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Prostaglandin I2 Receptor (IP) Signaling Inhibits Antigen Uptake but Induce the Ability of Migration on Immature Dendritic Cells

Journal of allergy and clinical immunology, 2012-02, Vol.129 (2), p.AB161-AB161

2012

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Testosterone Attenuates Group 2 Innate Lymphoid Cell-Mediated Airway Inflammation

Cell reports (Cambridge), 2017-11, Vol.21 (9), p.2487-2499

2017

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IL-17A-mediated STAT6 Independent Mucus Production in Mice

Journal of allergy and clinical immunology, 2012-02, Vol.129 (2), p.AB199-AB199

2012

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Attenuated Allergic Airway Inflammation in gp91phox KO Mice is Due to Altered Dendritic Cell Function

Journal of allergy and clinical immunology, 2011-02, Vol.127 (2), p.AB259-AB259

2011

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Prostaglandin I2 Receptor (IP) Signaling Inhibits TLR4 Cell Surface Expression and Reduces LPS-induced Lung Inflammation

Journal of allergy and clinical immunology, 2011-02, Vol.127 (2), p.AB92-AB92

2011

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Il-13 Receptor Alpha 1 Is Expressed On T Cells In Vivo And Il-13 Regulates RSV-Induced Il-17A Production In STAT1-/- Mice

Journal of allergy and clinical immunology, 2011, Vol.127 (2), p.AB263-AB263

2011

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TGF-β, IL-1β, And IL-23 Are Required For IL-13Rα1 Expression On Human Th17 Cells And IL-13 Attenuates IL-17A Production At Restimulation

Journal of allergy and clinical immunology, 2011, Vol.127 (2), p.AB68-AB68

2011

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Prostaglandin I2 Signaling Exacerbates Experimental Autoimmune Encephalomyelitis

Journal of allergy and clinical immunology, 2011-02, Vol.127 (2), p.AB232-AB232

2011

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The Deficiency of PGI2 Signaling Induces STAT6-independent Allergic Airway Inflammation

Journal of allergy and clinical immunology, 2011-02, Vol.127 (2), p.AB57-AB57

2011

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Human CD4+ Th17 Cells Express A Functional IL-13 Receptor And IL-13 Signaling Negatively Regulates IL-17A Production

Journal of allergy and clinical immunology, 2010-02, Vol.125 (2), p.AB117-AB117

2010

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IL-13 Decreases IL-17a Production In Mouse Th17 Cells Through An IL-10 Dependent Mechanism

Journal of allergy and clinical immunology, 2011-02, Vol.127 (2), p.AB69-AB69

2011

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Signaling through the PGI2 Receptor Promotes IL-17 Production by CD4 T Cells

Journal of allergy and clinical immunology, 2010-02, Vol.125 (2), p.AB143-AB143

2010

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Lack of p47phox , the Active Subunit of the NADPH Oxidase Complex, Augments Ovalbumin-induced Allergic Airway Inflammation in Mice

Journal of allergy and clinical immunology, 2010-02, Vol.125 (2), p.AB105-AB105

2010

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PGE2 in Synergy with IL-23 Drives Robust IL-17 Production and Th17 Differentiation by Inhibiting the STAT6 Signaling Pathway

Journal of allergy and clinical immunology, 2010-02, Vol.125 (2), p.AB117-AB117

2010

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PGE2 And EP2 Receptor Signaling Suppress IL-17 Production By Polarized Mouse Th17 Cells

Journal of allergy and clinical immunology, 2009-02, Vol.123 (2), p.S91-S91

2009

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A Functional IL-13 Receptor is Expressed on Polarized Murine CD4 + Th17 Cells and IL-13 Signaling Negatively Regulates Th17 Cytokine Production

Journal of allergy and clinical immunology, 2009-02, Vol.123 (2), p.S138-S138

2009

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STAT1 Negatively Regulates Basophil IL-4 Expression Induced by Respiratory Syncytial Vrus Infection

Journal of allergy and clinical immunology, 2009-02, Vol.123 (2), p.S153-S153

2009

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Respiratory Syncytial Virus Infections Augment Allergen-Specific T Cell Secretion of Lung IL-17A

Journal of allergy and clinical immunology, 2008-02, Vol.121 (2), p.S135-S135

2008

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A Clinical Isolate Strain of Respiratory Syncytial Virus (RSV) Causes Airway Obstruction and Histopathology in Mice Similar to Severe RSV Disease in Infants

Journal of allergy and clinical immunology, 2008-02, Vol.121 (2), p.S268-S268

2008

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Natural Killer (NK) Cells, Not T Cells, Produce the Cytokine IL-4 in Response to Primary Respiratory Syncytial Virus Infection

Journal of allergy and clinical immunology, 2007-02, Vol.119 (2), p.523-523

2007

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C29 SENSATIONS IN THE LUNG: COUGHING AND CONSTRICTING: Small Heat Shock Protein 20 Phospho Peptide Inhibits Allergen-Induced Airway Responsiveness To Methacholine

American journal of respiratory and critical care medicine, 2015-01, Vol.191, p.1

2015

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Respiratory Syncytial Virus-Induced Airway Dysfunction Depends on the Virus Strain

Journal of Allergy and Clinical Immunology, 2006-02, Vol.117 (2), p.S206-S206

2006

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Respiratory Syncytial Virus in Allergic Lung Inflammation Increases Muc5ac and Gob-5

American journal of respiratory and critical care medicine, 2004-08, Vol.170 (3), p.306-312

2004

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Prostaglandin I 2 Receptor (IP) Signaling Inhibits TLR4 Cell Surface Expression and Reduces LPS-induced Lung Inflammation

Journal of allergy and clinical immunology, 2011, Vol.127 (2), p.AB92-AB92

2011

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Respiratory syncytial virus infection in the absence of STAT1 results in airway dysfunction, airway mucus, and augmented IL-17 levels

Journal of Allergy and Clinical Immunology, 2005-09, Vol.116 (3), p.550-557

2005

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Prostaglandin I2 Receptor (IP) Signaling Inhibits Antigen Uptake but Induce the Ability of Migration on Immature Dendritic Cells

Testosterone Attenuates Group 2 Innate Lymphoid Cell-Mediated Airway Inflammation

IL-17A-mediated STAT6 Independent Mucus Production in Mice

Attenuated Allergic Airway Inflammation in gp91phox KO Mice is Due to Altered Dendritic Cell Function

Prostaglandin I2 Receptor (IP) Signaling Inhibits TLR4 Cell Surface Expression and Reduces LPS-induced Lung Inflammation

Il-13 Receptor Alpha 1 Is Expressed On T Cells In Vivo And Il-13 Regulates RSV-Induced Il-17A Production In STAT1-/- Mice

TGF-β, IL-1β, And IL-23 Are Required For IL-13Rα1 Expression On Human Th17 Cells And IL-13 Attenuates IL-17A Production At Restimulation

Prostaglandin I2 Signaling Exacerbates Experimental Autoimmune Encephalomyelitis

The Deficiency of PGI2 Signaling Induces STAT6-independent Allergic Airway Inflammation

Human CD4+ Th17 Cells Express A Functional IL-13 Receptor And IL-13 Signaling Negatively Regulates IL-17A Production

IL-13 Decreases IL-17a Production In Mouse Th17 Cells Through An IL-10 Dependent Mechanism

Signaling through the PGI2 Receptor Promotes IL-17 Production by CD4 T Cells

Lack of p47phox , the Active Subunit of the NADPH Oxidase Complex, Augments Ovalbumin-induced Allergic Airway Inflammation in Mice

PGE2 in Synergy with IL-23 Drives Robust IL-17 Production and Th17 Differentiation by Inhibiting the STAT6 Signaling Pathway

PGE2 And EP2 Receptor Signaling Suppress IL-17 Production By Polarized Mouse Th17 Cells

A Functional IL-13 Receptor is Expressed on Polarized Murine CD4 + Th17 Cells and IL-13 Signaling Negatively Regulates Th17 Cytokine Production

STAT1 Negatively Regulates Basophil IL-4 Expression Induced by Respiratory Syncytial Vrus Infection

Respiratory Syncytial Virus Infections Augment Allergen-Specific T Cell Secretion of Lung IL-17A

A Clinical Isolate Strain of Respiratory Syncytial Virus (RSV) Causes Airway Obstruction and Histopathology in Mice Similar to Severe RSV Disease in Infants

Natural Killer (NK) Cells, Not T Cells, Produce the Cytokine IL-4 in Response to Primary Respiratory Syncytial Virus Infection

C29 SENSATIONS IN THE LUNG: COUGHING AND CONSTRICTING: Small Heat Shock Protein 20 Phospho Peptide Inhibits Allergen-Induced Airway Responsiveness To Methacholine

Respiratory Syncytial Virus-Induced Airway Dysfunction Depends on the Virus Strain

Respiratory Syncytial Virus in Allergic Lung Inflammation Increases Muc5ac and Gob-5

Prostaglandin I 2 Receptor (IP) Signaling Inhibits TLR4 Cell Surface Expression and Reduces LPS-induced Lung Inflammation

Respiratory syncytial virus infection in the absence of STAT1 results in airway dysfunction, airway mucus, and augmented IL-17 levels

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