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Sequential Application of Anticancer Drugs Enhances Cell Death by Rewiring Apoptotic Signaling Networks

Cell, 2012-05, Vol.149 (4), p.780-794

2012

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Robust Activity of Avapritinib, Potent and Highly Selective Inhibitor of Mutated KIT, in Patient-derived Xenograft Models of Gastrointestinal Stromal Tumors

Clinical cancer research, 2019-01, Vol.25 (2), p.609-618

2019

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Structural determinants of 14-3-3 binding specificities and regulation of subcellular localization of 14-3-3-ligand complexes: A comparison of the X-ray crystal structures of all human 14-3-3 isoforms

Seminars in cancer biology, 2006-06, Vol.16 (3), p.173-182

2006

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14-3-3 proteins as signaling integration points for cell cycle control and apoptosis

Seminars in cell & developmental biology, 2011-09, Vol.22 (7), p.688-695

2011

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A precision therapy against cancers driven by KIT/PDGFRA mutations

Science translational medicine, 2017-11, Vol.9 (414)

2017

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Targeting kinases with precision

Molecular & cellular oncology, 2018-05, Vol.5 (3), p.e1435183-e1435183

2018

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Inhibitory effects of midostaurin and avapritinib on myeloid progenitors derived from patients with KIT D816V positive advanced systemic mastocytosis

Leukemia, 2019-05, Vol.33 (5), p.1195-1205

2019

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A Mitotic Phosphorylation Feedback Network Connects Cdk1, Plk1, 53BP1, and Chk2 to Inactivate the G2/M DNA Damage Checkpoint

PLoS biology, 2010-01, Vol.8 (1), p.e1000287

2010

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Transient Non-native Hydrogen Bonds Promote Activation of a Signaling Protein

Cell, 2009-12, Vol.139 (6), p.1109-1118

2009

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Functional Dynamics of Response Regulators Using NMR Relaxation Techniques

Methods in Enzymology, 2007, Vol.423, p.149-165

2007

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Cell-free Formation of RNA Granules: Low Complexity Sequence Domains Form Dynamic Fibers within Hydrogels

Cell, 2012-05, Vol.149 (4), p.753-767

2012

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Abstract B130: Elevated cancer-intrinsic type I interferon signaling confers a dependency on the RNA editor ADAR1

Molecular cancer therapeutics, 2019-12, Vol.18 (12_Supplement), p.B130-B130

2019

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Abstract A112: Drug discovery efforts on the RNA protein methyltransferase METTL3/METTL14

Molecular cancer therapeutics, 2019-12, Vol.18 (12_Supplement), p.A112-A112

2019

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Abstract 2081: Robust activity of BLU-285, a potent and highly selective inhibitor of mutant KIT and PDGFRα, in patient-derived xenograft (PDX) models of gastrointestinal stromal tumor (GIST)

Cancer research (Chicago, Ill.), 2017-07, Vol.77 (13_Supplement), p.2081-2081

2017

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Efficacy of BLU-285, a novel, potent inhibitor of Exon 17 Mutant KIT and PDGFRA D842V, in patient-derived xenograft model of gastrointestinal stromal tumor (GIST)

Journal of clinical oncology, 2016-05, Vol.34 (15_suppl), p.11030-11030

2016

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Abstract 791: BLU-285, the first selective inhibitor of PDGFRα D842V and KIT Exon 17 mutants

Cancer research (Chicago, Ill.), 2015-08, Vol.75 (15_Supplement), p.791-791

2015

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Sequential Application of Anti-Cancer Drugs Enhances Cell Death by Re-wiring Apoptotic Signaling Networks

Cell, 2012-05, Vol.149 (4), p.780-794

2012

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The NMR Solution Structure of BeF3−-Activated Spo0F Reveals the Conformational Switch in a Phosphorelay System

Journal of molecular biology, 2003-08, Vol.331 (1), p.245-254

2003

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A Mitotic Phosphorylation Feedback Network Connects Cdk1, Plk1, 53BP1, and Chk2 to Inactivate the G2/M DNA Damage Checkpoint: e1000287

PLoS biology, 2010-01, Vol.8 (1)

2010

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The NMR solution structure of BeF(3)(-)-activated Spo0F reveals the conformational switch in a phosphorelay system

Journal of molecular biology, 2003-08, Vol.331 (1), p.245-254

2003

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The NMR Solution Structure of BeF 3−-Activated Spo0F Reveals the Conformational Switch in a Phosphorelay System

Journal of molecular biology, 2003, Vol.331 (1), p.245-254

2003

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Inhibitory Effects of Midostaurin and Blu-285 on Myeloid Progenitor Cells Derived from Patients with Multi-Mutated KIT D816V+ Advanced Systemic Mastocytosis

Blood, 2016-12, Vol.128 (22), p.1964-1964

2016

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First Selective KIT D816V Inhibitor for Patients with Systemic Mastocytosis

Blood, 2014-12, Vol.124 (21), p.3217-3217

2014

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Sequential Application of Anticancer Drugs Enhances Cell Death by Rewiring Apoptotic Signaling Networks

Robust Activity of Avapritinib, Potent and Highly Selective Inhibitor of Mutated KIT, in Patient-derived Xenograft Models of Gastrointestinal Stromal Tumors

Structural determinants of 14-3-3 binding specificities and regulation of subcellular localization of 14-3-3-ligand complexes: A comparison of the X-ray crystal structures of all human 14-3-3 isoforms

14-3-3 proteins as signaling integration points for cell cycle control and apoptosis

A precision therapy against cancers driven by KIT/PDGFRA mutations

Targeting kinases with precision

Inhibitory effects of midostaurin and avapritinib on myeloid progenitors derived from patients with KIT D816V positive advanced systemic mastocytosis

A Mitotic Phosphorylation Feedback Network Connects Cdk1, Plk1, 53BP1, and Chk2 to Inactivate the G2/M DNA Damage Checkpoint

Transient Non-native Hydrogen Bonds Promote Activation of a Signaling Protein

Functional Dynamics of Response Regulators Using NMR Relaxation Techniques

Cell-free Formation of RNA Granules: Low Complexity Sequence Domains Form Dynamic Fibers within Hydrogels

Abstract B130: Elevated cancer-intrinsic type I interferon signaling confers a dependency on the RNA editor ADAR1

Abstract A112: Drug discovery efforts on the RNA protein methyltransferase METTL3/METTL14

Abstract 2081: Robust activity of BLU-285, a potent and highly selective inhibitor of mutant KIT and PDGFRα, in patient-derived xenograft (PDX) models of gastrointestinal stromal tumor (GIST)

Efficacy of BLU-285, a novel, potent inhibitor of Exon 17 Mutant KIT and PDGFRA D842V, in patient-derived xenograft model of gastrointestinal stromal tumor (GIST)

Abstract 791: BLU-285, the first selective inhibitor of PDGFRα D842V and KIT Exon 17 mutants

Sequential Application of Anti-Cancer Drugs Enhances Cell Death by Re-wiring Apoptotic Signaling Networks

The NMR Solution Structure of BeF3−-Activated Spo0F Reveals the Conformational Switch in a Phosphorelay System

A Mitotic Phosphorylation Feedback Network Connects Cdk1, Plk1, 53BP1, and Chk2 to Inactivate the G2/M DNA Damage Checkpoint: e1000287

The NMR solution structure of BeF(3)(-)-activated Spo0F reveals the conformational switch in a phosphorelay system

The NMR Solution Structure of BeF 3−-Activated Spo0F Reveals the Conformational Switch in a Phosphorelay System

Inhibitory Effects of Midostaurin and Blu-285 on Myeloid Progenitor Cells Derived from Patients with Multi-Mutated KIT D816V+ Advanced Systemic Mastocytosis

First Selective KIT D816V Inhibitor for Patients with Systemic Mastocytosis

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