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From keratinocyte to cancer: the pathogenesis and modeling of cutaneous squamous cell carcinoma

The Journal of clinical investigation, 2012-02, Vol.122 (2), p.464-472

2012

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Forced Expression of the H11 Heat Shock Protein Can Be Regulated by DNA Methylation and Trigger Apoptosis in Human Cells

The Journal of biological chemistry, 2003-09, Vol.278 (39), p.37600-37609

2003

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Herpes simplex virus type 2 encodes a heat shock protein homologue with apoptosis regulatory functions

Frontiers in bioscience, 2005-09, Vol.10, p.2788

2005

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Human Natural Killer T Cells Infiltrate into the Skin at Elicitation Sites of Allergic Contact Dermatitis

Journal of investigative dermatology, 2008-06, Vol.128 (6), p.1460-1469

2008

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Voriconazole enhances UV‐induced DNA damage by inhibiting catalase and promoting oxidative stress

Experimental dermatology, 2020-01, Vol.29 (1), p.29-38

2020

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Reconstructing skin cancers using animal models

Cancer and metastasis reviews, 2013-06, Vol.32 (1-2), p.123-128

2013

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The Herpes simplex virus gene Pol expressed in herpes-associated erythema multiforme lesions upregulates/activates SP1 and inflammatory cytokines

Dermatology (Basel), 2007-01, Vol.215 (2), p.97

2007

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Intranasal Administration of the Growth-Compromised HSV-2 Vector ΔRR Prevents Kainate-Induced Seizures and Neuronal Loss in Rats and Mice

Molecular therapy, 2006-05, Vol.13 (5), p.870-881

2006

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Novel Homozygous Frameshift Mutation of EVER1 Gene in an Epidermodysplasia Verruciformis Patient

Journal of investigative dermatology, 2007-04, Vol.127 (4), p.817-820

2007

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The growth compromised HSV-2 mutant ΔRR prevents kainic acid-induced apoptosis and loss of function in organotypic hippocampal cultures

Brain research, 2006-11, Vol.1119 (1), p.26-39

2006

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Stress up-regulates neuronal expression of the herpes simplex virus type 2 large subunit of ribonucleotide reductase (R1; ICP10) by activating activator protein 1

Journal of neurovirology, 2005-08, Vol.11 (4), p.329-336

2005

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Overload of the heat-shock protein H11/HspB8 triggers melanoma cell apoptosis through activation of transforming growth factor-β-activated kinase 1

Oncogene, 2007-05, Vol.26 (24), p.3521-3531

2007

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Allergic Contact Dermatitis

Current directions in autoimmunity, 2008, Vol.10, p.1-26

2008

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1069. The HSV-2 Vector ΔRR Prevents Excitotoxicity-Induced Neuronal Cell Loss through Activation of Survival Pathways and Glial Cell Modulation

Molecular therapy, 2006-05, Vol.13 (S1), p.S410-S410

2006

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1069. The HSV-2 Vector [Delta]RR Prevents Excitotoxicity-Induced Neuronal Cell Loss through Activation of Survival Pathways and Glial Cell Modulation

Molecular therapy, 2006-05, Vol.13 (S1), p.S410

2006

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A case of adult-onset asthma with periocular xanthogranulomas

Archives of dermatology (1960), 2011-10, Vol.147 (10), p.1230

2011

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The growth compromised HSV-2 mutant DeltaRR prevents kainic acid-induced apoptosis and loss of function in organotypic hippocampal cultures

Brain research, 2006-11, Vol.1119 (1), p.26

2006

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THE HSV VECTOR ΔRR DELIVERS THE ANTI-APOPTOTIC PROTEIN ICP10PK AND PREVENTS APOPTOSIS AND LOSS OF FUNCTION IN KAINIC ACID TREATED ORGANOTYPIC HIPPOCAMPAL CULTURES THROUGH ERK AND AKT ACTIVATION

Brain research, 2006-10, Vol.1119 (1), p.26-39

2006

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Corrigendum to “Intranasal Administration of the Growth-compromised HSV-2 Vector ΔRR Prevents Kainate-induced Seizures and Neuronal Loss in Rats and Mice”

Molecular therapy, 2007-09, Vol.15 (9), p.1734-1734

2007

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Corrigendum to "Intranasal Administration of the Growth-compromised HSV-2 Vector [Delta]RR Prevents Kainate-induced Seizures and Neuronal Loss in Rats and Mice"

Molecular therapy, 2007-09, Vol.15 (9), p.1734

2007

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Intranasal Administration of the Growth-Compromised HSV-2 Vector [Delta]RR Prevents Kainate-Induced Seizures and Neuronal Loss in Rats and Mice

Molecular therapy, 2006-05, Vol.13 (5), p.870

2006

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Intranasal administration of the growth-compromised HSV-2 vector DeltaRR prevents kainate-induced seizures and neuronal loss in rats and mice

Molecular therapy, 2006-05, Vol.13 (5), p.870

2006

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Exposing the molecular heterogeneity of glycosylated biotherapeutics

Nature communications, 2024-04, Vol.15 (1), p.3259-3259, Article 3259

2024

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High-Throughput Analyses of Therapeutic Antibodies Using High-Field Asymmetric Waveform Ion Mobility Spectrometry Combined with SampleStream and Intact Protein Mass Spectrometry

Analytical chemistry (Washington), 2023-11, Vol.95 (47), p.17263-17272

2023

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Prevalence, management, and clinical consequences of QT interval prolongation during treatment with arsenic trioxide

Journal of clinical oncology, 2014-11, Vol.32 (33), p.3723-3728

2014

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From keratinocyte to cancer: the pathogenesis and modeling of cutaneous squamous cell carcinoma

Forced Expression of the H11 Heat Shock Protein Can Be Regulated by DNA Methylation and Trigger Apoptosis in Human Cells

Herpes simplex virus type 2 encodes a heat shock protein homologue with apoptosis regulatory functions

Human Natural Killer T Cells Infiltrate into the Skin at Elicitation Sites of Allergic Contact Dermatitis

Voriconazole enhances UV‐induced DNA damage by inhibiting catalase and promoting oxidative stress

Reconstructing skin cancers using animal models

The Herpes simplex virus gene Pol expressed in herpes-associated erythema multiforme lesions upregulates/activates SP1 and inflammatory cytokines

Intranasal Administration of the Growth-Compromised HSV-2 Vector ΔRR Prevents Kainate-Induced Seizures and Neuronal Loss in Rats and Mice

Novel Homozygous Frameshift Mutation of EVER1 Gene in an Epidermodysplasia Verruciformis Patient

The growth compromised HSV-2 mutant ΔRR prevents kainic acid-induced apoptosis and loss of function in organotypic hippocampal cultures

Stress up-regulates neuronal expression of the herpes simplex virus type 2 large subunit of ribonucleotide reductase (R1; ICP10) by activating activator protein 1

Overload of the heat-shock protein H11/HspB8 triggers melanoma cell apoptosis through activation of transforming growth factor-β-activated kinase 1

Allergic Contact Dermatitis

1069. The HSV-2 Vector ΔRR Prevents Excitotoxicity-Induced Neuronal Cell Loss through Activation of Survival Pathways and Glial Cell Modulation

1069. The HSV-2 Vector [Delta]RR Prevents Excitotoxicity-Induced Neuronal Cell Loss through Activation of Survival Pathways and Glial Cell Modulation

A case of adult-onset asthma with periocular xanthogranulomas

The growth compromised HSV-2 mutant DeltaRR prevents kainic acid-induced apoptosis and loss of function in organotypic hippocampal cultures

THE HSV VECTOR ΔRR DELIVERS THE ANTI-APOPTOTIC PROTEIN ICP10PK AND PREVENTS APOPTOSIS AND LOSS OF FUNCTION IN KAINIC ACID TREATED ORGANOTYPIC HIPPOCAMPAL CULTURES THROUGH ERK AND AKT ACTIVATION

Corrigendum to “Intranasal Administration of the Growth-compromised HSV-2 Vector ΔRR Prevents Kainate-induced Seizures and Neuronal Loss in Rats and Mice”

Corrigendum to "Intranasal Administration of the Growth-compromised HSV-2 Vector [Delta]RR Prevents Kainate-induced Seizures and Neuronal Loss in Rats and Mice"

Intranasal Administration of the Growth-Compromised HSV-2 Vector [Delta]RR Prevents Kainate-Induced Seizures and Neuronal Loss in Rats and Mice

Intranasal administration of the growth-compromised HSV-2 vector DeltaRR prevents kainate-induced seizures and neuronal loss in rats and mice

Exposing the molecular heterogeneity of glycosylated biotherapeutics

High-Throughput Analyses of Therapeutic Antibodies Using High-Field Asymmetric Waveform Ion Mobility Spectrometry Combined with SampleStream and Intact Protein Mass Spectrometry

Prevalence, management, and clinical consequences of QT interval prolongation during treatment with arsenic trioxide

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