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N‐glycan processing selects ERAD‐resistant misfolded proteins for ER‐to‐lysosome‐associated degradation

The EMBO journal, 2021-08, Vol.40 (15), p.e107240-n/a

2021

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Translocon component Sec62 acts in endoplasmic reticulum turnover during stress recovery

Nature cell biology, 2016-11, Vol.18 (11), p.1173-1184

2016

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Stringent requirement for HRD1, SEL1L, and OS-9/XTP3-B for disposal of ERAD-LS substrates

The Journal of cell biology, 2010-01, Vol.188 (2), p.223-235

2010

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TMX4-driven LINC complex disassembly and asymmetric autophagy of the nuclear envelope upon acute ER stress

Nature communications, 2023-06, Vol.14 (1), p.3497-20, Article 3497

2023

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Segregation and rapid turnover of EDEM1 by an autophagy-like mechanism modulates standard ERAD and folding activities

Biochemical and biophysical research communications, 2008-07, Vol.371 (3), p.405-410

2008

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Malectin participates in a backup glycoprotein quality control pathway in the mammalian ER

PloS one, 2011-01, Vol.6 (1), p.e16304-e16304

2011

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Role of EDEM in the Release of Misfolded Glycoproteins from the Calnexin Cycle

Science (American Association for the Advancement of Science), 2003-02, Vol.299 (5611), p.1397-1400

2003

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Decoding Seafood: Multi-Marker Metabarcoding for Authenticating Processed Seafood

Foods, 2024-07, Vol.13 (15), p.2382

2024

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Cyclosporine A-sensitive, cyclophilin B-dependent endoplasmic reticulum-associated degradation

PloS one, 2010-09, Vol.5 (9), p.e13008

2010

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A Novel Stress-induced EDEM Variant Regulating Endoplasmic Reticulum-associated Glycoprotein Degradation

The Journal of biological chemistry, 2005-01, Vol.280 (4), p.2424-2428

2005

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RETRACTED: Autoadaptive ER-Associated Degradation Defines a Preemptive Unfolded Protein Response Pathway

Molecular cell, 2013-12, Vol.52 (6), p.783-793

2013

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Endoplasmic Reticulum and Lysosomal Quality Control of Four Nonsense Mutants of Iduronate 2-Sulfatase Linked to Hunter's Syndrome

DNA and cell biology, 2020-02, Vol.39 (2), p.226-234

2020

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Contrasting Functions of Calreticulin and Calnexin in Glycoprotein Folding and ER Quality Control

Molecular cell, 2004-01, Vol.13 (1), p.125-135

2004

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Sequential Assistance of Molecular Chaperones and Transient Formation of Covalent Complexes during Protein Degradation from the ER

The Journal of cell biology, 2002-07, Vol.158 (2), p.247-257

2002

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Persistent Glycoprotein Misfolding Activates the Glucosidase II/UGT1-Driven Calnexin Cycle to Delay Aggregation and Loss of Folding Competence

Molecular cell, 2005-11, Vol.20 (4), p.503-512

2005

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Erratum: Corrigendum: Translocon component Sec62 acts in endoplasmic reticulum turnover during stress recovery

Nature cell biology, 2017-01, Vol.19 (1), p.76-76

2017

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The Use of Calnexin and Calreticulin by Cellular and Viral Glycoproteins

The Journal of biological chemistry, 2005-08, Vol.280 (31), p.28265-28271

2005

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Chronic delivery of antibody fragments using immunoisolated cell implants as a passive vaccination tool

PloS one, 2011-04, Vol.6 (4), p.e18268

2011

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RETRACTED: Role of the SEL1L:LC3-I Complex as an ERAD Tuning Receptor in the Mammalian ER

Molecular cell, 2012-06, Vol.46 (6), p.809-819

2012

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Selective inhibition of Ii-dependent antigen presentation by Helicobacter pylori toxin VacA

The Journal of experimental medicine, 1998-01, Vol.187 (1), p.135-140

1998

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ER‐to‐lysosome‐associated degradation of proteasome‐resistant ATZ polymers occurs via receptor‐mediated vesicular transport

The EMBO journal, 2018-09, Vol.37 (17), p.n/a

2018

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ER-to-lysosome-associated degradation acts as failsafe mechanism upon ERAD dysfunction

EMBO reports, 2024-06, Vol.25 (6), p.2773-2785

2024

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Vacuoles Induced by Helicobacter pylori Toxin Contain Both Late Endosomal and Lysosomal Markers

The Journal of biological chemistry, 1997-10, Vol.272 (40), p.25339-25344

1997

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EDEM Contributes to Maintenance of Protein Folding Efficiency and Secretory Capacity

The Journal of biological chemistry, 2004-10, Vol.279 (43), p.44600-44605

2004

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TMX5/TXNDC15, a natural trapping mutant of the PDI family is a client of the proteostatic factor ERp44

Life science alliance, 2024-12, Vol.7 (12), p.e202403047

2024

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N‐glycan processing selects ERAD‐resistant misfolded proteins for ER‐to‐lysosome‐associated degradation

Translocon component Sec62 acts in endoplasmic reticulum turnover during stress recovery

Stringent requirement for HRD1, SEL1L, and OS-9/XTP3-B for disposal of ERAD-LS substrates

TMX4-driven LINC complex disassembly and asymmetric autophagy of the nuclear envelope upon acute ER stress

Segregation and rapid turnover of EDEM1 by an autophagy-like mechanism modulates standard ERAD and folding activities

Malectin participates in a backup glycoprotein quality control pathway in the mammalian ER

Role of EDEM in the Release of Misfolded Glycoproteins from the Calnexin Cycle

Decoding Seafood: Multi-Marker Metabarcoding for Authenticating Processed Seafood

Cyclosporine A-sensitive, cyclophilin B-dependent endoplasmic reticulum-associated degradation

A Novel Stress-induced EDEM Variant Regulating Endoplasmic Reticulum-associated Glycoprotein Degradation

RETRACTED: Autoadaptive ER-Associated Degradation Defines a Preemptive Unfolded Protein Response Pathway

Endoplasmic Reticulum and Lysosomal Quality Control of Four Nonsense Mutants of Iduronate 2-Sulfatase Linked to Hunter's Syndrome

Contrasting Functions of Calreticulin and Calnexin in Glycoprotein Folding and ER Quality Control

Sequential Assistance of Molecular Chaperones and Transient Formation of Covalent Complexes during Protein Degradation from the ER

Persistent Glycoprotein Misfolding Activates the Glucosidase II/UGT1-Driven Calnexin Cycle to Delay Aggregation and Loss of Folding Competence

Erratum: Corrigendum: Translocon component Sec62 acts in endoplasmic reticulum turnover during stress recovery

The Use of Calnexin and Calreticulin by Cellular and Viral Glycoproteins

Chronic delivery of antibody fragments using immunoisolated cell implants as a passive vaccination tool

RETRACTED: Role of the SEL1L:LC3-I Complex as an ERAD Tuning Receptor in the Mammalian ER

Selective inhibition of Ii-dependent antigen presentation by Helicobacter pylori toxin VacA

ER‐to‐lysosome‐associated degradation of proteasome‐resistant ATZ polymers occurs via receptor‐mediated vesicular transport

ER-to-lysosome-associated degradation acts as failsafe mechanism upon ERAD dysfunction

Vacuoles Induced by Helicobacter pylori Toxin Contain Both Late Endosomal and Lysosomal Markers

EDEM Contributes to Maintenance of Protein Folding Efficiency and Secretory Capacity

TMX5/TXNDC15, a natural trapping mutant of the PDI family is a client of the proteostatic factor ERp44

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