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The prevalence of quadriceps weakness in COPD and the relationship with disease severity

The European respiratory journal, 2010-07, Vol.36 (1), p.81-88

2010

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The mechanisms of cachexia underlying muscle dysfunction in COPD

Journal of applied physiology (1985), 2013-05, Vol.114 (9), p.1253-1262

2013

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TNF-α-Induced NF-κB Activation Stimulates Skeletal Muscle Glycolytic Metabolism Through Activation of HIF-1α

Endocrinology (Philadelphia), 2015-05, Vol.156 (5), p.1770-1781

2015

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Peroxisome proliferator-activated receptor expression is reduced in skeletal muscle in COPD

The European respiratory journal, 2007-08, Vol.30 (2), p.245-252

2007

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Cytokine profile in quadriceps muscles of patients with severe COPD

Thorax, 2008-02, Vol.63 (2), p.100-107

2008

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Differential sensitivity of oxidative and glycolytic muscles to hypoxia-induced muscle atrophy

Journal of applied physiology (1985), 2015-01, Vol.118 (2), p.200-211

2015

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Reduced mitochondrial density in the vastus lateralis muscle of patients with COPD

The European respiratory journal, 2007-07, Vol.30 (1), p.73-79

2007

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A novel technique for nonvolitional assessment of quadriceps muscle endurance in humans

Journal of applied physiology (1985), 2007-09, Vol.103 (3), p.739-746

2007

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Skeletal muscle fibre-type shifting and metabolic profile in patients with chronic obstructive pulmonary disease

The European respiratory journal, 2002-04, Vol.19 (4), p.617-625

2002

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Myopathological features in skeletal muscle of patients with chronic obstructive pulmonary disease

The European respiratory journal, 2003-08, Vol.22 (2), p.280-285

2003

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Triggers and mechanisms of skeletal muscle wasting in chronic obstructive pulmonary disease

The international journal of biochemistry & cell biology, 2013-10, Vol.45 (10), p.2245-2256

2013

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Peroxisome proliferator-activated receptors: a therapeutic target in COPD?

The European respiratory journal, 2008-03, Vol.31 (3), p.502-508

2008

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Skeletal muscle dysfunction in chronic obstructive pulmonary disease and chronic heart failure : underlying mechanisms and therapy perspectives

The American journal of clinical nutrition, 2000-05, Vol.71 (5), p.1033-1047

2000

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Regulation of PGC-1α expression by a GSK-3β-TFEB signaling axis in skeletal muscle

Biochimica et biophysica acta. Molecular cell research, 2020-02, Vol.1867 (2), p.118610, Article 118610

2020

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Inactivation of glycogen synthase kinase-3β (GSK-3β) enhances skeletal muscle oxidative metabolism

Biochimica et biophysica acta. Molecular basis of disease, 2017-12, Vol.1863 (12), p.3075-3086

2017

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Chemosensory function and food perception is affected in COPD, but unrelated to sarcopenia risk

Clinical nutrition (Edinburgh, Scotland), 2024-01, Vol.43 (1), p.218-223

2024

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Inactivation of glycogen synthase kinase 3β (GSK-3β) enhances mitochondrial biogenesis during myogenesis

Biochimica et biophysica acta. Molecular basis of disease, 2018-09, Vol.1864 (9), p.2913-2926

2018

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Regulation of mitochondrial biogenesis during myogenesis

Molecular and cellular endocrinology, 2010-02, Vol.315 (1), p.113-120

2010

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TNF-α impairs regulation of muscle oxidative phenotype: implications for cachexia?

The FASEB journal, 2010-12, Vol.24 (12), p.5052-5062

2010

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Molecular signalling towards mitochondrial breakdown is enhanced in skeletal muscle of patients with chronic obstructive pulmonary disease (COPD)

Scientific reports, 2018-10, Vol.8 (1), p.15007-13, Article 15007

2018

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Towards Personalized Management of Sarcopenia in COPD

International journal of chronic obstructive pulmonary disease, 2021-01, Vol.16, p.25-40

2021

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Regulation of skeletal muscle oxidative phenotype by classical NF-κB signalling

Biochimica et biophysica acta, 2013-08, Vol.1832 (8), p.1313-1325

2013

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Effect of chronic hypoxia on RAGE and its soluble forms in lungs and plasma of mice

Biochimica et biophysica acta, 2015-05, Vol.1852 (5), p.992-1000

2015

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Uncoupling protein-3 content is decreased in peripheral skeletal muscle of patients with COPD

The European respiratory journal, 2003-07, Vol.22 (1), p.88-93

2003

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Classical NF-κB activation impairs skeletal muscle oxidative phenotype by reducing IKK-α expression

Biochimica et biophysica acta, 2014-02, Vol.1842 (2), p.175-185

2014

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The prevalence of quadriceps weakness in COPD and the relationship with disease severity

The mechanisms of cachexia underlying muscle dysfunction in COPD

TNF-α-Induced NF-κB Activation Stimulates Skeletal Muscle Glycolytic Metabolism Through Activation of HIF-1α

Peroxisome proliferator-activated receptor expression is reduced in skeletal muscle in COPD

Cytokine profile in quadriceps muscles of patients with severe COPD

Differential sensitivity of oxidative and glycolytic muscles to hypoxia-induced muscle atrophy

Reduced mitochondrial density in the vastus lateralis muscle of patients with COPD

A novel technique for nonvolitional assessment of quadriceps muscle endurance in humans

Skeletal muscle fibre-type shifting and metabolic profile in patients with chronic obstructive pulmonary disease

Myopathological features in skeletal muscle of patients with chronic obstructive pulmonary disease

Triggers and mechanisms of skeletal muscle wasting in chronic obstructive pulmonary disease

Peroxisome proliferator-activated receptors: a therapeutic target in COPD?

Skeletal muscle dysfunction in chronic obstructive pulmonary disease and chronic heart failure : underlying mechanisms and therapy perspectives

Regulation of PGC-1α expression by a GSK-3β-TFEB signaling axis in skeletal muscle

Inactivation of glycogen synthase kinase-3β (GSK-3β) enhances skeletal muscle oxidative metabolism

Chemosensory function and food perception is affected in COPD, but unrelated to sarcopenia risk

Inactivation of glycogen synthase kinase 3β (GSK-3β) enhances mitochondrial biogenesis during myogenesis

Regulation of mitochondrial biogenesis during myogenesis

TNF-α impairs regulation of muscle oxidative phenotype: implications for cachexia?

Molecular signalling towards mitochondrial breakdown is enhanced in skeletal muscle of patients with chronic obstructive pulmonary disease (COPD)

Towards Personalized Management of Sarcopenia in COPD

Regulation of skeletal muscle oxidative phenotype by classical NF-κB signalling

Effect of chronic hypoxia on RAGE and its soluble forms in lungs and plasma of mice

Uncoupling protein-3 content is decreased in peripheral skeletal muscle of patients with COPD

Classical NF-κB activation impairs skeletal muscle oxidative phenotype by reducing IKK-α expression

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