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Chemokines in neuron–glial cell interaction and pathogenesis of neuropathic pain

Cellular and molecular life sciences : CMLS, 2017-09, Vol.74 (18), p.3275-3291

2017

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Targeting Astrocyte Signaling for Chronic Pain

Neurotherapeutics, 2010-10, Vol.7 (4), p.482-493

2010

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Chemokine contribution to neuropathic pain: Respective induction of CXCL1 and CXCR2 in spinal cord astrocytes and neurons

Pain (Amsterdam), 2013-10, Vol.154 (10), p.2185-2197

2013

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Toll-like receptor 4 contributes to chronic itch, alloknesis, and spinal astrocyte activation in male mice

Pain (Amsterdam), 2016-04, Vol.157 (4), p.806-817

2016

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Chemokine CXCL13 mediates orofacial neuropathic pain via CXCR5/ERK pathway in the trigeminal ganglion of mice

Journal of neuroinflammation, 2016-07, Vol.13 (1), p.183-183, Article 183

2016

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A long noncoding RNA contributes to neuropathic pain by silencing Kcna2 in primary afferent neurons

Nature neuroscience, 2013-08, Vol.16 (8), p.1024-1031

2013

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Paclitaxel-activated astrocytes produce mechanical allodynia in mice by releasing tumor necrosis factor-α and stromal-derived cell factor 1

Journal of neuroinflammation, 2019-11, Vol.16 (1), p.209-14, Article 209

2019

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Emerging role of Toll-like receptors in the control of pain and itch

Neuroscience bulletin, 2012-04, Vol.28 (2), p.131-144

2012

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MicroRNA-146a-5p attenuates neuropathic pain via suppressing TRAF6 signaling in the spinal cord

Brain, behavior, and immunity, 2015-10, Vol.49, p.119-129

2015

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Chemokine receptor CCR2 contributes to neuropathic pain and the associated depression via increasing NR2B-mediated currents in both D1 and D2 dopamine receptor-containing medium spiny neurons in the nucleus accumbens shell

Neuropsychopharmacology (New York, N.Y.), 2018-10, Vol.43 (11), p.2320-2330

2018

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Spinal injection of TNF-α-activated astrocytes produces persistent pain symptom mechanical allodynia by releasing monocyte chemoattractant protein-1

Glia, 2010-11, Vol.58 (15), p.1871-1880

2010

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JNK-Induced MCP-1 Production in Spinal Cord Astrocytes Contributes to Central Sensitization and Neuropathic Pain

The Journal of neuroscience, 2009-04, Vol.29 (13), p.4096-4108

2009

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Chemokine CXCL1 enhances inflammatory pain and increases NMDA receptor activity and COX-2 expression in spinal cord neurons via activation of CXCR2

Experimental neurology, 2014-11, Vol.261, p.328-336

2014

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TRAF6 Contributes to CFA-Induced Spinal Microglial Activation and Chronic Inflammatory Pain in Mice

Cellular and molecular neurobiology, 2022-07, Vol.42 (5), p.1543-1555

2022

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Distinct roles of matrix metalloproteases in the early- and late-phase development of neuropathic pain

Nature medicine, 2008-03, Vol.14 (3), p.331-336

2008

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Descending dopaminergic pathway facilitates itch signal processing via activating spinal GRPR + neurons

EMBO reports, 2023-10, Vol.24 (10), p.e56098

2023

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Activation of JNK pathway in persistent pain

Neuroscience letters, 2008-06, Vol.437 (3), p.180-183

2008

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Chemokines in chronic pain: cellular and molecular mechanisms and therapeutic potential

Pharmacology & therapeutics (Oxford), 2020-08, Vol.212, p.107581, Article 107581

2020

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TLR3 deficiency impairs spinal cord synaptic transmission, central sensitization, and pruritus in mice

The Journal of clinical investigation, 2012-06, Vol.122 (6), p.2195-2207

2012

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Light touch induces ERK activation in superficial dorsal horn neurons after inflammation: involvement of spinal astrocytes and JNK signaling in touch-evoked central sensitization and mechanical allodynia

Journal of neurochemistry, 2010-10, Vol.115 (2), p.505-514

2010

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Interferon alpha inhibits spinal cord synaptic and nociceptive transmission via neuronal-glial interactions

Scientific reports, 2016-09, Vol.6 (1), p.34356-34356, Article 34356

2016

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β-arrestin-2 regulates NMDA receptor function in spinal lamina II neurons and duration of persistent pain

Nature communications, 2016-08, Vol.7 (1), p.12531-12531, Article 12531

2016

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Nerve Injury-Induced γH2AX Reduction in Primary Sensory Neurons Is Involved in Neuropathic Pain Processing

International journal of molecular sciences, 2023-06, Vol.24 (12), p.10148

2023

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Exogenous induction of HO-1 alleviates vincristine-induced neuropathic pain by reducing spinal glial activation in mice

Neurobiology of disease, 2015-07, Vol.79, p.100-110

2015

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Editorial: Pain, immunity, and neurological and autoimmune disorders

Frontiers in immunology, 2023-04, Vol.14, p.1195204-1195204

2023

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Chemokines in neuron–glial cell interaction and pathogenesis of neuropathic pain

Targeting Astrocyte Signaling for Chronic Pain

Chemokine contribution to neuropathic pain: Respective induction of CXCL1 and CXCR2 in spinal cord astrocytes and neurons

Toll-like receptor 4 contributes to chronic itch, alloknesis, and spinal astrocyte activation in male mice

Chemokine CXCL13 mediates orofacial neuropathic pain via CXCR5/ERK pathway in the trigeminal ganglion of mice

A long noncoding RNA contributes to neuropathic pain by silencing Kcna2 in primary afferent neurons

Paclitaxel-activated astrocytes produce mechanical allodynia in mice by releasing tumor necrosis factor-α and stromal-derived cell factor 1

Emerging role of Toll-like receptors in the control of pain and itch

MicroRNA-146a-5p attenuates neuropathic pain via suppressing TRAF6 signaling in the spinal cord

Chemokine receptor CCR2 contributes to neuropathic pain and the associated depression via increasing NR2B-mediated currents in both D1 and D2 dopamine receptor-containing medium spiny neurons in the nucleus accumbens shell

Spinal injection of TNF-α-activated astrocytes produces persistent pain symptom mechanical allodynia by releasing monocyte chemoattractant protein-1

JNK-Induced MCP-1 Production in Spinal Cord Astrocytes Contributes to Central Sensitization and Neuropathic Pain

Chemokine CXCL1 enhances inflammatory pain and increases NMDA receptor activity and COX-2 expression in spinal cord neurons via activation of CXCR2

TRAF6 Contributes to CFA-Induced Spinal Microglial Activation and Chronic Inflammatory Pain in Mice

Distinct roles of matrix metalloproteases in the early- and late-phase development of neuropathic pain

Descending dopaminergic pathway facilitates itch signal processing via activating spinal GRPR + neurons

Activation of JNK pathway in persistent pain

Chemokines in chronic pain: cellular and molecular mechanisms and therapeutic potential

TLR3 deficiency impairs spinal cord synaptic transmission, central sensitization, and pruritus in mice

Light touch induces ERK activation in superficial dorsal horn neurons after inflammation: involvement of spinal astrocytes and JNK signaling in touch-evoked central sensitization and mechanical allodynia

Interferon alpha inhibits spinal cord synaptic and nociceptive transmission via neuronal-glial interactions

β-arrestin-2 regulates NMDA receptor function in spinal lamina II neurons and duration of persistent pain

Nerve Injury-Induced γH2AX Reduction in Primary Sensory Neurons Is Involved in Neuropathic Pain Processing

Exogenous induction of HO-1 alleviates vincristine-induced neuropathic pain by reducing spinal glial activation in mice

Editorial: Pain, immunity, and neurological and autoimmune disorders

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