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Goodbye to the bioassay

Toxicology research (Cambridge), 2018-07, Vol.7 (4), p.558-564

Goodman, Jay I

2018

Volltextzugriff (PDF)

Cellular quality control during gametogenesis

Experimental cell research, 2020-11, Vol.396 (1), p.112247-112247, Article 112247

2020

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In Memoriam: Jerry B. Hook (1938 –2021)

Toxicological sciences, 2021-04, Vol.180 (2), p.197-197

2021

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Meiotic cellular rejuvenation is coupled to nuclear remodeling in budding yeast

eLife, 2019-08, Vol.8

2019

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Mode of action and human relevance analysis for nuclear receptor-mediated liver toxicity: A case study with phenobarbital as a model constitutive androstane receptor (CAR) activator

Critical reviews in toxicology, 2014-01, Vol.44 (1), p.64-82

2014

Volltextzugriff (PDF)

sNASP and ASF1A function through both competitive and compatible modes of histone binding

Nucleic acids research, 2017-01, Vol.45 (2), p.643

2017

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Correction: Goodbye to the bioassay

Toxicology research (Cambridge), 2018-09, Vol.7 (5), p.994-994

Goodman, Jay I

2018

Volltextzugriff (PDF)

A High Through-put Platform for Recombinant Antibodies to Folded Proteins

Molecular & cellular proteomics, 2015-10, Vol.14 (10), p.2833-2847

2015

Volltextzugriff (PDF)

Categorizing the characteristics of human carcinogens: a need for specificity

Archives of toxicology, 2021-08, Vol.95 (8), p.2883-2889

2021

Volltextzugriff (PDF)

Incorporation of an epigenetic evaluation into safety assessment: What we first need to know

Current opinion in toxicology, 2017-04, Vol.3, p.20-24

Goodman, Jay I.

2017

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Computational modeling identifies key gene regulatory interactions underlying phenobarbital-mediated tumor promotion

Nucleic acids research, 2014-04, Vol.42 (7), p.4180

2014

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Phenobarbital and propiconazole toxicogenomic profiles in mice show major similarities consistent with the key role that constitutive androstane receptor (CAR) activation plays in their mode of action

Toxicology (Amsterdam), 2014-07, Vol.321, p.80-88

2014

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The Constitutive Active/Androstane Receptor Facilitates Unique Phenobarbital-Induced Expression Changes of Genes Involved in Key Pathways in Precancerous Liver and Liver Tumors

Toxicological sciences, 2009-08, Vol.110 (2), p.319-333

2009

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Response to Druwe and Burgoon, 2016 Letter to the Editor in Archives of Toxicology

Archives of toxicology, 2017-02, Vol.91 (2), p.999-1000

2017

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Antibiotic susceptibilities of bacteria isolated within the oral flora of Florida blacktip sharks: guidance for empiric antibiotic therapy

PloS one, 2014-08, Vol.9 (8), p.e104577-e104577

2014

Volltextzugriff (PDF)

Response to Druwe and Burgoon: Re: Druwe, I.L. and Burgoon, L.: revisiting Cohen et al. 2015, Cohen et al. 2014 and Waalkes et al. 2014: a bayesian re-analysis of tumor incidences

Archives of toxicology, 2016-12, Vol.90 (12), p.3129-3130

2016

Volltextzugriff (PDF)

Multiple Genes Exhibit Phenobarbital-Induced Constitutive Active/Androstane Receptor–Mediated DNA Methylation Changes during Liver Tumorigenesis and in Liver Tumors

Toxicological sciences, 2009-04, Vol.108 (2), p.273-289

2009

Volltextzugriff (PDF)

Re: Waalkes et al.: Lung tumors in mice induced by “whole-life” inorganic arsenic exposure at human-relevant doses, Arch Toxicol, 2014

Archives of toxicology, 2014-11, Vol.88 (11), p.2061-2062

2014

Volltextzugriff (PDF)

Identification of Genes that May Play Critical Roles in Phenobarbital (PB)-Induced Liver Tumorigenesis due to Altered DNA Methylation

Toxicological sciences, 2008-07, Vol.104 (1), p.86-99

2008

Volltextzugriff (PDF)

Phenobarbital Elicits Unique, Early Changes in the Expression of Hepatic Genes that Affect Critical Pathways in Tumor-Prone B6C3F1 Mice

Toxicological sciences, 2009-06, Vol.109 (2), p.193-205

2009

Volltextzugriff (PDF)

Changes in DNA methylation and gene expression during 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced suppression of the lipopolysaccharide-stimulated IgM response in splenocytes

Toxicological sciences, 2011-04, Vol.120 (2), p.339-348

2011

Volltextzugriff (PDF)

Illustrative case using the RISK21 roadmap and matrix: prioritization for evaluation of chemicals found in drinking water

Critical reviews in toxicology, 2016-01, Vol.46 (1), p.43-53

2016

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What do we need to know prior to thinking about incorporating an epigenetic evaluation into safety assessments?

Toxicological sciences, 2010-08, Vol.116 (2), p.375

2010

Volltextzugriff (PDF)

Arteriovenous malformation identification after conservative management of placenta percreta with uterine artery embolization and adjunctive therapy

American journal of obstetrics and gynecology, 2011-05, Vol.204 (5), p.e4-e8

2011

Volltextzugriff (PDF)

Inhalation of cigarette smoke induces regions of altered DNA methylation (RAMs) in SENCAR mouse lung

Toxicology (Amsterdam), 2009-06, Vol.260 (1), p.7-15

2009

Volltextzugriff (PDF)

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Goodbye to the bioassay

Cellular quality control during gametogenesis

In Memoriam: Jerry B. Hook (1938 –2021)

Meiotic cellular rejuvenation is coupled to nuclear remodeling in budding yeast

Mode of action and human relevance analysis for nuclear receptor-mediated liver toxicity: A case study with phenobarbital as a model constitutive androstane receptor (CAR) activator

sNASP and ASF1A function through both competitive and compatible modes of histone binding

Correction: Goodbye to the bioassay

A High Through-put Platform for Recombinant Antibodies to Folded Proteins

Categorizing the characteristics of human carcinogens: a need for specificity

Incorporation of an epigenetic evaluation into safety assessment: What we first need to know

Computational modeling identifies key gene regulatory interactions underlying phenobarbital-mediated tumor promotion

Phenobarbital and propiconazole toxicogenomic profiles in mice show major similarities consistent with the key role that constitutive androstane receptor (CAR) activation plays in their mode of action

The Constitutive Active/Androstane Receptor Facilitates Unique Phenobarbital-Induced Expression Changes of Genes Involved in Key Pathways in Precancerous Liver and Liver Tumors

Response to Druwe and Burgoon, 2016 Letter to the Editor in Archives of Toxicology

Antibiotic susceptibilities of bacteria isolated within the oral flora of Florida blacktip sharks: guidance for empiric antibiotic therapy

Response to Druwe and Burgoon: Re: Druwe, I.L. and Burgoon, L.: revisiting Cohen et al. 2015, Cohen et al. 2014 and Waalkes et al. 2014: a bayesian re-analysis of tumor incidences

Multiple Genes Exhibit Phenobarbital-Induced Constitutive Active/Androstane Receptor–Mediated DNA Methylation Changes during Liver Tumorigenesis and in Liver Tumors

Re: Waalkes et al.: Lung tumors in mice induced by “whole-life” inorganic arsenic exposure at human-relevant doses, Arch Toxicol, 2014

Identification of Genes that May Play Critical Roles in Phenobarbital (PB)-Induced Liver Tumorigenesis due to Altered DNA Methylation

Phenobarbital Elicits Unique, Early Changes in the Expression of Hepatic Genes that Affect Critical Pathways in Tumor-Prone B6C3F1 Mice

Changes in DNA methylation and gene expression during 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced suppression of the lipopolysaccharide-stimulated IgM response in splenocytes

Illustrative case using the RISK21 roadmap and matrix: prioritization for evaluation of chemicals found in drinking water

What do we need to know prior to thinking about incorporating an epigenetic evaluation into safety assessments?

Arteriovenous malformation identification after conservative management of placenta percreta with uterine artery embolization and adjunctive therapy

Inhalation of cigarette smoke induces regions of altered DNA methylation (RAMs) in SENCAR mouse lung

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