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Enhanced DNA binding capacity on up‐regulated epidermal wild‐type p53 in vitiligo by H2O2‐mediated oxidation: a possible repair mechanism for DNA damage

The FASEB journal, 2009-11, Vol.23 (11), p.3790-3807

2009

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Ultra-high resolution X-ray structures of two forms of human recombinant insulin at 100 K

BMC chemistry, 2017-08, Vol.11 (1), p.1

2017

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Oxidative Stress Via Hydrogen Peroxide Affects Proopiomelanocortin Peptides Directly in the Epidermis of Patients with Vitiligo

Journal of investigative dermatology, 2007-02, Vol.127 (2), p.411-420

2007

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Computer Simulation of Native Epidermal Enzyme Structures in the Presence and Absence of Hydrogen Peroxide (H2O2): Potential and Pitfalls

Journal of investigative dermatology, 2006-12, Vol.126 (12), p.2576-2582

2006

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Presence of epidermal allantoin further supports oxidative stress in vitiligo

Experimental dermatology, 2008-09, Vol.17 (9), p.761-770

2008

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Computer simulation of heterogeneous single nucleotide polymorphisms in the catalase gene indicates structural changes in the enzyme active site, NADPH-binding and tetramerization domains: a genetic predisposition for an altered catalase in patients with vitiligo?

Experimental dermatology, 2008-04, Vol.17 (4), p.366-371

2008

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Methionine Sulfoxide Reductases A and B Are Deactivated by Hydrogen Peroxide (H2O2) in the Epidermis of Patients with Vitiligo

Journal of investigative dermatology, 2008-04, Vol.128 (4), p.808-815

2008

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Perturbed 6-Tetrahydrobiopterin Recycling via Decreased Dihydropteridine Reductase in Vitiligo: More Evidence for H2O2 Stress

Journal of investigative dermatology, 2004-02, Vol.122 (2), p.307-313

2004

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KdPT, a tripeptide derivative of alpha-melanocyte-stimulating hormone, suppresses IL-1 beta-mediated cytokine expression and signaling in human sebocytes

The Journal of immunology (1950), 2010-08, Vol.185 (3), p.1903-1911

2010

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Butyrylcholinesterase is present in the human epidermis and is regulated by H2O2: more evidence for oxidative stress in vitiligo

Biochemical and biophysical research communications, 2006-10, Vol.349 (3), p.931-938

2006

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Calcium-activated butyrylcholinesterase in human skin protects acetylcholinesterase against suicide inhibition by neurotoxic organophosphates

Biochemical and biophysical research communications, 2007-04, Vol.355 (4), p.1069-1074

2007

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Structural and functional alterations in the β2-adrenoceptor are caused by a point mutation in patients with atopic eczema

Experimental dermatology, 2007-10, Vol.16 (10), p.807-813

2007

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Activation/deactivation of acetylcholinesterase by H2O2: more evidence for oxidative stress in vitiligo

Biochemical and biophysical research communications, 2004-03, Vol.315 (2), p.502-508

2004

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Structural and functional alterations in the beta 2-adrenoceptor are caused by a point mutation in patients with atopic eczema

Experimental dermatology, 2007-10, Vol.16 (10), p.807-813

2007

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Human phenylalanine hydroxylase is activated by H2O2: a novel mechanism for increasing the L-tyrosine supply for melanogenesis in melanocytes

Biochemical and biophysical research communications, 2004-09, Vol.322 (1), p.88-92

2004

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Enhanced DNA binding capacity on up‐regulated epidermal wild‐type p53 in vitiligo by H 2 O 2 ‐mediated oxidation: a possible repair mechanism for DNA damage

The FASEB journal, 2009-11, Vol.23 (11), p.3790-3807

2009

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K d PT, a Tripeptide Derivative of α-Melanocyte–Stimulating Hormone, Suppresses IL-1β–Mediated Cytokine Expression and Signaling in Human Sebocytes

The Journal of immunology (1950), 2010-08, Vol.185 (3), p.1903-1911

2010

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In situ and in vitro evidence for DCoH/HNF-1α transcription of tyrosinase in human skin melanocytes

Biochemical and biophysical research communications, 2003-02, Vol.301 (2), p.610-616

2003

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Structural and functional alterations in the beta2-adrenoceptor are caused by a point mutation in patients with atopic eczema

Experimental dermatology, 2007-10, Vol.16 (10), p.807-813

2007

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Identification, Design and Biological Evaluation of Bisaryl Quinolones Targeting Plasmodium falciparum Type II NADH:Quinone Oxidoreductase (PfNDH2)

Journal of medicinal chemistry, 2012-03, Vol.55 (5), p.1831-1843

2012

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Ultrahigh-throughput Generation and Characterization of Cellular Aggregates in Laser-ablated Microwells of Poly(dimethylsiloxane)

RSC advances, 2016-01, Vol.6 (11), p.8980-8991

2016

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Molybdenum catalysts based on salan ligands for the deoxydehydration reaction

Catalysis science & technology, 2024-07, Vol.14 (13), p.366-3673

2024

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Associations between Potentially Modifiable Risk Factors and Alzheimer Disease: A Mendelian Randomization Study

PLoS medicine, 2015-06, Vol.12 (6), p.e1001841; discussion e1001841

2015

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Effectiveness of fluticasone furoate plus vilanterol on asthma control in clinical practice: an open-label, parallel group, randomised controlled trial

The Lancet (British edition), 2017-11, Vol.390 (10109), p.2247-2255

2017

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Common variants at the MHC locus and at chromosome 16q24.1 predispose to Barrett's esophagus

Nature genetics, 2012-10, Vol.44 (10), p.1131-1136

2012

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Enhanced DNA binding capacity on up‐regulated epidermal wild‐type p53 in vitiligo by H2O2‐mediated oxidation: a possible repair mechanism for DNA damage

Ultra-high resolution X-ray structures of two forms of human recombinant insulin at 100 K

Oxidative Stress Via Hydrogen Peroxide Affects Proopiomelanocortin Peptides Directly in the Epidermis of Patients with Vitiligo

Computer Simulation of Native Epidermal Enzyme Structures in the Presence and Absence of Hydrogen Peroxide (H2O2): Potential and Pitfalls

Presence of epidermal allantoin further supports oxidative stress in vitiligo

Computer simulation of heterogeneous single nucleotide polymorphisms in the catalase gene indicates structural changes in the enzyme active site, NADPH-binding and tetramerization domains: a genetic predisposition for an altered catalase in patients with vitiligo?

Methionine Sulfoxide Reductases A and B Are Deactivated by Hydrogen Peroxide (H2O2) in the Epidermis of Patients with Vitiligo

Perturbed 6-Tetrahydrobiopterin Recycling via Decreased Dihydropteridine Reductase in Vitiligo: More Evidence for H2O2 Stress

KdPT, a tripeptide derivative of alpha-melanocyte-stimulating hormone, suppresses IL-1 beta-mediated cytokine expression and signaling in human sebocytes

Butyrylcholinesterase is present in the human epidermis and is regulated by H2O2: more evidence for oxidative stress in vitiligo

Calcium-activated butyrylcholinesterase in human skin protects acetylcholinesterase against suicide inhibition by neurotoxic organophosphates

Structural and functional alterations in the β2-adrenoceptor are caused by a point mutation in patients with atopic eczema

Activation/deactivation of acetylcholinesterase by H2O2: more evidence for oxidative stress in vitiligo

Structural and functional alterations in the beta 2-adrenoceptor are caused by a point mutation in patients with atopic eczema

Human phenylalanine hydroxylase is activated by H2O2: a novel mechanism for increasing the L-tyrosine supply for melanogenesis in melanocytes

Enhanced DNA binding capacity on up‐regulated epidermal wild‐type p53 in vitiligo by H 2 O 2 ‐mediated oxidation: a possible repair mechanism for DNA damage

K d PT, a Tripeptide Derivative of α-Melanocyte–Stimulating Hormone, Suppresses IL-1β–Mediated Cytokine Expression and Signaling in Human Sebocytes

In situ and in vitro evidence for DCoH/HNF-1α transcription of tyrosinase in human skin melanocytes

Structural and functional alterations in the beta2-adrenoceptor are caused by a point mutation in patients with atopic eczema

Identification, Design and Biological Evaluation of Bisaryl Quinolones Targeting Plasmodium falciparum Type II NADH:Quinone Oxidoreductase (PfNDH2)

Ultrahigh-throughput Generation and Characterization of Cellular Aggregates in Laser-ablated Microwells of Poly(dimethylsiloxane)

Molybdenum catalysts based on salan ligands for the deoxydehydration reaction

Associations between Potentially Modifiable Risk Factors and Alzheimer Disease: A Mendelian Randomization Study

Effectiveness of fluticasone furoate plus vilanterol on asthma control in clinical practice: an open-label, parallel group, randomised controlled trial

Common variants at the MHC locus and at chromosome 16q24.1 predispose to Barrett's esophagus

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