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An mTORC1-to-CDK1 Switch Maintains Autophagy Suppression during Mitosis

Molecular cell, 2020-01, Vol.77 (2), p.228-240.e7

2020

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Targeting melanoma’s MCL1 bias unleashes the apoptotic potential of BRAF and ERK1/2 pathway inhibitors

Nature communications, 2019-11, Vol.10 (1), p.5167-19, Article 5167

2019

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Adaptation to mTOR kinase inhibitors by amplification of eIF4E to maintain cap-dependent translation

Journal of cell science, 2014-02, Vol.127 (Pt 4), p.788

2014

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ERK1/2-dependent phosphorylation of BimEL promotes its rapid dissociation from Mcl-1 and Bcl-xL

The EMBO journal, 2007-06, Vol.26 (12), p.2856-2867

2007

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MEK1/2 inhibitor withdrawal reverses acquired resistance driven by BRAFV600E amplification whereas KRASG13D amplification promotes EMT-chemoresistance

Nature communications, 2019-05, Vol.10 (1), p.1-22, Article 2030

2019

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Control of cell death and mitochondrial fission by ERK1/2 MAP kinase signalling

The FEBS journal, 2017-12, Vol.284 (24), p.4177-4195

2017

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Control of cell death and mitochondrial fission by ERK 1/2 MAP kinase signalling

The FEBS journal, 2017-12, Vol.284 (24), p.4177-4195

2017

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Reporter cell lines to screen for inhibitors or regulators of the KRAS-RAF-MEK1/2-ERK1/2 pathway

Biochemical journal, 2024-03, Vol.481 (6), p.405-422

2024

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ERK5 and its role in tumour development

Biochemical Society transactions, 2012-02, Vol.40 (1), p.251-256

2012

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Adverse Childhood Experiences in Adolescents Who Have Engaged in Sexually Abusive Behavior: The Impact of Polyvictimization on Relevant Outcomes

Gilley, Rebecca

2019

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Tumor cells with KRAS or BRAF mutations or ERK5/MAPK7 amplification are not addicted to ERK5 activity for cell proliferation

Cell cycle (Georgetown, Tex.), 2016-02, Vol.15 (4), p.506-518

2016

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ERK1/2, but not ERK5, is necessary and sufficient for phosphorylation and activation of c-Fos

Cellular signalling, 2009-06, Vol.21 (6), p.969-977

2009

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Visualization of Endogenous ERK1/2 in Cells with a Bioorthogonal Covalent Probe

Bioconjugate chemistry, 2017-06, Vol.28 (6), p.1677-1683

2017

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CDK1, not ERK1/2 or ERK5, is required for mitotic phosphorylation of BIMEL

Cellular signalling, 2012-01, Vol.24 (1), p.170-180

2012

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Adaptation to chronic mTOR inhibition in cancer and in aging

Biochemical Society transactions, 2013-08, Vol.41 (4), p.956-961

2013

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The duration of ERK1/2 activity determines the activation of c-Fos and Fra-1 and the composition and quantitative transcriptional output of AP-1

Cellular signalling, 2007-04, Vol.19 (4), p.695-704

2007

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CDK1, not ERK1/2 or ERK5, is required for mitotic phosphorylation of BIM sub(EL)

Cellular signalling, 2012-01, Vol.24 (1), p.170-180

2012

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MEK1/2 inhibitor withdrawal reverses acquired resistance driven by BRAF V600E amplification whereas KRAS G13D amplification promotes EMT-chemoresistance

Nature communications, 2019-05, Vol.10 (1), p.2030

2019

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ERK1/2-dependent phosphorylation of Bim EL promotes its rapid dissociation from Mcl-1 and Bcl-x L

The EMBO journal, 2007-06, Vol.26 (12), p.2856-2867

2007

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Species-independent contribution of ZBP1/DAI/DLM-1-triggered necroptosis in host defense against HSV1

Cell death & disease, 2018-07, Vol.9 (8), p.816-11, Article 816

2018

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Two-Stage (Collaborative) Testing in Science Teaching: Does It Improve Grades on Short-Answer Questions and Retention of Material?

Journal of college science teaching, 2019-03, Vol.48 (4), p.64-73

2019

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The proapoptotic dp5 gene is a direct target of the MLK-JNK-c-Jun pathway in sympathetic neurons

Nucleic acids research, 2009-05, Vol.37 (9), p.3044-3060

2009

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The effect of supplemental grape seed extract on pig growth performance and body composition during heat stress

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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An mTORC1-to-CDK1 Switch Maintains Autophagy Suppression during Mitosis

Targeting melanoma’s MCL1 bias unleashes the apoptotic potential of BRAF and ERK1/2 pathway inhibitors

Adaptation to mTOR kinase inhibitors by amplification of eIF4E to maintain cap-dependent translation

ERK1/2-dependent phosphorylation of BimEL promotes its rapid dissociation from Mcl-1 and Bcl-xL

MEK1/2 inhibitor withdrawal reverses acquired resistance driven by BRAFV600E amplification whereas KRASG13D amplification promotes EMT-chemoresistance

Control of cell death and mitochondrial fission by ERK1/2 MAP kinase signalling

Control of cell death and mitochondrial fission by ERK 1/2 MAP kinase signalling

Reporter cell lines to screen for inhibitors or regulators of the KRAS-RAF-MEK1/2-ERK1/2 pathway

ERK5 and its role in tumour development

Adverse Childhood Experiences in Adolescents Who Have Engaged in Sexually Abusive Behavior: The Impact of Polyvictimization on Relevant Outcomes

Tumor cells with KRAS or BRAF mutations or ERK5/MAPK7 amplification are not addicted to ERK5 activity for cell proliferation

ERK1/2, but not ERK5, is necessary and sufficient for phosphorylation and activation of c-Fos

Visualization of Endogenous ERK1/2 in Cells with a Bioorthogonal Covalent Probe

CDK1, not ERK1/2 or ERK5, is required for mitotic phosphorylation of BIMEL

Adaptation to chronic mTOR inhibition in cancer and in aging

The duration of ERK1/2 activity determines the activation of c-Fos and Fra-1 and the composition and quantitative transcriptional output of AP-1

CDK1, not ERK1/2 or ERK5, is required for mitotic phosphorylation of BIM sub(EL)

MEK1/2 inhibitor withdrawal reverses acquired resistance driven by BRAF V600E amplification whereas KRAS G13D amplification promotes EMT-chemoresistance

ERK1/2-dependent phosphorylation of Bim EL promotes its rapid dissociation from Mcl-1 and Bcl-x L

Species-independent contribution of ZBP1/DAI/DLM-1-triggered necroptosis in host defense against HSV1

Enrichment of SARM1 alleles encoding variants with constitutively hyperactive NADase in patients with ALS and other motor nerve disorders

Two-Stage (Collaborative) Testing in Science Teaching: Does It Improve Grades on Short-Answer Questions and Retention of Material?

The proapoptotic dp5 gene is a direct target of the MLK-JNK-c-Jun pathway in sympathetic neurons

The effect of supplemental grape seed extract on pig growth performance and body composition during heat stress

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