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Actin Mediates the Nanoscale Membrane Organization of the Clustered Membrane Protein Influenza Hemagglutinin

Biophysical journal, 2013-05, Vol.104 (10), p.2182-2192

2013

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Antimicrobial agent triclosan is a proton ionophore uncoupler of mitochondria in living rat and human mast cells and in primary human keratinocytes

Journal of applied toxicology, 2016-06, Vol.36 (6), p.777-789

2016

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Arsenic as an Endocrine Disruptor: Effects of Arsenic on Estrogen Receptor–Mediated Gene Expression In Vivo and in Cell Culture

Toxicological sciences, 2007-07, Vol.98 (1), p.75-86

2007

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Phosphatidylinositol 4,5-Bisphosphate Mediates the Co-Distribution of Influenza A Hemagglutinin and Matrix Protein M1 at the Plasma Membrane

Viruses, 2022-11, Vol.14 (11), p.2509

2022

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Cetylpyridinium chloride (CPC) reduces zebrafish mortality from influenza infection: Super-resolution microscopy reveals CPC interference with multiple protein interactions with phosphatidylinositol 4,5-bisphosphate in immune function

Toxicology and applied pharmacology, 2022-04, Vol.440, p.115913-115913, Article 115913

2022

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Triclosan exposure, transformation, and human health effects

Journal of toxicology and environmental health. Part B, Critical reviews, 2017-01, Vol.20 (8), p.447-469

2017

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Elevated nitrate alters the metabolic activity of embryonic zebrafish

Environmental pollution (1987), 2018-04, Vol.235, p.180-185

2018

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Chronic Exposure to Arsenic in the Drinking Water Alters the Expression of Immune Response Genes in Mouse Lung

Environmental health perspectives, 2009-07, Vol.117 (7), p.1108-1115

2009

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Visualizing the Molecular Timing of a Physiological Decision at the Nanoscale

Biophysical journal, 2013-12, Vol.105 (12), p.2617-2618

2013

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Temporally resolved interactions between antigen-stimulated IgE receptors and Lyn kinase on living cells

The Journal of cell biology, 2005-11, Vol.171 (3), p.527-536

2005

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Lipid segregation and IgE receptor signaling: A decade of progress

Biochimica et biophysica acta, 2005-12, Vol.1746 (3), p.252-259

2005

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Antimicrobial cetylpyridinium chloride causes functional inhibition of mitochondria as potently as canonical mitotoxicants, nanostructural disruption of mitochondria, and mitochondrial Ca 2+ efflux in living rodent and primary human cells

Food and chemical toxicology, 2024-04, Vol.186, p.114547

2024

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Pharmaceutical agent cetylpyridinium chloride inhibits immune mast cell function by interfering with calcium mobilization

Food and chemical toxicology, 2023-09, Vol.179, p.113980-113980, Article 113980

2023

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Antimicrobial agent triclosan disrupts mitochondrial structure, revealed by super-resolution microscopy, and inhibits mast cell signaling via calcium modulation

Toxicology and applied pharmacology, 2018-06, Vol.349, p.39-54

2018

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Antimicrobial cetylpyridinium chloride causes functional inhibition of mitochondria as potently as canonical mitotoxicants, nanostructural disruption of mitochondria, and mitochondrial Ca2+ efflux in living rodent and primary human cells

Food and chemical toxicology, 2024-04, Vol.186, p.114547-114547, Article 114547

2024

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Maternal responsivity and oxytocin in opioid‐dependent mothers

Developmental psychobiology, 2020-01, Vol.62 (1), p.21-35

2020

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Triclosan disrupts immune cell function by depressing Ca2+ influx following acidification of the cytoplasm

Toxicology and applied pharmacology, 2020-10, Vol.405, p.115205-115205, Article 115205

2020

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Association between In Utero arsenic exposure, placental gene expression, and infant birth weight: a US birth cohort study

Environmental health, 2013-07, Vol.12 (1), p.58-58, Article 58

2013

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Searching for tryptase in the RBL‐2H3 mast cell model: Preparation for comparative mast cell toxicology studies with zebrafish

Journal of applied toxicology, 2019-03, Vol.39 (3), p.473-484

2019

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Nanoscale imaging of molecular positions and anisotropies

Nature methods, 2008-12, Vol.5 (12), p.1027-1030

2008

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Arsenic Disruption of Steroid Receptor Gene Activation: Complex Dose−Response Effects Are Shared by Several Steroid Receptors

Chemical research in toxicology, 2006-12, Vol.19 (12), p.1619-1629

2006

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Antimicrobial agent triclosan suppresses mast cell signaling via phospholipase D inhibition

Journal of applied toxicology, 2019-12, Vol.39 (12), p.1672-1690

2019

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Monomethylated trivalent arsenic species disrupt steroid receptor interactions with their DNA response elements at non-cytotoxic cellular concentrations

Journal of applied toxicology, 2014-05, Vol.34 (5), p.498-505

2014

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Activation of Hedgehog Signaling by the Environmental Toxicant Arsenic May Contribute to the Etiology of Arsenic-Induced Tumors

Cancer research (Chicago, Ill.), 2010-03, Vol.70 (5), p.1981-1988

2010

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A microplate assay to assess chemical effects on RBL-2H3 mast cell degranulation: effects of triclosan without use of an organic solvent

Journal of visualized experiments, 2013-11 (81), p.e50671-e50671

2013

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Actin Mediates the Nanoscale Membrane Organization of the Clustered Membrane Protein Influenza Hemagglutinin

Antimicrobial agent triclosan is a proton ionophore uncoupler of mitochondria in living rat and human mast cells and in primary human keratinocytes

Arsenic as an Endocrine Disruptor: Effects of Arsenic on Estrogen Receptor–Mediated Gene Expression In Vivo and in Cell Culture

Phosphatidylinositol 4,5-Bisphosphate Mediates the Co-Distribution of Influenza A Hemagglutinin and Matrix Protein M1 at the Plasma Membrane

Cetylpyridinium chloride (CPC) reduces zebrafish mortality from influenza infection: Super-resolution microscopy reveals CPC interference with multiple protein interactions with phosphatidylinositol 4,5-bisphosphate in immune function

Triclosan exposure, transformation, and human health effects

Elevated nitrate alters the metabolic activity of embryonic zebrafish

Chronic Exposure to Arsenic in the Drinking Water Alters the Expression of Immune Response Genes in Mouse Lung

Visualizing the Molecular Timing of a Physiological Decision at the Nanoscale

Temporally resolved interactions between antigen-stimulated IgE receptors and Lyn kinase on living cells

Lipid segregation and IgE receptor signaling: A decade of progress

Antimicrobial cetylpyridinium chloride causes functional inhibition of mitochondria as potently as canonical mitotoxicants, nanostructural disruption of mitochondria, and mitochondrial Ca 2+ efflux in living rodent and primary human cells

Pharmaceutical agent cetylpyridinium chloride inhibits immune mast cell function by interfering with calcium mobilization

Antimicrobial agent triclosan disrupts mitochondrial structure, revealed by super-resolution microscopy, and inhibits mast cell signaling via calcium modulation

Antimicrobial cetylpyridinium chloride causes functional inhibition of mitochondria as potently as canonical mitotoxicants, nanostructural disruption of mitochondria, and mitochondrial Ca2+ efflux in living rodent and primary human cells

Maternal responsivity and oxytocin in opioid‐dependent mothers

Triclosan disrupts immune cell function by depressing Ca2+ influx following acidification of the cytoplasm

Association between In Utero arsenic exposure, placental gene expression, and infant birth weight: a US birth cohort study

Searching for tryptase in the RBL‐2H3 mast cell model: Preparation for comparative mast cell toxicology studies with zebrafish

Nanoscale imaging of molecular positions and anisotropies

Arsenic Disruption of Steroid Receptor Gene Activation: Complex Dose−Response Effects Are Shared by Several Steroid Receptors

Antimicrobial agent triclosan suppresses mast cell signaling via phospholipase D inhibition

Monomethylated trivalent arsenic species disrupt steroid receptor interactions with their DNA response elements at non-cytotoxic cellular concentrations

Activation of Hedgehog Signaling by the Environmental Toxicant Arsenic May Contribute to the Etiology of Arsenic-Induced Tumors

A microplate assay to assess chemical effects on RBL-2H3 mast cell degranulation: effects of triclosan without use of an organic solvent

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