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Sphingolipid-Containing Outer Membrane Vesicles Serve as a Delivery Vehicle To Limit Macrophage Immune Response to Porphyromonas gingivalis

Infection and immunity, 2021-03, Vol.89 (4)

2021

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Macrophage-specific TLR2 signaling mediates pathogen-induced TNF-dependent inflammatory oral bone loss

The Journal of immunology (1950), 2013-02, Vol.190 (3), p.1148-1157

2013

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Immunization with cell‐free‐generated vaccine protects from Porphyromonas gingivalis‐induced alveolar bone loss

Journal of clinical periodontology, 2019-02, Vol.46 (2), p.197-205

2019

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Porphyromonas gingivalis accelerates inflammatory atherosclerosis in the innominate artery of ApoE deficient mice

Atherosclerosis, 2011-03, Vol.215 (1), p.52-59

2011

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Innate immune recognition of invasive bacteria accelerates atherosclerosis in apolipoprotein E-deficient mice

Circulation (New York, N.Y.), 2004-06, Vol.109 (22), p.2801-2806

2004

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Toll-like receptor 2 plays a critical role in the progression of atherosclerosis that is independent of dietary lipids

Atherosclerosis, 2008-01, Vol.196 (1), p.146-154

2008

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Engagement of specific innate immune signaling pathways during Porphyromonas gingivalis induced chronic inflammation and atherosclerosis

Frontiers in bioscience, 2008-01, Vol.13 (13), p.2041

2008

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A Porphyromonas gingivalis hypothetical protein controlled by the type I-C CRISPR-Cas system is a novel adhesin important in virulence

mSystems, 2024-03, Vol.9 (3), p.e0123123-e0123123

2024

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Protective role for TLR4 signaling in atherosclerosis progression as revealed by infection with a common oral pathogen

The Journal of immunology (1950), 2012-10, Vol.189 (7), p.3681-3688

2012

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Characterization of a Bacterial Kinase That Phosphorylates Dihydrosphingosine to Form dhS1P

Microbiology spectrum, 2022-04, Vol.10 (2), p.e0000222-e0000222

2022

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Virulence of the Pathogen Porphyromonas gingivalis Is Controlled by the CRISPR-Cas Protein Cas3

mSystems, 2020-09, Vol.5 (5)

2020

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Signaling events in pathogen-induced macrophage foam cell formation

Pathogens and disease, 2016-08, Vol.74 (6), p.ftw074

2016

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Porphyromonas gingivalis mediated periodontal disease and atherosclerosis: disparate diseases with commonalities in pathogenesis through TLRs

Current pharmaceutical design, 2007-12, Vol.13 (36), p.3665-3675

2007

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Macrophage-Elicited Osteoclastogenesis in Response to Bacterial Stimulation Requires Toll-Like Receptor 2-Dependent Tumor Necrosis Factor-Alpha Production

Infection and Immunity, 2008-02, Vol.76 (2), p.812-819

2008

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Cell Wall Hydrolytic Enzymes Enhance Antimicrobial Drug Activity Against Mycobacterium

Current microbiology, 2019-04, Vol.76 (4), p.398-409

2019

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Fimbria‐dependent activation of pro‐inflammatory molecules in Porphyromonas gingivalis infected human aortic endothelial cells

Cellular microbiology, 2006-05, Vol.8 (5), p.738-757

2006

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Inflammatory response to Porphyromonas gingivalis partially requires interferon regulatory factor (IRF) 3

Innate immunity (London, England), 2014-04, Vol.20 (3), p.312-319

2014

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Pathogen-Mediated Inflammatory Atherosclerosis Is Mediated in Part via Toll-Like Receptor 2-Induced Inflammatory Responses

Journal of innate immunity, 2010-01, Vol.2 (4), p.334-343

2010

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A Porphyromonas gingivalis Capsule-Conjugate Vaccine Protects From Experimental Oral Bone Loss

Frontiers in oral health, 2021-07, Vol.2, p.686402-686402

2021

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Bacterial Fimbriae Stimulate Proinflammatory Activation in the Endothelium through Distinct TLRs

The Journal of immunology (1950), 2008-02, Vol.180 (4), p.2187-2195

2008

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Roles of the host oxidative immune response and bacterial antioxidant rubrerythrin during Porphyromonas gingivalis infection

PLoS pathogens, 2006-07, Vol.2 (7), p.e76-e76

2006

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Immuno-Pathogenesis of Periodontal Disease: Current and Emerging Paradigms

Current oral health reports, 2014-06, Vol.1 (2), p.124-132

2014

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Distinct gene signatures in aortic tissue from ApoE-/- mice exposed to pathogens or Western diet

BMC genomics, 2014-12, Vol.15 (1), p.1176-1176, Article 1176

2014

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Sensitization of Human Aortic Endothelial Cells to Lipopolysaccharide via Regulation of Toll-Like Receptor 4 by Bacterial Fimbria-Dependent Invasion

Infection and Immunity, 2005-12, Vol.73 (12), p.8050-8059

2005

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Porphyromonas gingivalis Fimbria-Dependent Activation of Inflammatory Genes in Human Aortic Endothelial Cells

Infection and Immunity, 2005-09, Vol.73 (9), p.5367-5378

2005

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Sphingolipid-Containing Outer Membrane Vesicles Serve as a Delivery Vehicle To Limit Macrophage Immune Response to Porphyromonas gingivalis

Macrophage-specific TLR2 signaling mediates pathogen-induced TNF-dependent inflammatory oral bone loss

Immunization with cell‐free‐generated vaccine protects from Porphyromonas gingivalis‐induced alveolar bone loss

Porphyromonas gingivalis accelerates inflammatory atherosclerosis in the innominate artery of ApoE deficient mice

Innate immune recognition of invasive bacteria accelerates atherosclerosis in apolipoprotein E-deficient mice

Toll-like receptor 2 plays a critical role in the progression of atherosclerosis that is independent of dietary lipids

Engagement of specific innate immune signaling pathways during Porphyromonas gingivalis induced chronic inflammation and atherosclerosis

A Porphyromonas gingivalis hypothetical protein controlled by the type I-C CRISPR-Cas system is a novel adhesin important in virulence

Protective role for TLR4 signaling in atherosclerosis progression as revealed by infection with a common oral pathogen

Characterization of a Bacterial Kinase That Phosphorylates Dihydrosphingosine to Form dhS1P

Virulence of the Pathogen Porphyromonas gingivalis Is Controlled by the CRISPR-Cas Protein Cas3

Signaling events in pathogen-induced macrophage foam cell formation

Porphyromonas gingivalis mediated periodontal disease and atherosclerosis: disparate diseases with commonalities in pathogenesis through TLRs

Macrophage-Elicited Osteoclastogenesis in Response to Bacterial Stimulation Requires Toll-Like Receptor 2-Dependent Tumor Necrosis Factor-Alpha Production

Cell Wall Hydrolytic Enzymes Enhance Antimicrobial Drug Activity Against Mycobacterium

Fimbria‐dependent activation of pro‐inflammatory molecules in Porphyromonas gingivalis infected human aortic endothelial cells

Inflammatory response to Porphyromonas gingivalis partially requires interferon regulatory factor (IRF) 3

Pathogen-Mediated Inflammatory Atherosclerosis Is Mediated in Part via Toll-Like Receptor 2-Induced Inflammatory Responses

A Porphyromonas gingivalis Capsule-Conjugate Vaccine Protects From Experimental Oral Bone Loss

Bacterial Fimbriae Stimulate Proinflammatory Activation in the Endothelium through Distinct TLRs

Roles of the host oxidative immune response and bacterial antioxidant rubrerythrin during Porphyromonas gingivalis infection

Immuno-Pathogenesis of Periodontal Disease: Current and Emerging Paradigms

Distinct gene signatures in aortic tissue from ApoE-/- mice exposed to pathogens or Western diet

Sensitization of Human Aortic Endothelial Cells to Lipopolysaccharide via Regulation of Toll-Like Receptor 4 by Bacterial Fimbria-Dependent Invasion

Porphyromonas gingivalis Fimbria-Dependent Activation of Inflammatory Genes in Human Aortic Endothelial Cells

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