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Fusion transcripts FYN-TRAF3IP2 and KHDRBS1-LCK hijack T cell receptor signaling in peripheral T-cell lymphoma, not otherwise specified

Nature communications, 2021-06, Vol.12 (1), p.3705-3705, Article 3705

2021

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JAK2 rearrangements, including the novel SEC31A-JAK2 fusion, are recurrent in classical Hodgkin lymphoma

Blood, 2011-04, Vol.117 (15), p.4056-4064

2011

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PSEN1-selective gamma-secretase inhibition in combination with kinase or XPO-1 inhibitors effectively targets T cell acute lymphoblastic leukemia

Journal of hematology and oncology, 2021-06, Vol.14 (1), p.97-14, Article 97

2021

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NUP214-ABL1-mediated cell proliferation in T-cell acute lymphoblastic leukemia is dependent on the LCK kinase and various interacting proteins

Haematologica (Roma), 2014-01, Vol.99 (1), p.85

2014

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Ruxolitinib Synergizes With Dexamethasone for the Treatment of T‐cell Acute Lymphoblastic Leukemia

HemaSphere, 2019-12, Vol.3 (6), p.e310-n/a

2019

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The CCR4-NOT complex is a tumor suppressor in Drosophila melanogaster eye cancer models

Journal of hematology and oncology, 2018-08, Vol.11 (1), p.108-108, Article 108

2018

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Monitoring of Leukemia Clones in B‐cell Acute Lymphoblastic Leukemia at Diagnosis and During Treatment by Single‐cell DNA Amplicon Sequencing

HemaSphere, 2022-04, Vol.6 (4), p.e700-n/a

2022

Volltextzugriff (PDF)

Kinase Activation and Transformation by NUP214-ABL1 Is Dependent on the Context of the Nuclear Pore

Molecular cell, 2008-07, Vol.31 (1), p.134-142

2008

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BCR‐ABL1 positive B‐ALL can undergo T‐cell lineage shift to become CD19 negative T‐ALL

HemaSphere, 2018-06, Vol.2 (3), p.e42-n/a

2018

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JAK1 mutation analysis in T-cell acute lymphoblastic leukemia cell lines

Haematologica (Roma), 2009-03, Vol.94 (3), p.435-437

2009

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Single-cell DNA amplicon sequencing reveals clonal heterogeneity and evolution in T-cell acute lymphoblastic leukemia

Blood, 2021-02, Vol.137 (6), p.801-811

2021

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Optimizing the diagnostic workflow for acute lymphoblastic leukemia by optical genome mapping

American journal of hematology, 2022-05, Vol.97 (5), p.548-561

2022

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JAK3 mutants transform hematopoietic cells through JAK1 activation, causing T-cell acute lymphoblastic leukemia in a mouse model

Blood, 2014-11, Vol.124 (20), p.3092-3100

2014

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Aberrant MYCN expression drives oncogenic hijacking of EZH2 as a transcriptional activator in peripheral T-cell lymphoma

Blood, 2022-12, Vol.140 (23), p.2463-2476

2022

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Suz12 inactivation cooperates with JAK3 mutant signaling in the development of T-cell acute lymphoblastic leukemia

Blood, 2019-10, Vol.134 (16), p.1323-1336

2019

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The XPO1 Inhibitor KPT-8602 Synergizes with Dexamethasone in Acute Lymphoblastic Leukemia

Clinical cancer research, 2020-11, Vol.26 (21), p.5747-5758

2020

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HOXA9 Cooperates with Activated JAK/STAT Signaling to Drive Leukemia Development

Cancer discovery, 2018-05, Vol.8 (5), p.616-631

2018

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Hedgehog pathway activation in T-cell acute lymphoblastic leukemia predicts response to SMO and GLI1 inhibitors

Blood, 2016-12, Vol.128 (23), p.2642-2654

2016

Volltextzugriff (PDF)

Opportunities of Genome Imaging for Genetic Diagnosis in Acute Lymphoblastic Leukemia

Blood, 2020-11, Vol.136 (Supplement 1), p.10-11

2020

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Evolution of Clinically Relevant Subclones during Chemotherapy Treatment of ALL As Determined By Single-Cell DNA and RNA Sequencing

Blood, 2019-11, Vol.134 (Supplement_1), p.2749-2749

2019

Volltextzugriff (PDF)

Distinct Acute Lymphoblastic Leukemia (ALL)-associated Janus Kinase 3 (JAK3) Mutants Exhibit Different Cytokine-Receptor Requirements and JAK Inhibitor Specificities

The Journal of biological chemistry, 2015-11, Vol.290 (48), p.29022-29034

2015

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Mutant JAK3 signaling is increased by loss of wild-type JAK3 or by acquisition of secondary JAK3 mutations in T-ALL

Blood, 2018-01, Vol.131 (4), p.421-425

2018

Volltextzugriff (PDF)

Oncogenic cooperation between TCF7-SPI1 and NRAS(G12D) requires β-catenin activity to drive T-cell acute lymphoblastic leukemia

Nature communications, 2021-07, Vol.12 (1), p.1-15, Article 4164

2021

Volltextzugriff (PDF)

Development of a siRNA and shRNA screening system based on a kinase fusion protein

RNA (Cambridge), 2012-06, Vol.18 (6), p.1296-1306

2012

Volltextzugriff (PDF)

Synergism Between HOXA9 and Mutant JAK3 (M511I) Leads to Rapid Leukemia Development within an in Vivo Murine Bone Marrow Transplant Model

Blood, 2014-12, Vol.124 (21), p.1078-1078

2014

Volltextzugriff (PDF)

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Fusion transcripts FYN-TRAF3IP2 and KHDRBS1-LCK hijack T cell receptor signaling in peripheral T-cell lymphoma, not otherwise specified

JAK2 rearrangements, including the novel SEC31A-JAK2 fusion, are recurrent in classical Hodgkin lymphoma

PSEN1-selective gamma-secretase inhibition in combination with kinase or XPO-1 inhibitors effectively targets T cell acute lymphoblastic leukemia

NUP214-ABL1-mediated cell proliferation in T-cell acute lymphoblastic leukemia is dependent on the LCK kinase and various interacting proteins

Ruxolitinib Synergizes With Dexamethasone for the Treatment of T‐cell Acute Lymphoblastic Leukemia

The CCR4-NOT complex is a tumor suppressor in Drosophila melanogaster eye cancer models

Monitoring of Leukemia Clones in B‐cell Acute Lymphoblastic Leukemia at Diagnosis and During Treatment by Single‐cell DNA Amplicon Sequencing

Kinase Activation and Transformation by NUP214-ABL1 Is Dependent on the Context of the Nuclear Pore

BCR‐ABL1 positive B‐ALL can undergo T‐cell lineage shift to become CD19 negative T‐ALL

JAK1 mutation analysis in T-cell acute lymphoblastic leukemia cell lines

Single-cell DNA amplicon sequencing reveals clonal heterogeneity and evolution in T-cell acute lymphoblastic leukemia

Optimizing the diagnostic workflow for acute lymphoblastic leukemia by optical genome mapping

JAK3 mutants transform hematopoietic cells through JAK1 activation, causing T-cell acute lymphoblastic leukemia in a mouse model

Aberrant MYCN expression drives oncogenic hijacking of EZH2 as a transcriptional activator in peripheral T-cell lymphoma

Suz12 inactivation cooperates with JAK3 mutant signaling in the development of T-cell acute lymphoblastic leukemia

The XPO1 Inhibitor KPT-8602 Synergizes with Dexamethasone in Acute Lymphoblastic Leukemia

HOXA9 Cooperates with Activated JAK/STAT Signaling to Drive Leukemia Development

Hedgehog pathway activation in T-cell acute lymphoblastic leukemia predicts response to SMO and GLI1 inhibitors

Opportunities of Genome Imaging for Genetic Diagnosis in Acute Lymphoblastic Leukemia

Evolution of Clinically Relevant Subclones during Chemotherapy Treatment of ALL As Determined By Single-Cell DNA and RNA Sequencing

Distinct Acute Lymphoblastic Leukemia (ALL)-associated Janus Kinase 3 (JAK3) Mutants Exhibit Different Cytokine-Receptor Requirements and JAK Inhibitor Specificities

Mutant JAK3 signaling is increased by loss of wild-type JAK3 or by acquisition of secondary JAK3 mutations in T-ALL

Oncogenic cooperation between TCF7-SPI1 and NRAS(G12D) requires β-catenin activity to drive T-cell acute lymphoblastic leukemia

Development of a siRNA and shRNA screening system based on a kinase fusion protein

Synergism Between HOXA9 and Mutant JAK3 (M511I) Leads to Rapid Leukemia Development within an in Vivo Murine Bone Marrow Transplant Model

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