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KRIT1 in vascular biology and beyond

Bioscience reports, 2024-07, Vol.44 (7)

Glading, Angela J

2024

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KRIT1 Protein Depletion Modifies Endothelial Cell Behavior via Increased Vascular Endothelial Growth Factor (VEGF) Signaling

The Journal of biological chemistry, 2014-11, Vol.289 (47), p.33054-33065

2014

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Up-regulation of NADPH oxidase-mediated redox signaling contributes to the loss of barrier function in KRIT1 deficient endothelium

Scientific reports, 2017-08, Vol.7 (1), p.8296-12, Article 8296

2017

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Is Location Everything? Regulation of the Endothelial CCM Signaling Complex

Frontiers in cardiovascular medicine, 2022-07, Vol.9, p.954780-954780

2022

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Oxidative stress and inflammation in cerebral cavernous malformation disease pathogenesis: Two sides of the same coin

The international journal of biochemistry & cell biology, 2016-12, Vol.81 (Pt B), p.254-270

2016

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KRIT1‐mediated regulation of neutrophil adhesion and motility

The FEBS journal, 2023-02, Vol.290 (4), p.1078-1095

2023

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Contribution of protein-protein interactions to the endothelial-barrier-stabilizing function of KRIT1

Journal of cell science, 2022-01, Vol.135 (2)

2022

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Phospholipase Cε Modulates Rap1 Activity and the Endothelial Barrier

PloS one, 2016-09, Vol.11 (9), p.e0162338-e0162338

2016

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KRIT1: A Traffic Warden at the Busy Crossroads Between Redox Signaling and the Pathogenesis of Cerebral Cavernous Malformation Disease

Antioxidants & redox signaling, 2023-03, Vol.38 (7-9), p.496

2023

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Measurement of Endothelial Barrier Function in Mouse Models of Cerebral Cavernous Malformations Using Intravital Microscopy

Methods in molecular biology (Clifton, N.J.), 2020, Vol.2152, p.387

Glading, Angela J

2020

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Microvascular Mimetics for the Study of Leukocyte–Endothelial Interactions

Cellular and molecular bioengineering, 2020-04, Vol.13 (2), p.125-139

2020

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VEGF signalling enhances lesion burden in KRIT1 deficient mice

Journal of cellular and molecular medicine, 2020-01, Vol.24 (1), p.632-639

2020

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Isolation of Cerebral Endothelial Cells from CCM1/KRIT1 Null Mouse Brain

Methods in molecular biology (Clifton, N.J.), 2020, Vol.2152, p.259

2020

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Protein kinase Cα regulates the nucleocytoplasmic shuttling of KRIT1

Journal of cell science, 2021-02, Vol.134 (3)

2021

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Rap1 and its effector KRIT1/CCM1 regulate beta-catenin signaling

Disease models & mechanisms, 2010-01, Vol.3 (1-2), p.73-83

2010

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Control of vascular permeability by adhesion molecules

Tissue barriers, 2015-01, Vol.3 (1-2), p.e985954-e985954

2015

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Decreased Krev interaction-trapped 1 expression leads to increased vascular permeability and modifies inflammatory responses in vivo

Arteriosclerosis, thrombosis, and vascular biology, 2012-11, Vol.32 (11), p.2702-2710

2012

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A systematic review and meta-analysis of preanalytical factors and methodological differences influencing the measurement of circulating vascular endothelial growth factor

PloS one, 2022-07, Vol.17 (7), p.e0270232

2022

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Effect of the double bond conjugation on the vascular physiology and nitric oxide production of isomers of eicosapentaenoic and docosahexaenoic acids prepared from shark oil

PloS one, 2020-02, Vol.15 (2), p.e0229435-e0229435

2020

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Disease models in cerebral cavernous malformations

Drug discovery today. Disease models, 2020, Vol.31, p.21-29

2020

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Protein kinase Cα regulates the nucleocytoplasmic shuttling of KRIT1

Free radical biology & medicine, 2021-12, Vol.177, p.S61

2021

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Phospholipase C[epsilon] Modulates Rap1 Activity and the Endothelial Barrier

PloS one, 2016-09, Vol.11 (9), p.e0162338

2016

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Rap1 and its effector KRIT1/CCM1 regulate β-catenin signaling

Journal of cell science, 2010-02, Vol.123 (3), p.e1-e1

2010

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Protein kinase Cα (PKCα) regulates the nucleocytoplasmic shuttling of KRIT1

Journal of cell science, 2020-01

2020

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Activation of m-Calpain (Calpain II) by Epidermal Growth Factor Is Limited by Protein Kinase A Phosphorylation of m-Calpain

Molecular and Cellular Biology, 2002-04, Vol.22 (8), p.2716-2727

2002

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KRIT1 in vascular biology and beyond

KRIT1 Protein Depletion Modifies Endothelial Cell Behavior via Increased Vascular Endothelial Growth Factor (VEGF) Signaling

Up-regulation of NADPH oxidase-mediated redox signaling contributes to the loss of barrier function in KRIT1 deficient endothelium

Is Location Everything? Regulation of the Endothelial CCM Signaling Complex

Oxidative stress and inflammation in cerebral cavernous malformation disease pathogenesis: Two sides of the same coin

KRIT1‐mediated regulation of neutrophil adhesion and motility

Contribution of protein-protein interactions to the endothelial-barrier-stabilizing function of KRIT1

Phospholipase Cε Modulates Rap1 Activity and the Endothelial Barrier

KRIT1: A Traffic Warden at the Busy Crossroads Between Redox Signaling and the Pathogenesis of Cerebral Cavernous Malformation Disease

Measurement of Endothelial Barrier Function in Mouse Models of Cerebral Cavernous Malformations Using Intravital Microscopy

Microvascular Mimetics for the Study of Leukocyte–Endothelial Interactions

VEGF signalling enhances lesion burden in KRIT1 deficient mice

Isolation of Cerebral Endothelial Cells from CCM1/KRIT1 Null Mouse Brain

Protein kinase Cα regulates the nucleocytoplasmic shuttling of KRIT1

Rap1 and its effector KRIT1/CCM1 regulate beta-catenin signaling

Control of vascular permeability by adhesion molecules

Decreased Krev interaction-trapped 1 expression leads to increased vascular permeability and modifies inflammatory responses in vivo

A systematic review and meta-analysis of preanalytical factors and methodological differences influencing the measurement of circulating vascular endothelial growth factor

Effect of the double bond conjugation on the vascular physiology and nitric oxide production of isomers of eicosapentaenoic and docosahexaenoic acids prepared from shark oil

Disease models in cerebral cavernous malformations

Protein kinase Cα regulates the nucleocytoplasmic shuttling of KRIT1

Phospholipase C[epsilon] Modulates Rap1 Activity and the Endothelial Barrier

Rap1 and its effector KRIT1/CCM1 regulate β-catenin signaling

Protein kinase Cα (PKCα) regulates the nucleocytoplasmic shuttling of KRIT1

Activation of m-Calpain (Calpain II) by Epidermal Growth Factor Is Limited by Protein Kinase A Phosphorylation of m-Calpain

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