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Barrett's oesophagus and oesophageal adenocarcinoma: time for a new synthesis

Nature reviews. Cancer, 2010-02, Vol.10 (2), p.87-101

2010

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NSAIDs modulate CDKN2A, TP53, and DNA content risk for progression to esophageal adenocarcinoma

PLoS medicine, 2007-02, Vol.4 (2), p.e67-e67

2007

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Extrachromosomal DNA in the cancerous transformation of Barrett’s oesophagus

Nature (London), 2023-04, Vol.616 (7958), p.798-805

2023

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NSAIDs modulate clonal evolution in Barrett's esophagus

PLoS genetics, 2013-06, Vol.9 (6), p.e1003553

2013

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Somatic whole genome dynamics of precancer in Barrett’s esophagus reveals features associated with disease progression

Nature communications, 2022-04, Vol.13 (1), p.2300-2300, Article 2300

2022

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Temporal and Spatial Evolution of Somatic Chromosomal Alterations: A Case-Cohort Study of Barrett's Esophagus

Cancer prevention research (Philadelphia, Pa.), 2014-01, Vol.7 (1), p.114-127

2014

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Genetic clonal diversity predicts progression to esophageal adenocarcinoma

Nature genetics, 2006-04, Vol.38 (4), p.468-473

2006

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Within‐patient phylogenetic reconstruction reveals early events in Barrett’s Esophagus

Evolutionary applications, 2021-02, Vol.14 (2), p.399-415

2021

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The combination of genetic instability and clonal expansion predicts progression to esophageal adenocarcinoma

Cancer research (Chicago, Ill.), 2004-10, Vol.64 (20), p.7629-7633

2004

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p16 mutation spectrum in the premalignant condition Barrett's esophagus

PloS one, 2008-11, Vol.3 (11), p.e3809-e3809

2008

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Assessment of Esophageal Adenocarcinoma Risk Using Somatic Chromosome Alterations in Longitudinal Samples in Barrett's Esophagus

Cancer prevention research (Philadelphia, Pa.), 2015-09, Vol.8 (9), p.845-856

2015

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NSAID use and somatic exomic mutations in Barrett's esophagus

Genome medicine, 2018-02, Vol.10 (1), p.17-17, Article 17

2018

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Selectively advantageous mutations and hitchhikers in neoplasms: P16 lesions are selected in Barrett's esophagus

Cancer research (Chicago, Ill.), 2004-05, Vol.64 (10), p.3414-3427

2004

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Predictors of progression in Barrett's esophagus II: baseline 17p (p53) loss of heterozygosity identifies a patient subset at increased risk for neoplastic progression

The American journal of gastroenterology, 2001-10, Vol.96 (10), p.2839-2848

2001

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Cell Proliferation, Cell Cycle Abnormalities, and Cancer Outcome in Patients with Barrett's Esophagus: A Long-term Prospective Study

Clinical cancer research, 2008-11, Vol.14 (21), p.6988-6995

2008

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Correction to: NSAID use and somatic exomic mutations in Barrett's esophagus

Genome medicine, 2019-03, Vol.11 (1), p.14-14, Article 14

2019

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Crypt dysplasia with surface maturation: a clinical, pathologic, and molecular study of a Barrett's esophagus cohort

The American journal of surgical pathology, 2006-04, Vol.30 (4), p.423

2006

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Predictors of progression in Barrett’s esophagus II: baseline 17p (p53) loss of heterozygosity identifies a patient subset at increased risk for neoplastic progression

The American journal of gastroenterology, 2001, Vol.96 (10), p.2839-2848

2001

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Wisdom Of The Crowd For Early Detection In Barrett's Esophagus

2021 IEEE 18th International Symposium on Biomedical Imaging (ISBI), 2021, p.531-535

2021

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Clonal expansion and loss of heterozygosity at chromosomes 9p and 17p in premalignant esophageal (Barrett's) tissue

JNCI : Journal of the National Cancer Institute, 1999-12, Vol.91 (24), p.2087-2095

1999

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Evolution of neoplastic cell lineages in Barrett oesophagus

Nature genetics, 1999-05, Vol.22 (1), p.106-109

1999

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Assessment of TROP2, CEACAM5 and DLL3 in metastatic prostate cancer: Expression landscape and molecular correlates

NPJ precision oncology, 2024-05, Vol.8 (1), p.104-9, Article 104

2024

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p53-Mutant clones and field effects in Barrett's esophagus

Cancer research (Chicago, Ill.), 1999-10, Vol.59 (19), p.4784-4787

1999

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Single nucleotide polymorphism-based genome-wide chromosome copy change, loss of heterozygosity, and aneuploidy in Barrett's esophagus neoplastic progression

Cancer prevention research (Philadelphia, Pa.), 2008-11, Vol.1 (6), p.413-423

2008

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Direct inference of SNP heterozygosity rates and resolution of LOH detection

PLoS computational biology, 2007-11, Vol.3 (11), p.e244-e244

2007

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Barrett's oesophagus and oesophageal adenocarcinoma: time for a new synthesis

NSAIDs modulate CDKN2A, TP53, and DNA content risk for progression to esophageal adenocarcinoma

Extrachromosomal DNA in the cancerous transformation of Barrett’s oesophagus

NSAIDs modulate clonal evolution in Barrett's esophagus

Somatic whole genome dynamics of precancer in Barrett’s esophagus reveals features associated with disease progression

Temporal and Spatial Evolution of Somatic Chromosomal Alterations: A Case-Cohort Study of Barrett's Esophagus

Genetic clonal diversity predicts progression to esophageal adenocarcinoma

Within‐patient phylogenetic reconstruction reveals early events in Barrett’s Esophagus

The combination of genetic instability and clonal expansion predicts progression to esophageal adenocarcinoma

p16 mutation spectrum in the premalignant condition Barrett's esophagus

Assessment of Esophageal Adenocarcinoma Risk Using Somatic Chromosome Alterations in Longitudinal Samples in Barrett's Esophagus

NSAID use and somatic exomic mutations in Barrett's esophagus

Selectively advantageous mutations and hitchhikers in neoplasms: P16 lesions are selected in Barrett's esophagus

Predictors of progression in Barrett's esophagus II: baseline 17p (p53) loss of heterozygosity identifies a patient subset at increased risk for neoplastic progression

Cell Proliferation, Cell Cycle Abnormalities, and Cancer Outcome in Patients with Barrett's Esophagus: A Long-term Prospective Study

Correction to: NSAID use and somatic exomic mutations in Barrett's esophagus

Crypt dysplasia with surface maturation: a clinical, pathologic, and molecular study of a Barrett's esophagus cohort

Predictors of progression in Barrett’s esophagus II: baseline 17p (p53) loss of heterozygosity identifies a patient subset at increased risk for neoplastic progression

Wisdom Of The Crowd For Early Detection In Barrett's Esophagus

Clonal expansion and loss of heterozygosity at chromosomes 9p and 17p in premalignant esophageal (Barrett's) tissue

Evolution of neoplastic cell lineages in Barrett oesophagus

Assessment of TROP2, CEACAM5 and DLL3 in metastatic prostate cancer: Expression landscape and molecular correlates

p53-Mutant clones and field effects in Barrett's esophagus

Single nucleotide polymorphism-based genome-wide chromosome copy change, loss of heterozygosity, and aneuploidy in Barrett's esophagus neoplastic progression

Direct inference of SNP heterozygosity rates and resolution of LOH detection

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