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JC Virus infected choroid plexus epithelial cells produce extracellular vesicles that infect glial cells independently of the virus attachment receptor

PLoS pathogens, 2020-03, Vol.16 (3), p.e1008371-e1008371

2020

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JC Polyomavirus Uses Extracellular Vesicles To Infect Target Cells

mBio, 2019-04, Vol.10 (2)

2019

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Genetic and Functional Dissection of the Role of Individual 5-HT2 Receptors as Entry Receptors for JC Polyomavirus

Cell reports (Cambridge), 2019-05, Vol.27 (7), p.1960-1966.e6

2019

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5-HT2 Receptors Facilitate JC Polyomavirus Entry

Journal of Virology, 2013-12, Vol.87 (24), p.13490-13498

2013

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The Greater Affinity of JC Polyomavirus Capsid for α2,6-Linked Lactoseries Tetrasaccharide c than for Other Sialylated Glycans Is a Major Determinant of Infectivity

Journal of virology, 2015-06, Vol.89 (12), p.6364-6375

2015

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The Human Polyomavirus, JCV, Uses Serotonin Receptors to Infect Cells

Science (American Association for the Advancement of Science), 2004-11, Vol.306 (5700), p.1380-1383

2004

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Trichodysplasia spinulosa-Associated Polyomavirus Uses a Displaced Binding Site on VP1 to Engage Sialylated Glycolipids

PLoS pathogens, 2015-08, Vol.11 (8), p.e1005112

2015

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Teriflunomide Inhibits JCPyV Infection and Spread in Glial Cells and Choroid Plexus Epithelial Cells

International journal of molecular sciences, 2021-09, Vol.22 (18), p.9809

2021

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Phosphoinositide 3'-Kinase γ Facilitates Polyomavirus Infection

Viruses, 2020-10, Vol.12 (10), p.1190

2020

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Downregulated microRNAs in the differential diagnosis of malignant pleural mesothelioma

International journal of cancer, 2010-12, Vol.127 (12), p.2859-2869

2010

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Interaction between Simian Virus 40 Major Capsid Protein VP1 and Cell Surface Ganglioside GM1 Triggers Vacuole Formation

mBio, 2016-03, Vol.7 (2), p.e00297-e00297

2016

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A retrograde trafficking inhibitor of ricin and Shiga-like toxins inhibits infection of cells by human and monkey polyomaviruses

mBio, 2013-11, Vol.4 (6), p.e00729

2013

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Role of N-Linked Glycosylation of the 5-HT2A Receptor in JC Virus Infection

Journal of Virology, 2010-10, Vol.84 (19), p.9677-9684

2010

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Progressive multifocal leukoencephalopathy-associated mutations in the JC polyomavirus capsid disrupt lactoseries tetrasaccharide c binding

mBio, 2013-06, Vol.4 (3), p.e00247-e00213

2013

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Biogenesis of JC polyomavirus associated extracellular vesicles

Journal of extracellular biology, 2022-05, Vol.1 (5), p.n/a

2022

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Separation of full, empty, and partial adeno-associated virus capsids via anion-exchange chromatography with continuous recycling and accumulation

Journal of chromatography. B, Analytical technologies in the biomedical and life sciences, 2024-07, Vol.1242, p.124206, Article 124206

2024

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Susceptibility of Primary Human Choroid Plexus Epithelial Cells and Meningeal Cells to Infection by JC Virus

Journal of virology, 2018-04, Vol.92 (8)

2018

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NFAT4 Is Required for JC Virus Infection of Glial Cells

Journal of Virology, 2006-12, Vol.80 (24), p.12079-12085

2006

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The Greater Affinity of JC Polyomavirus Capsid for alpha 2,6-Linked Lactoseries Tetrasaccharide c than for Other Sialylated Glycans Is a Major Determinant of Infectivity

Journal of virology, 2015-06, Vol.89 (12), p.6364-6375

2015

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Modeling a Sialic Acid Binding Pocket in the External Loops of JC Virus VP1

The Journal of biological chemistry, 2004-11, Vol.279 (47), p.49172-49176

2004

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The role of sialic acid in human polyomavirus infections

Glycoconjugate journal, 2006-02, Vol.23 (1-2), p.19-26

2006

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Pseudovirus mimics cell entry and trafficking of the human polyomavirus JCPyV

Virus research, 2013-12, Vol.178 (2), p.281-286

2013

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Modulation of a pore in the capsid of JC polyomavirus reduces infectivity and prevents exposure of the minor capsid proteins

Journal of virology, 2015-04, Vol.89 (7), p.3910-3921

2015

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Host cell autophagy promotes BK virus infection

Virology (New York, N.Y.), 2014-05, Vol.456, p.87-95

2014

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Microarray analysis of glial cells resistant to JCV infection suggests a correlation between viral infection and inflammatory cytokine gene expression

Virology (New York, N.Y.), 2007-09, Vol.366 (2), p.394-404

2007

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JC Virus infected choroid plexus epithelial cells produce extracellular vesicles that infect glial cells independently of the virus attachment receptor

JC Polyomavirus Uses Extracellular Vesicles To Infect Target Cells

Genetic and Functional Dissection of the Role of Individual 5-HT2 Receptors as Entry Receptors for JC Polyomavirus

5-HT2 Receptors Facilitate JC Polyomavirus Entry

The Greater Affinity of JC Polyomavirus Capsid for α2,6-Linked Lactoseries Tetrasaccharide c than for Other Sialylated Glycans Is a Major Determinant of Infectivity

The Human Polyomavirus, JCV, Uses Serotonin Receptors to Infect Cells

Trichodysplasia spinulosa-Associated Polyomavirus Uses a Displaced Binding Site on VP1 to Engage Sialylated Glycolipids

Teriflunomide Inhibits JCPyV Infection and Spread in Glial Cells and Choroid Plexus Epithelial Cells

Phosphoinositide 3'-Kinase γ Facilitates Polyomavirus Infection

Downregulated microRNAs in the differential diagnosis of malignant pleural mesothelioma

Interaction between Simian Virus 40 Major Capsid Protein VP1 and Cell Surface Ganglioside GM1 Triggers Vacuole Formation

A retrograde trafficking inhibitor of ricin and Shiga-like toxins inhibits infection of cells by human and monkey polyomaviruses

Role of N-Linked Glycosylation of the 5-HT2A Receptor in JC Virus Infection

Progressive multifocal leukoencephalopathy-associated mutations in the JC polyomavirus capsid disrupt lactoseries tetrasaccharide c binding

Biogenesis of JC polyomavirus associated extracellular vesicles

Separation of full, empty, and partial adeno-associated virus capsids via anion-exchange chromatography with continuous recycling and accumulation

Susceptibility of Primary Human Choroid Plexus Epithelial Cells and Meningeal Cells to Infection by JC Virus

NFAT4 Is Required for JC Virus Infection of Glial Cells

The Greater Affinity of JC Polyomavirus Capsid for alpha 2,6-Linked Lactoseries Tetrasaccharide c than for Other Sialylated Glycans Is a Major Determinant of Infectivity

Modeling a Sialic Acid Binding Pocket in the External Loops of JC Virus VP1

The role of sialic acid in human polyomavirus infections

Pseudovirus mimics cell entry and trafficking of the human polyomavirus JCPyV

Modulation of a pore in the capsid of JC polyomavirus reduces infectivity and prevents exposure of the minor capsid proteins

Host cell autophagy promotes BK virus infection

Microarray analysis of glial cells resistant to JCV infection suggests a correlation between viral infection and inflammatory cytokine gene expression

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