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Differentiation of Intervertebral Notochordal Cells Through Live Automated Cell Imaging System In Vitro

Spine (Philadelphia, Pa. 1976), 2009-11, Vol.34 (23), p.2486-2493

2009

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Gene transfer of the catabolic inhibitor TIMP-1 increases measured proteoglycans in cells from degenerated human intervertebral discs

Spine (Philadelphia, Pa. 1976), 2003-10, Vol.28 (20), p.2331-2337

2003

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Effect of growth factors on matrix synthesis by ligament fibroblasts

Journal of orthopaedic research, 1997-01, Vol.15 (1), p.18-23

1997

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Does Nitric Oxide Help Explain the Differential Healing Capacity of the Anterior Cruciate, Posterior Cruciate, and Medial Collateral Ligaments?

American journal of sports medicine, 2000-03, Vol.28 (2), p.176-182

2000

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The biochemical and histological effects of artificial ligament wear particles: In vitro and in vivo studies

The American journal of sports medicine, 1988-11, Vol.16 (6), p.558-570

1988

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Poster 172: Acute Effects of Botulinum Neurotoxin-A (BoNT-A) on In Vitro Rabbit Articular Chondrocytes

Archives of physical medicine and rehabilitation, 2008, Vol.89 (11), p.e77-e77

2008

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Early expression of marker genes in the rabbit medial collateral and anterior cruciate ligaments: The use of different viral vectors and the effects of injury

Journal of orthopaedic research, 1999-01, Vol.17 (1), p.37-42

1999

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Approaches to enhancing the retroviral transduction of human synoviocytes

Arthritis research, 2001-01, Vol.3 (4), p.259-263, Article 259

2001

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The effects of age on rabbit MCL fibroblast matrix synthesis in response to TGF- β1 or EGF

Mechanisms of ageing and development, 1997, Vol.97 (2), p.121-130

1997

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Herniated lumbar intervertebral discs spontaneously produce matrix metalloproteinases, nitric oxide, interleukin-6, and prostaglandin E2

Spine (Philadelphia, Pa. 1976), 1996-02, Vol.21 (3), p.271-277

1996

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Toward a biochemical understanding of human intervertebral disc degeneration and herniation contributions of nitric oxide, interleukins, prostaglandin E2, and matrix metalloproteinases

Spine (Philadelphia, Pa. 1976), 1997-05, Vol.22 (10), p.1065-1073

1997

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Increased matrix synthesis following adenoviral transfer of a transforming growth factor β1 gene into articular chondrocytes

Journal of orthopaedic research, 2000-07, Vol.18 (4), p.585-592

2000

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HIG-82: An Established Cell Line from Rabbit Periarticular Soft Tissue, Which Retains the "Activatable" Phenotype

In Vitro Cellular & Developmental Biology, 1988-10, Vol.24 (10), p.1015-1022

1988

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Suppression of antigen-induced arthritis in rabbits by ex vivo gene therapy

The Journal of immunology (1950), 1996-05, Vol.156 (9), p.3558-3562

1996

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Herniated cervical intervertebral discs spontaneously produce matrix metalloproteinases, nitric oxide, interleukin-6, and prostaglandin E2

Spine (Philadelphia, PA. 1976), 1995-11, Vol.20 (22), p.2373-2378

1995

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Articular chondrocytes synthesize nitric oxide in response to cytokines and lipopolysaccharide

The Journal of immunology (1950), 1991-12, Vol.147 (11), p.3915-3920

1991

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Constitutive intra-articular expression of human IL-1 beta following gene transfer to rabbit synovium produces all major pathologies of human rheumatoid arthritis

The Journal of immunology (1950), 1997-10, Vol.159 (7), p.3604-3612

1997

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Chondrocyte activation in response to factor(s) produced by a continuous line of lapine synovial fibroblasts

Experimental cell research, 1986-11, Vol.167 (1), p.218-226

1986

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Ex vivo gene transfer to chondrocytes in full-thickness articular cartilage defects: a feasibility study

Osteoarthritis and cartilage, 1997-03, Vol.5 (2), p.139-143

1997

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Observations on the senescence of cells derived from articular cartilage

Mechanisms of ageing and development, 1983-06, Vol.22 (2), p.179-191

1983

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Increased matrix synthesis following adenoviral transfer of a transforming growth factor (beta 1) gene into articular chondrocytes

Journal of orthopaedic research, 2000-07, Vol.18 (4), p.585

2000

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Increased matrix synthesis following adenoviral transfer of a transforming growth factor beta sub(1) gene into articular chondrocytes

Journal of orthopaedic research, 2000-07, Vol.18 (4), p.585-592

2000

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Increased matrix synthesis following adenoviral transfer of a transforming growth factor β 1 gene into articular chondrocytes

Journal of orthopaedic research, 2000-07, Vol.18 (4), p.585-592

2000

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In vivo suppression of early experimental osteoarthritis by interleukin‐1 receptor antagonist using gene therapy

Arthritis and rheumatism, 1997-06, Vol.40 (6), p.1012-1019

1997

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Inducible synthesis of collagenase and other neutral metalloproteinases by cells of aortic origin

The Journal of surgical research, 1991-11, Vol.51 (5), p.399-404

1991

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Differentiation of Intervertebral Notochordal Cells Through Live Automated Cell Imaging System In Vitro

Gene transfer of the catabolic inhibitor TIMP-1 increases measured proteoglycans in cells from degenerated human intervertebral discs

Effect of growth factors on matrix synthesis by ligament fibroblasts

Does Nitric Oxide Help Explain the Differential Healing Capacity of the Anterior Cruciate, Posterior Cruciate, and Medial Collateral Ligaments?

The biochemical and histological effects of artificial ligament wear particles: In vitro and in vivo studies

Poster 172: Acute Effects of Botulinum Neurotoxin-A (BoNT-A) on In Vitro Rabbit Articular Chondrocytes

Early expression of marker genes in the rabbit medial collateral and anterior cruciate ligaments: The use of different viral vectors and the effects of injury

Approaches to enhancing the retroviral transduction of human synoviocytes

The effects of age on rabbit MCL fibroblast matrix synthesis in response to TGF- β1 or EGF

Herniated lumbar intervertebral discs spontaneously produce matrix metalloproteinases, nitric oxide, interleukin-6, and prostaglandin E2

Toward a biochemical understanding of human intervertebral disc degeneration and herniation contributions of nitric oxide, interleukins, prostaglandin E2, and matrix metalloproteinases

Increased matrix synthesis following adenoviral transfer of a transforming growth factor β1 gene into articular chondrocytes

HIG-82: An Established Cell Line from Rabbit Periarticular Soft Tissue, Which Retains the "Activatable" Phenotype

Suppression of antigen-induced arthritis in rabbits by ex vivo gene therapy

Herniated cervical intervertebral discs spontaneously produce matrix metalloproteinases, nitric oxide, interleukin-6, and prostaglandin E2

Articular chondrocytes synthesize nitric oxide in response to cytokines and lipopolysaccharide

Constitutive intra-articular expression of human IL-1 beta following gene transfer to rabbit synovium produces all major pathologies of human rheumatoid arthritis

Chondrocyte activation in response to factor(s) produced by a continuous line of lapine synovial fibroblasts

Ex vivo gene transfer to chondrocytes in full-thickness articular cartilage defects: a feasibility study

Observations on the senescence of cells derived from articular cartilage

Increased matrix synthesis following adenoviral transfer of a transforming growth factor (beta 1) gene into articular chondrocytes

Increased matrix synthesis following adenoviral transfer of a transforming growth factor beta sub(1) gene into articular chondrocytes

Increased matrix synthesis following adenoviral transfer of a transforming growth factor β 1 gene into articular chondrocytes

In vivo suppression of early experimental osteoarthritis by interleukin‐1 receptor antagonist using gene therapy

Inducible synthesis of collagenase and other neutral metalloproteinases by cells of aortic origin

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