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Inhibition of HSP27 alone or in combination with pAKT inhibition as therapeutic approaches to target SPARC-induced glioma cell survival

Molecular cancer, 2012-04, Vol.11 (1), p.20-20, Article 20

2012

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PTEN suppresses SPARC-induced pMAPKAPK2 and inhibits SPARC-induced Ser78 HSP27 phosphorylation in glioma

Neuro-oncology (Charlottesville, Va.), 2013-04, Vol.15 (4), p.451-461

2013

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Schwann cell-derived exosomes ameliorate peripheral neuropathy induced by ablation of dicer in Schwann cells

Frontiers in cellular neuroscience, 2024-09, Vol.18, p.1462228

2024

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HSP27 mediates SPARC-induced changes in glioma morphology, migration, and invasion

Glia, 2008-08, Vol.56 (10), p.1061-1075

2008

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Delayed (21 Days) Post Stroke Treatment With RPh201, a Botany-Derived Compound, Improves Neurological Functional Recovery in a Rat Model of Embolic Stroke

Frontiers in neuroscience, 2020-07, Vol.14, p.813-813

2020

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Secreted protein acidic and rich in cysteine promotes glioma invasion and delays tumor growth in vivo

Cancer research (Chicago, Ill.), 2002-11, Vol.62 (21), p.6270-6277

2002

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Loss of Sparc in p53-null Astrocytes Promotes Macrophage Activation and Phagocytosis Resulting in Decreased Tumor Size and Tumor Cell Survival

Brain pathology (Zurich, Switzerland), 2015-07, Vol.25 (4), p.391-400

2015

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Deletion of the SPARC acidic domain or EGF-like module reduces SPARC-induced migration and signaling through p38 MAPK/HSP27 in glioma

Carcinogenesis (New York), 2012-02, Vol.33 (2), p.275-284

2012

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PTEN suppresses SPARC-induced pMAPKAPK2 and inhibits SPARC-induced Ser78 HSP27 phosphorylation in glioma

Neuro-oncology (Charlottesville, Va.), 2013-04, Vol.15 (4), p.451-461

2013

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LOSS OF SPARC IN P53-NULL ASTROCYTES PROMOTES MACROPHAGE ACTIVATION AND PHAGOCYTOSIS RESULTING IN DECREASED TUMOR SIZE AND TUMOR CELL SURVIVAL

Neuro-oncology (Charlottesville, Va.), 2014-07, Vol.16 (suppl 3), p.iii44-iii44

2014

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MiR-17-92 enriched exosomes derived from multipotent mesenchymal stromal cells enhance axon-myelin remodeling and motor electrophysiological recovery after stroke

Journal of cerebral blood flow and metabolism, 2021-05, Vol.41 (5), p.1131-1144

2021

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Abstract TP227: Identification Of Somatostatin (sst) Interneuron-specific Micrornas And Transcriptome Associated With Cerebral Ischemia

Stroke (1970), 2023-02, Vol.54 (Suppl_1)

2023

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Abstract TMP109: Labeling And Isolating Cell Specific Neuronal Mitochondria And Their Functional Analysis In Mice Post Stroke

Stroke (1970), 2023-02, Vol.54 (Suppl_1)

2023

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Targeted tPA overexpression in denervated spinal motor neurons promotes stroke recovery in mice

Journal of cerebral blood flow and metabolism, 2021-01, Vol.41 (1), p.92-104

2021

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Abstract 86: Dicer Ablation In Oligodendrocytes Impairs Oligodendrogenesis And Functional Recovery After Stroke

Stroke (1970), 2022-02, Vol.53 (Suppl_1)

2022

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SPARC: A Signal of Astrocytic Neoplastic Transformation and Reactive Response in Human Primary and Xenograft Gliomas

Journal of neuropathology and experimental neurology, 1998-12, Vol.57 (12), p.1112-1121

1998

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Abstract 36: Astrocyte-derived Small Extracellular Vesicles Mediate Astrocyte-neuron Communication To Promote Neurite Outgrowth After Stroke In Astrocyte-vesicle Reporter Mice

Stroke (1970), 2023-02, Vol.54 (Suppl_1)

2023

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Plasminogen deficiency causes reduced angiogenesis and behavioral recovery after stroke in mice

Journal of cerebral blood flow and metabolism, 2021-10, Vol.41 (10), p.2583-2592

2021

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cDNA array analysis of SPARC-modulated changes in glioma gene expression

Journal of neuro-oncology, 2002-12, Vol.60 (3), p.213

2002

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Cathepsin B expression and localization in glioma progression and invasion

Cancer research (Chicago, Ill.), 1994-12, Vol.54 (23), p.6027-6031

1994

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Abstract TP113: Delayed (21 Days) Post Stroke Treatment With RPh201, A Botany-Derived Compound, Improves Neurological Functional Recovery in a Rat Model of Embolic Stroke

Stroke (1970), 2020-02, Vol.51 (Suppl_1)

2020

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Abstract TMP36: miR-17-92 Cluster Enriched MSC Exosomes Promote Myelination and Axonal Extension Which Contribute to Increased Emg Conduction and Functional Recovery After Stroke in Rats

Stroke (1970), 2019-02, Vol.50 (Suppl_1)

2019

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Abstract 2363: Loss of Sparc in p53-null astrocytes alters collagen deposition and promotes macrophage activation and tumor phagocytosis

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.2363-2363

2015

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SPARC‐induced increase in glioma matrix and decrease in vascularity are associated with reduced VEGF expression and secretion

International journal of cancer, 2008-06, Vol.122 (12), p.2735-2743

2008

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Loss of Sparc in p 53 ‐null Astrocytes Promotes Macrophage Activation and Phagocytosis Resulting in Decreased Tumor Size and Tumor Cell Survival

Brain pathology (Zurich, Switzerland), 2015-07, Vol.25 (4), p.391-400

2015

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Inhibition of HSP27 alone or in combination with pAKT inhibition as therapeutic approaches to target SPARC-induced glioma cell survival

PTEN suppresses SPARC-induced pMAPKAPK2 and inhibits SPARC-induced Ser78 HSP27 phosphorylation in glioma

Schwann cell-derived exosomes ameliorate peripheral neuropathy induced by ablation of dicer in Schwann cells

HSP27 mediates SPARC-induced changes in glioma morphology, migration, and invasion

Delayed (21 Days) Post Stroke Treatment With RPh201, a Botany-Derived Compound, Improves Neurological Functional Recovery in a Rat Model of Embolic Stroke

Secreted protein acidic and rich in cysteine promotes glioma invasion and delays tumor growth in vivo

Loss of Sparc in p53-null Astrocytes Promotes Macrophage Activation and Phagocytosis Resulting in Decreased Tumor Size and Tumor Cell Survival

Deletion of the SPARC acidic domain or EGF-like module reduces SPARC-induced migration and signaling through p38 MAPK/HSP27 in glioma

PTEN suppresses SPARC-induced pMAPKAPK2 and inhibits SPARC-induced Ser78 HSP27 phosphorylation in glioma

LOSS OF SPARC IN P53-NULL ASTROCYTES PROMOTES MACROPHAGE ACTIVATION AND PHAGOCYTOSIS RESULTING IN DECREASED TUMOR SIZE AND TUMOR CELL SURVIVAL

MiR-17-92 enriched exosomes derived from multipotent mesenchymal stromal cells enhance axon-myelin remodeling and motor electrophysiological recovery after stroke

Abstract TP227: Identification Of Somatostatin (sst) Interneuron-specific Micrornas And Transcriptome Associated With Cerebral Ischemia

Abstract TMP109: Labeling And Isolating Cell Specific Neuronal Mitochondria And Their Functional Analysis In Mice Post Stroke

Targeted tPA overexpression in denervated spinal motor neurons promotes stroke recovery in mice

Abstract 86: Dicer Ablation In Oligodendrocytes Impairs Oligodendrogenesis And Functional Recovery After Stroke

SPARC: A Signal of Astrocytic Neoplastic Transformation and Reactive Response in Human Primary and Xenograft Gliomas

Abstract 36: Astrocyte-derived Small Extracellular Vesicles Mediate Astrocyte-neuron Communication To Promote Neurite Outgrowth After Stroke In Astrocyte-vesicle Reporter Mice

Plasminogen deficiency causes reduced angiogenesis and behavioral recovery after stroke in mice

cDNA array analysis of SPARC-modulated changes in glioma gene expression

Cathepsin B expression and localization in glioma progression and invasion

Abstract TP113: Delayed (21 Days) Post Stroke Treatment With RPh201, A Botany-Derived Compound, Improves Neurological Functional Recovery in a Rat Model of Embolic Stroke

Abstract TMP36: miR-17-92 Cluster Enriched MSC Exosomes Promote Myelination and Axonal Extension Which Contribute to Increased Emg Conduction and Functional Recovery After Stroke in Rats

Abstract 2363: Loss of Sparc in p53-null astrocytes alters collagen deposition and promotes macrophage activation and tumor phagocytosis

SPARC‐induced increase in glioma matrix and decrease in vascularity are associated with reduced VEGF expression and secretion

Loss of Sparc in p 53 ‐null Astrocytes Promotes Macrophage Activation and Phagocytosis Resulting in Decreased Tumor Size and Tumor Cell Survival

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