow inactivation

Nav1.5 mutations linked to dilated cardiomyopathy phenotypes: Is the gating pore current the missing link?

Honeybee CaV4 has distinct permeation, inactivation, and pharmacology from homologous NaV channels

Gating pore currents, a new pathological mechanism underlying cardiac arrhythmias associated with dilated cardiomyopathy

Substitutions of the S4DIV R2 residue (R1451) in NaV1.4 lead to complex forms of paramyotonia congenita and periodic paralyses

Biophysical Characterization of the Honeybee's DSC1 Ortholog Highlights a New Voltage Dependant Calcium Channel Subfamily

Abstract 15933: Scn5a Gating Pore Current Causes Cardiac Arrhythmias Associated With Dilated Cardiomyopathy

Characterization of the honeybee AmNaV1 channel and tools to assess the toxicity of insecticides

Biophysical Characterization of Two NaV1.4 Mutations Making a Clinical Overlap between the Myotonia-Hyperkalemic and Hypokalemic Periodic Paralysis Clusters of Disorders

Mutations in the Voltage Sensors of Domains I and II of Nav1.5 that are Associated with Arrhythmias and Dilated Cardiomyopathy Generate Gating Pore Currents

Gating Pore Currents are Common Defects of Two Nav1.5 Mutations in Patients with Mixed Arrhythmias and Dilated Cardiomyopathy

Biophysical Characterization of Two Nav1.4 Mutations Identified in Patients with Cold-Induced Myotonia and Periodic Paralysis

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