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Personalized medicine: Vinpocetine to reverse effects of GABRB3 mutation

Epilepsia (Copenhagen), 2019-12, Vol.60 (12), p.2459-2465

2019

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The KCNQ2/3 selective channel opener ICA-27243 binds to a novel voltage-sensor domain site

Neuroscience letters, 2009-11, Vol.465 (2), p.138-142

2009

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Pharmacological Removal of Human Ether-Ã -go-go-Related Gene Potassium Channel Inactivation by 3-Nitro-N-(4-phenoxyphenyl) Benzamide (ICA-105574)

Molecular pharmacology, 2010-01, Vol.77 (1), p.58-68

2010

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TRPA1 ankyrin repeat six interacts with a small molecule inhibitor chemotype

Proceedings of the National Academy of Sciences - PNAS, 2018-11, Vol.115 (48), p.12301-12306

2018

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A comprehensive approach to identifying repurposed drugs to treat SCN8A epilepsy

Epilepsia (Copenhagen), 2018-04, Vol.59 (4), p.802-813

2018

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A novel selective and orally bioavailable Nav1.8 channel blocker, PF‐01247324, attenuates nociception and sensory neuron excitability

British journal of pharmacology, 2015-05, Vol.172 (10), p.2654-2670

2015

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Pain relief in a neuropathy patient by lacosamide: Proof of principle of clinical translation from patient-specific iPS cell-derived nociceptors

EBioMedicine, 2019-01, Vol.39, p.401-408

2019

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Small Conductance Ca2+-Activated K+ Channels Modulate Synaptic Plasticity and Memory Encoding

The Journal of neuroscience, 2002-12, Vol.22 (23), p.10163-10171

2002

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Validation of ion channel targets

Channels (Austin, Tex.), 2015-11, Vol.9 (6), p.376-379

2015

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Discovery of a Series of Indazole TRPA1 Antagonists

ACS medicinal chemistry letters, 2017-06, Vol.8 (6), p.666-671

2017

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Antiepileptic Drug Discovery and Development: What Have We Learned and Where Are We Going?

Pharmaceuticals, 2010-09, Vol.3 (9), p.2884-2899

2010

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Repurposing Approved Drugs as Inhibitors of Kv7.1 and Nav1.8 to Treat Pitt Hopkins Syndrome

Pharmaceutical research, 2019-09, Vol.36 (9), p.1-10, Article 137

2019

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Trafficking of the Ca2+-activated K+Channel, hIK1, Is Dependent upon a C-terminal Leucine Zipper

The Journal of biological chemistry, 2003-03, Vol.278 (10), p.8476-8486

2003

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Activation kinetics of the slow afterhyperpolarization in hippocampal CA1 neurons

Pflügers Archiv, 2004-05, Vol.448 (2), p.187-196

2004

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Repurposing Approved Drugs as Inhibitors of K.sub.v7.1 and Na.sub.v1.8 to Treat Pitt Hopkins Syndrome

Pharmaceutical research, 2019-09, Vol.36 (9)

2019

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Residues at TRPA1 S4-S5 Linker N-Terminus Are Critical for Translating Covalent Modification to Channel Activation

Biophysical journal, 2018-02, Vol.114 (3), p.397a-397a

2018

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Kinase-dependent Regulation of the Intermediate Conductance, Calcium-dependent Potassium Channel, hIK1

The Journal of biological chemistry, 2000-01, Vol.275 (1), p.585-598

2000

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Pharmacological activation of cloned intermediate- and small-conductance Ca2+-activated K+ channels

American Journal of Physiology: Cell Physiology, 2000-03, Vol.278 (3), p.C570

2000

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ATP-dependent Activation of the Intermediate Conductance, Ca2+-activated K+ Channel, hIK1, Is Conferred by a C-terminal Domain

The Journal of biological chemistry, 2001-04, Vol.276 (14), p.10963-10970

2001

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ΔKPQ Nav1.5 channels for late current cardiac pharmacology assessment

Journal of pharmacological and toxicological methods, 2018-09, Vol.93, p.116-116

2018

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Repurposing Approved Drugs as Inhibitors of K v 7.1 and Na v 1.8 to Treat Pitt Hopkins Syndrome

Pharmaceutical research, 2019-07, Vol.36 (9), p.137

2019

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A comprehensive approach to identifying repurposed drugs to treat SCN 8A epilepsy

Epilepsia (Copenhagen), 2018-04, Vol.59 (4), p.802-813

2018

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A novel selective and orally bioavailable Na sub(v)1.8 channel blocker, PF-01247324, attenuates nociception and sensory neuron excitability

British journal of pharmacology, 2015-05, Vol.172 (10), p.2654-2670

2015

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A novel selective and orally bioavailable N a v 1.8 channel blocker, PF ‐01247324, attenuates nociception and sensory neuron excitability

British journal of pharmacology, 2015-05, Vol.172 (10), p.2654-2670

2015

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Pharmacological activation of cloned intermediate- and small-conductance Ca(2+)-activated K(+) channels

American Journal of Physiology: Cell Physiology, 2000-03, Vol.278 (3), p.C570-C581

2000

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Personalized medicine: Vinpocetine to reverse effects of GABRB3 mutation

The KCNQ2/3 selective channel opener ICA-27243 binds to a novel voltage-sensor domain site

Pharmacological Removal of Human Ether-Ã -go-go-Related Gene Potassium Channel Inactivation by 3-Nitro-N-(4-phenoxyphenyl) Benzamide (ICA-105574)

TRPA1 ankyrin repeat six interacts with a small molecule inhibitor chemotype

A comprehensive approach to identifying repurposed drugs to treat SCN8A epilepsy

A novel selective and orally bioavailable Nav1.8 channel blocker, PF‐01247324, attenuates nociception and sensory neuron excitability

Pain relief in a neuropathy patient by lacosamide: Proof of principle of clinical translation from patient-specific iPS cell-derived nociceptors

Small Conductance Ca2+-Activated K+ Channels Modulate Synaptic Plasticity and Memory Encoding

Validation of ion channel targets

Discovery of a Series of Indazole TRPA1 Antagonists

Antiepileptic Drug Discovery and Development: What Have We Learned and Where Are We Going?

Repurposing Approved Drugs as Inhibitors of Kv7.1 and Nav1.8 to Treat Pitt Hopkins Syndrome

Trafficking of the Ca2+-activated K+Channel, hIK1, Is Dependent upon a C-terminal Leucine Zipper

Activation kinetics of the slow afterhyperpolarization in hippocampal CA1 neurons

Repurposing Approved Drugs as Inhibitors of K.sub.v7.1 and Na.sub.v1.8 to Treat Pitt Hopkins Syndrome

Residues at TRPA1 S4-S5 Linker N-Terminus Are Critical for Translating Covalent Modification to Channel Activation

Kinase-dependent Regulation of the Intermediate Conductance, Calcium-dependent Potassium Channel, hIK1

Pharmacological activation of cloned intermediate- and small-conductance Ca2+-activated K+ channels

ATP-dependent Activation of the Intermediate Conductance, Ca2+-activated K+ Channel, hIK1, Is Conferred by a C-terminal Domain

ΔKPQ Nav1.5 channels for late current cardiac pharmacology assessment

Repurposing Approved Drugs as Inhibitors of K v 7.1 and Na v 1.8 to Treat Pitt Hopkins Syndrome

A comprehensive approach to identifying repurposed drugs to treat SCN 8A epilepsy

A novel selective and orally bioavailable Na sub(v)1.8 channel blocker, PF-01247324, attenuates nociception and sensory neuron excitability

A novel selective and orally bioavailable N a v 1.8 channel blocker, PF ‐01247324, attenuates nociception and sensory neuron excitability

Pharmacological activation of cloned intermediate- and small-conductance Ca(2+)-activated K(+) channels

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