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Using intonation to disambiguate meaning: The role of empathy and proficiency in L2 perceptual development

Applied psycholinguistics, 2023-09, Vol.44 (5), p.913-940

2023

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The Natural Flavonoid Naringenin Elicits Analgesia through Inhibition of NaV1.8 Voltage-Gated Sodium Channels

ACS chemical neuroscience, 2019-12, Vol.10 (12), p.4834-4846

2019

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Plant and fungi derived analgesic natural products targeting voltage-gated sodium and calcium channels

Channels (Austin, Tex.), 2022-12, Vol.16 (1), p.198-215

2022

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Non-SUMOylated CRMP2 decreases NaV1.7 currents via the endocytic proteins Numb, Nedd4-2 and Eps15

Molecular brain, 2021-01, Vol.14 (1), p.1-20, Article 20

2021

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CaV3.2 calcium channels: new players in facial pain

Pain (Amsterdam), 2022-12, Vol.163 (12), p.e1166-e1168

2022

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Ca V 3.2 calcium channels: new players in facial pain

Pain (Amsterdam), 2022-12, Vol.163 (12), p.e1166-e1168

2022

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Navigating Spatial Enclosures: Race, Place, and School Policing

Social problems (Berkeley, Calif.), 2023-11

2023

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EL MÉTODO MONTESSORI COMO ALTERNATIVA DIDÁCTICA EN EDUCACIÓN INICIAL

DIALÉCTICA, 2024-03, Vol.1 (22)

Kimberly Gómez

2024

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Transcription Factor Sp1 Regulates the Expression of Calcium Channel α2δ-1 Subunit in Neuropathic Pain

Neuroscience, 2019-08, Vol.412, p.207-215

2019

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Putative roles of SLC7A5 (LAT1) transporter in pain

Neurobiology of pain, 2020-08, Vol.8, p.100050-100050, Article 100050

2020

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Pain's puzzle pieces: MAGI-1, NaV1.8, degradation, analgesia

Pain (Amsterdam), 2024-10

2024

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Betulinic acid analogs inhibit N- and T-type voltage-gated calcium channels to attenuate nerve-injury associated neuropathic and formalin models of pain

Neurobiology of pain, 2023-01, Vol.13, p.100116-100116, Article 100116

2023

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1-O-Acetylgeopyxin A, a derivative of a fungal metabolite, blocks tetrodotoxin-sensitive voltage-gated sodium, calcium channels and neuronal excitability which correlates with inhibition of neuropathic pain

Molecular brain, 2020-05, Vol.13 (1), p.73-73, Article 73

2020

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Cell specific regulation of NaV1.7 activity and trafficking in rat nodose ganglia neurons

Neurobiology of pain, 2022-08, Vol.12, p.100109-100109, Article 100109

2022

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Targeting T-type/CaV3.2 channels for chronic pain

Translational research : the journal of laboratory and clinical medicine, 2021-08, Vol.234, p.20-30

2021

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Evaluating Search Results: An Empirical Analysis of Middle School Students' Use of Source Attributes to Select Useful Sources

Journal of Educational Computing Research, 2009-01, Vol.41 (1), p.63-82

2009

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Targeting the facilitatory effect of cAMP on HCN2 channel opening promotes analgesic actions

Biophysical journal, 2024-02, Vol.123 (3), p.261a-261a

2024

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Transcription Factor Sp1 Regulates the Expression of Calcium Channel α 2 δ-1 Subunit in Neuropathic Pain

Neuroscience, 2019-08, Vol.412, p.207

2019

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Peptide and Peptidomimetic Inhibitors Targeting the Interaction of Collapsin Response Mediator Protein 2 with the N‑Type Calcium Channel for Pain Relief

ACS pharmacology & translational science, 2024-07, Vol.7 (7), p.1916-1936

2024

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SARS-CoV-2 spike protein co-opts VEGF-A/neuropilin-1 receptor signaling to induce analgesia

Pain (Amsterdam), 2021-01, Vol.162 (1), p.243-252

2021

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L5-6 Spinal Nerve Ligation-induced Neuropathy Changes the Location and Function of Ca2+ Channels and Cdk5 and Affects the Compound Action Potential in Adjacent Intact L4 Afferent Fibers

Neuroscience, 2021-09, Vol.471, p.20-31

2021

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Cdk5-Dependent Phosphorylation of CaV3.2 T-Type Channels: Possible Role in Nerve Ligation-Induced Neuropathic Allodynia and the Compound Action Potential in Primary Afferent C Fibers

The Journal of neuroscience, 2020-01, Vol.40 (2), p.283-296

2020

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Regulation of the voltage-gated Ca 2+ channel Ca V α 2 δ-1 subunit expression by the transcription factor Egr-1

Neuroscience letters, 2018-04, Vol.673, p.136-141

2018

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Regulation of the voltage-gated Ca2+ channel CaVα2δ-1 subunit expression by the transcription factor Egr-1

Neuroscience letters, 2018-04, Vol.673, p.136-141

2018

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SUMOylation and DeSUMOylation: Tug of War of Pain Signaling

Molecular neurobiology, 2024-09

2024

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Using intonation to disambiguate meaning: The role of empathy and proficiency in L2 perceptual development

The Natural Flavonoid Naringenin Elicits Analgesia through Inhibition of NaV1.8 Voltage-Gated Sodium Channels

Plant and fungi derived analgesic natural products targeting voltage-gated sodium and calcium channels

Non-SUMOylated CRMP2 decreases NaV1.7 currents via the endocytic proteins Numb, Nedd4-2 and Eps15

CaV3.2 calcium channels: new players in facial pain

Ca V 3.2 calcium channels: new players in facial pain

Navigating Spatial Enclosures: Race, Place, and School Policing

EL MÉTODO MONTESSORI COMO ALTERNATIVA DIDÁCTICA EN EDUCACIÓN INICIAL

Transcription Factor Sp1 Regulates the Expression of Calcium Channel α2δ-1 Subunit in Neuropathic Pain

Putative roles of SLC7A5 (LAT1) transporter in pain

Pain's puzzle pieces: MAGI-1, NaV1.8, degradation, analgesia

Betulinic acid analogs inhibit N- and T-type voltage-gated calcium channels to attenuate nerve-injury associated neuropathic and formalin models of pain

1-O-Acetylgeopyxin A, a derivative of a fungal metabolite, blocks tetrodotoxin-sensitive voltage-gated sodium, calcium channels and neuronal excitability which correlates with inhibition of neuropathic pain

Cell specific regulation of NaV1.7 activity and trafficking in rat nodose ganglia neurons

Targeting T-type/CaV3.2 channels for chronic pain

Evaluating Search Results: An Empirical Analysis of Middle School Students' Use of Source Attributes to Select Useful Sources

Targeting the facilitatory effect of cAMP on HCN2 channel opening promotes analgesic actions

Transcription Factor Sp1 Regulates the Expression of Calcium Channel α 2 δ-1 Subunit in Neuropathic Pain

Peptide and Peptidomimetic Inhibitors Targeting the Interaction of Collapsin Response Mediator Protein 2 with the N‑Type Calcium Channel for Pain Relief

SARS-CoV-2 spike protein co-opts VEGF-A/neuropilin-1 receptor signaling to induce analgesia

L5-6 Spinal Nerve Ligation-induced Neuropathy Changes the Location and Function of Ca2+ Channels and Cdk5 and Affects the Compound Action Potential in Adjacent Intact L4 Afferent Fibers

Cdk5-Dependent Phosphorylation of CaV3.2 T-Type Channels: Possible Role in Nerve Ligation-Induced Neuropathic Allodynia and the Compound Action Potential in Primary Afferent C Fibers

Regulation of the voltage-gated Ca 2+ channel Ca V α 2 δ-1 subunit expression by the transcription factor Egr-1

Regulation of the voltage-gated Ca2+ channel CaVα2δ-1 subunit expression by the transcription factor Egr-1

SUMOylation and DeSUMOylation: Tug of War of Pain Signaling

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