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Macrophage-Orbital Fibroblast Interaction and Hypoxia Promote Inflammation and Adipogenesis in Graves' Orbitopathy

Endocrinology (Philadelphia), 2022-12, Vol.164 (2)

2022

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Comparative Assessment of Female Mouse Model of Graves' Orbitopathy Under Different Environments, Accompanied by Proinflammatory Cytokine and T-Cell Responses to Thyrotropin Hormone Receptor Antigen

Endocrinology (Philadelphia), 2016-04, Vol.157 (4), p.1673-1682

2016

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Sphingolipids in thyroid eye disease

Frontiers in endocrinology (Lausanne), 2023-04, Vol.14, p.1170884-1170884

2023

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Graves' orbitopathy occurs sex-independently in an autoimmune hyperthyroid mouse model

Scientific reports, 2018-08, Vol.8 (1), p.13096-13, Article 13096

2018

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Pathogenic Phenotype of Adipogenesis and Hyaluronan in Orbital Fibroblasts From Female Graves' Orbitopathy Mouse Model

Endocrinology (Philadelphia), 2016-10, Vol.157 (10), p.3771-3778

2016

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Linsitinib, an IGF-1R inhibitor, attenuates disease development and progression in a model of thyroid eye disease

Frontiers in endocrinology (Lausanne), 2023-06, Vol.14, p.1211473-1211473

2023

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Potential involvement of the bone marrow in experimental Graves’ disease and thyroid eye disease

Frontiers in endocrinology (Lausanne), 2023-09, Vol.14, p.1252727-1252727

2023

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Cigarette smoke causes a bioenergetic crisis in RPE cells involving the downregulation of HIF-1α under normoxia

Cell death discovery, 2023-10, Vol.9 (1), p.398-398, Article 398

2023

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Hypoxia-Dependent HIF-1 Activation Impacts on Tissue Remodeling in Graves' Ophthalmopathy—Implications for Smoking

The journal of clinical endocrinology and metabolism, 2016-12, Vol.101 (12), p.4834-4842

2016

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An Early Wave of Macrophage Infiltration Intertwined with Antigen-Specific Proinflammatory T Cells and Browning of Adipose Tissue Characterizes the Onset of Orbital Inflammation in a Mouse Model of Graves' Orbitopathy

Thyroid (New York, N.Y.), 2022-03, Vol.32 (3), p.283

2022

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Current Therapeutic Approaches for Graves' Orbitopathy - are Targeted Therapies the Future?

Klinische Monatsblatter fur Augenheilkunde, 2024-01, Vol.241 (1), p.48

2024

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Orbital Fibroblasts From Graves' Orbitopathy Patients Share Functional and Immunophenotypic Properties With Mesenchymal Stem/Stromal Cells

Investigative ophthalmology & visual science, 2015-10, Vol.56 (11), p.6549-6557

2015

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Macrophage-Orbital Fibroblast Interaction and Hypoxia Promote Inflammation and Adipogenesis in Graves' Orbitopathy

Endocrinology (Philadelphia), 2023-02, Vol.164 (2), p.1

2023

Volltextzugriff (PDF)

OR26-03 Treatment With Linsitinib, An IGF-1 Receptor Inhibitor, Prevents Disease Development And Progression In An Experimental Model Of Thyroid Eye Disease

Journal of the Endocrine Society, 2023-10, Vol.7 (Supplement_1)

2023

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Role of orbital fibroblasts in tissue remodeling of graves' orbitopathy - involvement of hypoxia dependent and independent pathways

Görtz, Gina-Eva

2017

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Macrophage-Orbital Fibroblast Interaction and Hypoxia Promote Inflammation and Adipogenesis in Graves' Orbitopathy

Comparative Assessment of Female Mouse Model of Graves' Orbitopathy Under Different Environments, Accompanied by Proinflammatory Cytokine and T-Cell Responses to Thyrotropin Hormone Receptor Antigen

Sphingolipids in thyroid eye disease

Graves' orbitopathy occurs sex-independently in an autoimmune hyperthyroid mouse model

Pathogenic Phenotype of Adipogenesis and Hyaluronan in Orbital Fibroblasts From Female Graves' Orbitopathy Mouse Model

Linsitinib, an IGF-1R inhibitor, attenuates disease development and progression in a model of thyroid eye disease

Potential involvement of the bone marrow in experimental Graves’ disease and thyroid eye disease

Cigarette smoke causes a bioenergetic crisis in RPE cells involving the downregulation of HIF-1α under normoxia

Hypoxia-Dependent HIF-1 Activation Impacts on Tissue Remodeling in Graves' Ophthalmopathy—Implications for Smoking

An Early Wave of Macrophage Infiltration Intertwined with Antigen-Specific Proinflammatory T Cells and Browning of Adipose Tissue Characterizes the Onset of Orbital Inflammation in a Mouse Model of Graves' Orbitopathy

Current Therapeutic Approaches for Graves' Orbitopathy - are Targeted Therapies the Future?

Orbital Fibroblasts From Graves' Orbitopathy Patients Share Functional and Immunophenotypic Properties With Mesenchymal Stem/Stromal Cells

Macrophage-Orbital Fibroblast Interaction and Hypoxia Promote Inflammation and Adipogenesis in Graves' Orbitopathy

OR26-03 Treatment With Linsitinib, An IGF-1 Receptor Inhibitor, Prevents Disease Development And Progression In An Experimental Model Of Thyroid Eye Disease

Role of orbital fibroblasts in tissue remodeling of graves' orbitopathy - involvement of hypoxia dependent and independent pathways

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