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Increased somatic mutation burdens in normal human cells due to defective DNA polymerases

Nature genetics, 2021-10, Vol.53 (10), p.1434-1442

2021

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Targeting autophagy potentiates tyrosine kinase inhibitor-induced cell death in Philadelphia chromosome-positive cells, including primary CML stem cells

The Journal of clinical investigation, 2009-05, Vol.119 (5), p.1109-1123

2009

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Inhibition of oxidative metabolism leads to p53 genetic inactivation and transformation in neural stem cells

Proceedings of the National Academy of Sciences - PNAS, 2015-01, Vol.112 (4), p.1059-1064

2015

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A PML/Slit Axis Controls Physiological Cell Migration and Cancer Invasion in the CNS

Cell reports (Cambridge), 2017-07, Vol.20 (2), p.411-426

2017

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Metabolites in visceral fat: useful signals of metabolic syndrome?

Biochemical journal, 2018-05, Vol.475 (10), p.1789-1791

2018

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Long-Fiber Carbon Nanotubes Replicate Asbestos-Induced Mesothelioma with Disruption of the Tumor Suppressor Gene Cdkn2a (Ink4a/Arf)

Current biology, 2017-11, Vol.27 (21), p.3302-3314.e6

2017

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A novel genetic marker of fluoropyrimidine-induced cardiovascular toxicity

Journal of clinical oncology, 2023-06, Vol.41 (16_suppl), p.3028-3028

2023

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Increased somatic mutation burdens in normal human cells due to defective DNA polymerases

Nature genetics, 2021-10, Vol.53 (10), p.1434-4

2021

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96 Longitudinal Profiling of Carbon Nanotube-Induced Sporadic Mesothelioma Development: Defining the Adverse Outcome Pathway for Disease Progression

Annals of work exposures and health, 2023-05, Vol.67 (Supplement_1), p.i61-i61

2023

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156 Nitrogen-Doped Multi-Walled Carbon Nanotubes show Attenuated Pathogenicity in a Mouse Model of Pleural Exposure

Annals of work exposures and health, 2023-05, Vol.67 (Supplement_1), p.i76-i77

2023

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An epigenetically controlled PML/Slit axis at the root of cell migration in both normal and neoplastic cells in the CNS

Neuro-oncology (Charlottesville, Va.), 2018-01, Vol.20 (suppl_1), p.i10-i10

2018

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Predicting sensitivity to PI3K/mTOR inhibition in asbestos-induced malignant mesothelioma models

Toxicology letters, 2014-09, Vol.229, p.S153-S153

2014

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Role of pro-inflammatory cells in mesothelial cell oncogenesis induced by toxic fibres

Toxicology letters, 2014-09, Vol.229, p.S59-S59

2014

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P53THE METABOLIC AUTOPHAGY PATHWAY REGULATES THE RESPONSE TO TEMOZOLOMIDE IN GLIOBLASTOMA MULTIFORME-INITIATING STEM CELLS

Neuro-oncology (Charlottesville, Va.), 2014-10, Vol.16 (Suppl 6), p.vi9-vi9

2014

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Exposure to pathogenic fibres causes pro-oncogenic changes in the pleural cavity

Toxicology letters, 2014-09, Vol.229, p.S62-S62

2014

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P53 THE METABOLIC AUTOPHAGY PATHWAY REGULATES THE RESPONSE TO TEMOZOLOMIDE IN GLIOBLASTOMA MULTIFORME-INITIATING STEM CELLS

Neuro-oncology (Charlottesville, Va.), 2014-10, Vol.16 (suppl 6), p.vi9-vi9

2014

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Targeting autophagy potentiates tyrosine kinase inhibitor–induced cell death in Philadelphia chromosome–positive cells, including primary CML stem cells

The Journal of clinical investigation, 2013-08, Vol.123 (8), p.3634-3634

2013

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Inhibition of Autophagy in Combination with Ponatinib or Dual PI3K/mTOR Inhibition to Improve Treatment Response for Both Bcr-Abl Dependent and Independent Mechanisms of TKI-Resistance in CML

Blood, 2012-11, Vol.120 (21), p.1664-1664

2012

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OP01. A PML/POLYCOMB/SLIT AXIS REGULATES MIGRATION OF BOTH NORMAL AND NEOPLASTIC CELLS IN THE CNS

Neuro-oncology (Charlottesville, Va.), 2017-01, Vol.19 (suppl_1), p.i24-i24

2017

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PP07. A PML/POLYCOMB/SLIT AXIS REGULATES MIGRATION OF BOTH NORMAL AND NEOPLASTIC CELLS IN THE CNS

Neuro-oncology (Charlottesville, Va.), 2017-01, Vol.19 (suppl_1), p.i2-i2

2017

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BNOS 2016 Abstracts: Oral Presentations

Neuro-oncology (Charlottesville, Va.), 2017-01, Vol.19 (Suppl 1), p.i24-i24

2017

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A review of CARE’s Community Score Card experience and evidence

Health policy and planning, 2016-12, Vol.31 (10), p.1467-1478

2016

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Increased somatic mutation burdens in normal human cells due to defective DNA polymerases

Targeting autophagy potentiates tyrosine kinase inhibitor-induced cell death in Philadelphia chromosome-positive cells, including primary CML stem cells

Inhibition of oxidative metabolism leads to p53 genetic inactivation and transformation in neural stem cells

A PML/Slit Axis Controls Physiological Cell Migration and Cancer Invasion in the CNS

The p53 Codon 72 Pro/Pro Genotype Identifies Poor-Prognosis Neuroblastoma Patients: Correlation with Reduced Apoptosis and Enhanced Senescence by the p53-72P Isoform

Metabolites in visceral fat: useful signals of metabolic syndrome?

Long-Fiber Carbon Nanotubes Replicate Asbestos-Induced Mesothelioma with Disruption of the Tumor Suppressor Gene Cdkn2a (Ink4a/Arf)

A novel genetic marker of fluoropyrimidine-induced cardiovascular toxicity

Increased somatic mutation burdens in normal human cells due to defective DNA polymerases

96 Longitudinal Profiling of Carbon Nanotube-Induced Sporadic Mesothelioma Development: Defining the Adverse Outcome Pathway for Disease Progression

156 Nitrogen-Doped Multi-Walled Carbon Nanotubes show Attenuated Pathogenicity in a Mouse Model of Pleural Exposure

An epigenetically controlled PML/Slit axis at the root of cell migration in both normal and neoplastic cells in the CNS

Predicting sensitivity to PI3K/mTOR inhibition in asbestos-induced malignant mesothelioma models

Role of pro-inflammatory cells in mesothelial cell oncogenesis induced by toxic fibres

P53THE METABOLIC AUTOPHAGY PATHWAY REGULATES THE RESPONSE TO TEMOZOLOMIDE IN GLIOBLASTOMA MULTIFORME-INITIATING STEM CELLS

Exposure to pathogenic fibres causes pro-oncogenic changes in the pleural cavity

P53 THE METABOLIC AUTOPHAGY PATHWAY REGULATES THE RESPONSE TO TEMOZOLOMIDE IN GLIOBLASTOMA MULTIFORME-INITIATING STEM CELLS

Targeting autophagy potentiates tyrosine kinase inhibitor–induced cell death in Philadelphia chromosome–positive cells, including primary CML stem cells

Inhibition of Autophagy in Combination with Ponatinib or Dual PI3K/mTOR Inhibition to Improve Treatment Response for Both Bcr-Abl Dependent and Independent Mechanisms of TKI-Resistance in CML

The p53 Codon 72 Pro/Pro Genotype Identifies Poor-Prognosis Neuroblastoma Patients: Correlation with Reduced Apoptosis and Enhanced Senescence by the p53-72P Isoform12

OP01. A PML/POLYCOMB/SLIT AXIS REGULATES MIGRATION OF BOTH NORMAL AND NEOPLASTIC CELLS IN THE CNS

PP07. A PML/POLYCOMB/SLIT AXIS REGULATES MIGRATION OF BOTH NORMAL AND NEOPLASTIC CELLS IN THE CNS

BNOS 2016 Abstracts: Oral Presentations

Targeting Autophagy Potentiates Imatinib-Induced Cell Death in Philadelphia Positive Cells Including Primary CML Stem Cells

A review of CARE’s Community Score Card experience and evidence

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