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Upregulation of DJ-1 in Dopaminergic Neurons by a Physically-Modified Saline: Implications for Parkinson's Disease

International journal of molecular sciences, 2023-02, Vol.24 (5), p.4652

2023

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A physically-modified saline suppresses neuronal apoptosis, attenuates tau phosphorylation and protects memory in an animal model of Alzheimer's disease

PloS one, 2014-08, Vol.9 (8), p.e103606

2014

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Neuroprotective effect of the RNS60 in a mouse model of transient focal cerebral ischemia

PloS one, 2024-01, Vol.19 (1), p.e0295504-e0295504

2024

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Protection of Tregs, suppression of Th1 and Th17 cells, and amelioration of experimental allergic encephalomyelitis by a physically-modified saline

PloS one, 2012-12, Vol.7 (12), p.e51869-e51869

2012

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RNS60 exerts therapeutic effects in the SOD1 ALS mouse model through protective glia and peripheral nerve rescue

Journal of neuroinflammation, 2018-03, Vol.15 (1), p.65-65, Article 65

2018

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Engagement of collagen-binding integrins promotes matrix metalloproteinase-9-dependent e-cadherin ectodomain shedding in ovarian carcinoma cells

Cancer research (Chicago, Ill.), 2007-03, Vol.67 (5), p.2030-2039

2007

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Correction: A Physically-Modified Saline Suppresses Neuronal Apoptosis, Attenuates Tau Phosphorylation and Protects Memory in an Animal Model of Alzheimer's Disease

PloS one, 2017-06, Vol.12 (6), p.e0180602-e0180602

2017

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Enhancement of morphological plasticity in hippocampal neurons by a physically modified saline via phosphatidylinositol-3 kinase

PloS one, 2014-07, Vol.9 (7), p.e101883

2014

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A Phase I Assessment Of Safety and Tolerability Of RNS60, a Novel Therapeutic Containing Charge-Stabilized Nanostructures In Asthma

Journal of allergy and clinical immunology, 2014-02, Vol.133 (2), p.AB4-AB4

2014

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Polypyrimidine Tract-binding Protein (PTB) Differentially Affects Malignancy in a Cell Line-dependent Manner

The Journal of biological chemistry, 2008-07, Vol.283 (29), p.20277-20287

2008

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RNS60, a physically-modified saline, inhibits glial activation, suppresses neuronal apoptosis and protects memory in a mouse model of traumatic brain injury

Experimental neurology, 2020-06, Vol.328, p.113279-113279, Article 113279

2020

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Functional Relevance of Urinary-type Plasminogen Activator Receptor-α3β1 Integrin Association in Proteinase Regulatory Pathways

The Journal of biological chemistry, 2006-05, Vol.281 (19), p.13021-13029

2006

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Upregulation of Myelin Gene Expression by a Physically-Modified Saline via Phosphatidylinositol 3-Kinase-Mediated Activation of CREB: Implications for Multiple Sclerosis

Neurochemical research, 2018-02, Vol.43 (2), p.407-419

2018

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Abstract WP259: Acute Stroke Neuroprotection With Intravenous Rns60 In The Cynomolgus Macaque

Stroke (1970), 2022-02, Vol.53 (Suppl_1)

2022

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Abstract TP219: Rns60 Brain Protection In Mouse Ischemic Stroke Mitigates Plasma Tau And Cortical Microvascular Perfusion

Stroke (1970), 2023-02, Vol.54 (Suppl_1)

2023

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Suppression of Nuclear Factor-κB Activation and Inflammation in Microglia by Physically Modified Saline

The Journal of biological chemistry, 2012-08, Vol.287 (35), p.29529-29542

2012

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Protection of Dopaminergic Neurons in a Mouse Model of Parkinson’s Disease by a Physically-Modified Saline Containing Charge-Stabilized Nanobubbles

Journal of neuroimmune pharmacology, 2014-03, Vol.9 (2), p.218-232

2014

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Abstract WP257: RNS60 Provides Acute And Chronic Protection Of Brain Cells And Function In A Mouse Stroke Model

Stroke (1970), 2022-02, Vol.53 (Suppl_1)

2022

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Proteinase Suppression by E-cadherin-mediated Cell-Cell Attachment in Premalignant Oral Keratinocytes

The Journal of biological chemistry, 2002-10, Vol.277 (41), p.38159-38167

2002

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Priorities for Advancements in Neuroimaging in the Diagnostic Workup of Acute Stroke

Stroke (1970), 2023-12, Vol.54 (12), p.3190

2023

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Increase in Mitochondrial Biogenesis in Neuronal Cells by RNS60, a Physically-Modified Saline, via Phosphatidylinositol 3-Kinase-Mediated Upregulation of PGC1α

Journal of neuroimmune pharmacology, 2018-06, Vol.13 (2), p.143-162

2018

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Proteolytic modification of laminins: Functional consequences

Microscopy research and technique, 2000-11, Vol.51 (3), p.238-246

2000

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Nebulization of RNS60, a Physically-Modified Saline, Attenuates the Adoptive Transfer of Experimental Allergic Encephalomyelitis in Mice: Implications for Multiple Sclerosis Therapy

Neurochemical research, 2017-05, Vol.42 (5), p.1555-1570

2017

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Correction to: Nebulization of RNS60, a Physically-Modified Saline, Attenuates the Adoptive Transfer of Experimental Allergic Encephalomyelitis in Mice: Implications for Multiple Sclerosis Therapy

Neurochemical research, 2018-05, Vol.43 (5), p.1136-1139

2018

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Abstract 18804: A Saline Containing Charge-Stabilized Nanostructures Inhibits Apoptosis of Cardiomyocytes Subjected to Ischemia/Reperfusion Injury

Circulation (New York, N.Y.), 2013-11, Vol.128 (suppl_22)

2013

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Upregulation of DJ-1 in Dopaminergic Neurons by a Physically-Modified Saline: Implications for Parkinson's Disease

A physically-modified saline suppresses neuronal apoptosis, attenuates tau phosphorylation and protects memory in an animal model of Alzheimer's disease

Neuroprotective effect of the RNS60 in a mouse model of transient focal cerebral ischemia

Protection of Tregs, suppression of Th1 and Th17 cells, and amelioration of experimental allergic encephalomyelitis by a physically-modified saline

RNS60 exerts therapeutic effects in the SOD1 ALS mouse model through protective glia and peripheral nerve rescue

Engagement of collagen-binding integrins promotes matrix metalloproteinase-9-dependent e-cadherin ectodomain shedding in ovarian carcinoma cells

Correction: A Physically-Modified Saline Suppresses Neuronal Apoptosis, Attenuates Tau Phosphorylation and Protects Memory in an Animal Model of Alzheimer's Disease

Enhancement of morphological plasticity in hippocampal neurons by a physically modified saline via phosphatidylinositol-3 kinase

A Phase I Assessment Of Safety and Tolerability Of RNS60, a Novel Therapeutic Containing Charge-Stabilized Nanostructures In Asthma

Polypyrimidine Tract-binding Protein (PTB) Differentially Affects Malignancy in a Cell Line-dependent Manner

RNS60, a physically-modified saline, inhibits glial activation, suppresses neuronal apoptosis and protects memory in a mouse model of traumatic brain injury

Functional Relevance of Urinary-type Plasminogen Activator Receptor-α3β1 Integrin Association in Proteinase Regulatory Pathways

Upregulation of Myelin Gene Expression by a Physically-Modified Saline via Phosphatidylinositol 3-Kinase-Mediated Activation of CREB: Implications for Multiple Sclerosis

Abstract WP259: Acute Stroke Neuroprotection With Intravenous Rns60 In The Cynomolgus Macaque

Abstract TP219: Rns60 Brain Protection In Mouse Ischemic Stroke Mitigates Plasma Tau And Cortical Microvascular Perfusion

Suppression of Nuclear Factor-κB Activation and Inflammation in Microglia by Physically Modified Saline

Protection of Dopaminergic Neurons in a Mouse Model of Parkinson’s Disease by a Physically-Modified Saline Containing Charge-Stabilized Nanobubbles

Abstract WP257: RNS60 Provides Acute And Chronic Protection Of Brain Cells And Function In A Mouse Stroke Model

Proteinase Suppression by E-cadherin-mediated Cell-Cell Attachment in Premalignant Oral Keratinocytes

Priorities for Advancements in Neuroimaging in the Diagnostic Workup of Acute Stroke

Increase in Mitochondrial Biogenesis in Neuronal Cells by RNS60, a Physically-Modified Saline, via Phosphatidylinositol 3-Kinase-Mediated Upregulation of PGC1α

Proteolytic modification of laminins: Functional consequences

Nebulization of RNS60, a Physically-Modified Saline, Attenuates the Adoptive Transfer of Experimental Allergic Encephalomyelitis in Mice: Implications for Multiple Sclerosis Therapy

Correction to: Nebulization of RNS60, a Physically-Modified Saline, Attenuates the Adoptive Transfer of Experimental Allergic Encephalomyelitis in Mice: Implications for Multiple Sclerosis Therapy

Abstract 18804: A Saline Containing Charge-Stabilized Nanostructures Inhibits Apoptosis of Cardiomyocytes Subjected to Ischemia/Reperfusion Injury

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