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A systematic review and meta-analysis of preanalytical factors and methodological differences influencing the measurement of circulating vascular endothelial growth factor

PloS one, 2022-07, Vol.17 (7), p.e0270232

2022

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Effect of the double bond conjugation on the vascular physiology and nitric oxide production of isomers of eicosapentaenoic and docosahexaenoic acids prepared from shark oil

PloS one, 2020-02, Vol.15 (2), p.e0229435-e0229435

2020

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Epidermal Growth Factor Activates m-Calpain (Calpain II), at Least in Part, by Extracellular Signal-Regulated Kinase-Mediated Phosphorylation

Molecular and Cellular Biology, 2004-03, Vol.24 (6), p.2499-2512

2004

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Cutting to the chase: calpain proteases in cell motility

Trends in Cell Biology, 2002, Vol.12 (1), p.46-54

2002

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Delayed onset panuveitis following intravitreal aflibercept injection

BMJ case reports, 2014-06, Vol.2014, p.bcr2013202515

2014

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Activation of m-Calpain (Calpain II) by Epidermal Growth Factor Is Limited by Protein Kinase A Phosphorylation of m-Calpain

Molecular and Cellular Biology, 2002-04, Vol.22 (8), p.2716-2727

2002

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Epidermal Growth Factor Receptor Activation of Calpain Is Required for Fibroblast Motility and Occurs via an ERK/MAP Kinase Signaling Pathway

The Journal of biological chemistry, 2000-01, Vol.275 (4), p.2390-2398

2000

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Membrane Proximal ERK Signaling Is Required for M-calpain Activation Downstream of Epidermal Growth Factor Receptor Signaling

The Journal of biological chemistry, 2001-06, Vol.276 (26), p.23341-23348

2001

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Interferon-Inducible Protein 9 (CXCL11)-Induced Cell Motility in Keratinocytes Requires Calcium Flux-Dependent Activation of μ-Calpain

Molecular and Cellular Biology, 2005-03, Vol.25 (5), p.1922-1941

2005

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Epidermal Growth Factor Activates m-Calpain (Calpain II), at Least in Part, by Extracellular Signal-Regulated Kinase-Mediated Phosphorylation

Molecular and cellular biology, 2004-07, Vol.24 (13), p.6116-6116

2004

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Phosphorylation of phosphoprotein enriched in astrocytes (PEA-15) regulates extracellular signal-regulated kinase-dependent transcription and cell proliferation

Molecular biology of the cell, 2005-08, Vol.16 (8), p.3552-3561

2005

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IP-10 Inhibits Epidermal Growth Factor-Induced Motility by Decreasing Epidermal Growth Factor Receptor-Mediated Calpain Activity

The Journal of cell biology, 1999-07, Vol.146 (1), p.243-253

1999

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PEA-15 Binding to ERK1/2 MAPKs Is Required for Its Modulation of Integrin Activation

The Journal of biological chemistry, 2003-12, Vol.278 (52), p.52587-52597

2003

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Epidermal growth factor receptor-mediated motility in fibroblasts

Microscopy research and technique, 1998-12, Vol.43 (5), p.395-411

1998

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Epidermal Growth Factor Receptor-Mediated Motility in Fibroblasts

Microscopy research and technique, 1998-12, Vol.45 (5), p.395-411

1998

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Delayed onset panuveitis following intravitreal aflibercept injection

BMJ Case Reports, 2014, Vol.2014

2014

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KRIT1 in vascular biology and beyond

Bioscience reports, 2024-07, Vol.44 (7)

Glading, Angela J.

2024

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Oxidative stress and inflammation in cerebral cavernous malformation disease pathogenesis: Two sides of the same coin

The international journal of biochemistry & cell biology, 2016-12, Vol.81 (Pt B), p.254-270

2016

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KRIT1‐mediated regulation of neutrophil adhesion and motility

The FEBS journal, 2023-02, Vol.290 (4), p.1078-1095

2023

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Contribution of protein-protein interactions to the endothelial-barrier-stabilizing function of KRIT1

Journal of cell science, 2022-01, Vol.135 (2)

2022

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KRIT1 stabilizes endothelial adherens junctions independent of Rap1 via regulation of β1‐integrin

The FASEB journal, 2021-05, Vol.35 (S1), p.n/a, Article fasebj.2021.35.S1.04041

2021

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KRIT1: A Traffic Warden at the Busy Crossroads Between Redox Signaling and the Pathogenesis of Cerebral Cavernous Malformation Disease

Antioxidants & redox signaling, 2023-03, Vol.38 (7-9), p.496

2023

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Measurement of Endothelial Barrier Function in Mouse Models of Cerebral Cavernous Malformations Using Intravital Microscopy

Methods in molecular biology (Clifton, N.J.), 2020, Vol.2152, p.387

Glading, Angela J

2020

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VEGF signalling enhances lesion burden in KRIT1 deficient mice

Journal of cellular and molecular medicine, 2020-01, Vol.24 (1), p.632-639

2020

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KRIT1 Protein Depletion Modifies Endothelial Cell Behavior via Increased Vascular Endothelial Growth Factor (VEGF) Signaling

The Journal of biological chemistry, 2014-11, Vol.289 (47), p.33054-33065

2014

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A systematic review and meta-analysis of preanalytical factors and methodological differences influencing the measurement of circulating vascular endothelial growth factor

Effect of the double bond conjugation on the vascular physiology and nitric oxide production of isomers of eicosapentaenoic and docosahexaenoic acids prepared from shark oil

Epidermal Growth Factor Activates m-Calpain (Calpain II), at Least in Part, by Extracellular Signal-Regulated Kinase-Mediated Phosphorylation

Cutting to the chase: calpain proteases in cell motility

Delayed onset panuveitis following intravitreal aflibercept injection

Activation of m-Calpain (Calpain II) by Epidermal Growth Factor Is Limited by Protein Kinase A Phosphorylation of m-Calpain

Epidermal Growth Factor Receptor Activation of Calpain Is Required for Fibroblast Motility and Occurs via an ERK/MAP Kinase Signaling Pathway

Membrane Proximal ERK Signaling Is Required for M-calpain Activation Downstream of Epidermal Growth Factor Receptor Signaling

Interferon-Inducible Protein 9 (CXCL11)-Induced Cell Motility in Keratinocytes Requires Calcium Flux-Dependent Activation of μ-Calpain

Epidermal Growth Factor Activates m-Calpain (Calpain II), at Least in Part, by Extracellular Signal-Regulated Kinase-Mediated Phosphorylation

Phosphorylation of phosphoprotein enriched in astrocytes (PEA-15) regulates extracellular signal-regulated kinase-dependent transcription and cell proliferation

IP-10 Inhibits Epidermal Growth Factor-Induced Motility by Decreasing Epidermal Growth Factor Receptor-Mediated Calpain Activity

PEA-15 Binding to ERK1/2 MAPKs Is Required for Its Modulation of Integrin Activation

Epidermal growth factor receptor-mediated motility in fibroblasts

Epidermal Growth Factor Receptor-Mediated Motility in Fibroblasts

Delayed onset panuveitis following intravitreal aflibercept injection

KRIT1 in vascular biology and beyond

Oxidative stress and inflammation in cerebral cavernous malformation disease pathogenesis: Two sides of the same coin

KRIT1‐mediated regulation of neutrophil adhesion and motility

Contribution of protein-protein interactions to the endothelial-barrier-stabilizing function of KRIT1

KRIT1 stabilizes endothelial adherens junctions independent of Rap1 via regulation of β1‐integrin

KRIT1: A Traffic Warden at the Busy Crossroads Between Redox Signaling and the Pathogenesis of Cerebral Cavernous Malformation Disease

Measurement of Endothelial Barrier Function in Mouse Models of Cerebral Cavernous Malformations Using Intravital Microscopy

VEGF signalling enhances lesion burden in KRIT1 deficient mice

KRIT1 Protein Depletion Modifies Endothelial Cell Behavior via Increased Vascular Endothelial Growth Factor (VEGF) Signaling

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