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Human cytomegalovirus infection is associated with increased expression of the lissencephaly gene PAFAH1B1 encoding LIS1 in neural stem cells and congenitally infected brains

The Journal of pathology, 2021-05, Vol.254 (1), p.92-102, Article path.5640

2021

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Fatal encephalitis and Borna Disease Virus‐1 seropositivity in two kidney‐transplant patients living in the same nonendemic area

Transplant infectious disease, 2021-12, Vol.23 (6), p.e13734-n/a

2021

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Manipulation of the N‐terminal sequence of the Borna disease virus X protein improves its mitochondrial targeting and neuroprotective potential

The FASEB journal, 2016-04, Vol.30 (4), p.1523-1533

2016

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HSPA9/Mortalin mediates axo-protection and modulates mitochondrial dynamics in neurons

Scientific reports, 2021-09, Vol.11 (1), p.17705-17705, Article 17705

2021

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PPARγ Is Activated during Congenital Cytomegalovirus Infection and Inhibits Neuronogenesis from Human Neural Stem Cells

PLoS pathogens, 2016-04, Vol.12 (4), p.e1005547-e1005547

2016

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Effect of tryptase inhibition on joint inflammation: a pharmacological and lentivirus-mediated gene transfer study

Arthritis research & therapy, 2017-06, Vol.19 (1), p.124-124, Article 124

2017

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Neurons are MHC class I-dependent targets for CD8 T cells upon neurotropic viral infection

PLoS pathogens, 2011-11, Vol.7 (11), p.e1002393-e1002393

2011

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Borna disease virus phosphoprotein modulates epigenetic signaling in neurons to control viral replication

Journal of virology, 2015-06, Vol.89 (11), p.5996-6008

2015

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Borna Disease Virus 1 Phosphoprotein Forms a Tetramer and Interacts with Host Factors Involved in DNA Double-Strand Break Repair and mRNA Processing

Viruses, 2022-10, Vol.14 (11), p.2358

2022

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Analysis of Borna Disease Virus Trafficking in Live Infected Cells by Using a Virus Encoding a Tetracysteine-Tagged P Protein

Journal of Virology, 2013-11, Vol.87 (22), p.12339-12348

2013

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Borna disease virus phosphoprotein impairs the developmental program controlling neurogenesis and reduces human GABAergic neurogenesis

PLoS pathogens, 2015-04, Vol.11 (4), p.e1004859-e1004859

2015

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Borna disease virus docks on neuronal DNA double-strand breaks to replicate and dampens neuronal activity

iScience, 2022-01, Vol.25 (1), p.103621-103621, Article 103621

2022

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Production and purification of non replicative canine adenovirus type 2 derived vectors

Journal of visualized experiments, 2013-12 (82), p.50833-50833

2013

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Borna disease virus blocks potentiation of presynaptic activity through inhibition of protein kinase C signaling

PLoS pathogens, 2006-03, Vol.2 (3), p.e19-e19

2006

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Anaplastic Lymphoma Kinase Is a Dependence Receptor Whose Proapoptotic Functions Are Activated by Caspase Cleavage

Molecular and Cellular Biology, 2006-08, Vol.26 (16), p.6209-6222

2006

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Human Cytomegalovirus Infection Changes the Pattern of Surface Markers of Small Extracellular Vesicles Isolated From First Trimester Placental Long-Term Histocultures

Frontiers in cell and developmental biology, 2021-09, Vol.9 (689122), p.689122-689122

2021

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Proteomic Analysis Reveals Selective Impediment of Neuronal Remodeling upon Borna Disease Virus Infection

Journal of Virology, 2008-12, Vol.82 (24), p.12265-12279

2008

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Mutation of the protein kinase C site in borna disease virus phosphoprotein abrogates viral interference with neuronal signaling and restores normal synaptic activity

PLoS pathogens, 2009-05, Vol.5 (5), p.e1000425-e1000425

2009

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Borna Disease Virus Infection Impairs Synaptic Plasticity

Journal of Virology, 2007-08, Vol.81 (16), p.8833-8837

2007

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Borna Disease Virus Glycoprotein Is Required for Viral Dissemination in Neurons

Journal of Virology, 2003-11, Vol.77 (22), p.12222-12231

2003

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Protein kinase C-dependent phosphorylation of Borna disease virus P protein is required for efficient viral spread

Archives of virology, 2010-05, Vol.155 (5), p.789-793

2010

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Nanowire based bioprobes for electrical monitoring of electrogenic cells

Journal of physics. Condensed matter, 2018-11, Vol.30 (46), p.464001-464001

2018

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Synaptic Pathology in Borna Disease Virus Persistent Infection

Journal of Virology, 2000-04, Vol.74 (8), p.3441-3448

2000

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Mechanism of Borna disease virus entry into cells

Journal of Virology, 1998-01, Vol.72 (1), p.783-788

1998

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Borna Disease Virus Persistent Infection Activates Mitogen-activated Protein Kinase and Blocks Neuronal Differentiation of PC12 Cells

The Journal of biological chemistry, 2001-03, Vol.276 (10), p.7258-7265

2001

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Human cytomegalovirus infection is associated with increased expression of the lissencephaly gene PAFAH1B1 encoding LIS1 in neural stem cells and congenitally infected brains

Fatal encephalitis and Borna Disease Virus‐1 seropositivity in two kidney‐transplant patients living in the same nonendemic area

Manipulation of the N‐terminal sequence of the Borna disease virus X protein improves its mitochondrial targeting and neuroprotective potential

HSPA9/Mortalin mediates axo-protection and modulates mitochondrial dynamics in neurons

PPARγ Is Activated during Congenital Cytomegalovirus Infection and Inhibits Neuronogenesis from Human Neural Stem Cells

Effect of tryptase inhibition on joint inflammation: a pharmacological and lentivirus-mediated gene transfer study

Neurons are MHC class I-dependent targets for CD8 T cells upon neurotropic viral infection

Borna disease virus phosphoprotein modulates epigenetic signaling in neurons to control viral replication

Borna Disease Virus 1 Phosphoprotein Forms a Tetramer and Interacts with Host Factors Involved in DNA Double-Strand Break Repair and mRNA Processing

Analysis of Borna Disease Virus Trafficking in Live Infected Cells by Using a Virus Encoding a Tetracysteine-Tagged P Protein

Borna disease virus phosphoprotein impairs the developmental program controlling neurogenesis and reduces human GABAergic neurogenesis

Borna disease virus docks on neuronal DNA double-strand breaks to replicate and dampens neuronal activity

Production and purification of non replicative canine adenovirus type 2 derived vectors

Borna disease virus blocks potentiation of presynaptic activity through inhibition of protein kinase C signaling

Anaplastic Lymphoma Kinase Is a Dependence Receptor Whose Proapoptotic Functions Are Activated by Caspase Cleavage

Human Cytomegalovirus Infection Changes the Pattern of Surface Markers of Small Extracellular Vesicles Isolated From First Trimester Placental Long-Term Histocultures

Proteomic Analysis Reveals Selective Impediment of Neuronal Remodeling upon Borna Disease Virus Infection

Mutation of the protein kinase C site in borna disease virus phosphoprotein abrogates viral interference with neuronal signaling and restores normal synaptic activity

Borna Disease Virus Infection Impairs Synaptic Plasticity

Borna Disease Virus Glycoprotein Is Required for Viral Dissemination in Neurons

Protein kinase C-dependent phosphorylation of Borna disease virus P protein is required for efficient viral spread

Nanowire based bioprobes for electrical monitoring of electrogenic cells

Synaptic Pathology in Borna Disease Virus Persistent Infection

Mechanism of Borna disease virus entry into cells

Borna Disease Virus Persistent Infection Activates Mitogen-activated Protein Kinase and Blocks Neuronal Differentiation of PC12 Cells

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