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Tau phosphorylation in neuronal cell function and dysfunction

Journal of cell science, 2004-11, Vol.117 (Pt 24), p.5721-5729

2004

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The Crosstalk Between Pathological Tau Phosphorylation and Mitochondrial Dysfunction as a Key to Understanding and Treating Alzheimer’s Disease

Molecular neurobiology, 2020-12, Vol.57 (12), p.5103-5120

2020

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Impaired mitochondrial dynamics and Nrf2 signaling contribute to compromised responses to oxidative stress in striatal cells expressing full-length mutant huntingtin

PloS one, 2013-03, Vol.8 (3), p.e57932-e57932

2013

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Tauopathy-associated tau modifications selectively impact neurodegeneration and mitophagy in a novel C. elegans single-copy transgenic model

Molecular neurodegeneration, 2020-11, Vol.15 (1), p.65-65, Article 65

2020

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Histone deacetylase 6 interacts with the microtubule-associated protein tau

Journal of neurochemistry, 2008-09, Vol.106 (5), p.2119-2130

2008

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Deletion of Transglutaminase 2 from Mouse Astrocytes Significantly Improves Their Ability to Promote Neurite Outgrowth on an Inhibitory Matrix

International journal of molecular sciences, 2023-03, Vol.24 (7), p.6058

2023

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All-optical spatiotemporal mapping of ROS dynamics across mitochondrial microdomains in situ

Nature communications, 2023-09, Vol.14 (1), p.6036-16, Article 6036

2023

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Author Correction: All-optical spatiotemporal mapping of ROS dynamics across mitochondrial microdomains in situ

Nature communications, 2023-12, Vol.14 (1), p.8230-8230, Article 8230

2023

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Transglutaminase and Polyamination of Tubulin: Posttranslational Modification for Stabilizing Axonal Microtubules

Neuron (Cambridge, Mass.), 2013-04, Vol.78 (1), p.109-123

2013

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Intracellular localization and conformational state of transglutaminase 2: implications for cell death

PloS one, 2009-07, Vol.4 (7), p.e6123-e6123

2009

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Nrf2 reduces levels of phosphorylated tau protein by inducing autophagy adaptor protein NDP52

Nature communications, 2014-03, Vol.5 (1), p.3496-3496, Article 3496

2014

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Transglutaminase regulation of cell function

Physiological reviews, 2014-04, Vol.94 (2), p.383-417

2014

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interrelationship between mitochondrial dysfunction and transcriptional dysregulation in Huntington disease

Journal of bioenergetics and biomembranes, 2010-06, Vol.42 (3), p.199-205

2010

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Tau Is Hyperphosphorylated at Multiple Sites in Mouse Brain In Vivo After Streptozotocin-Induced Insulin Deficiency

Diabetes (New York, N.Y.), 2006-12, Vol.55 (12), p.3320-3325

2006

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Cytosolic guanine nucledotide binding deficient form of transglutaminase 2 (R580a) potentiates cell death in oxygen glucose deprivation

PloS one, 2011-01, Vol.6 (1), p.e16665-e16665

2011

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Autophagy in Alzheimer's disease

Reviews in the neurosciences, 2015-01, Vol.26 (4), p.385

2015

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Cyclin‐dependent kinase‐5 in neurodegeneration

Journal of neurochemistry, 2004-03, Vol.88 (6), p.1313-1326

2004

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Site-specific Phosphorylation and Caspase Cleavage Differentially Impact Tau-Microtubule Interactions and Tau Aggregation

The Journal of biological chemistry, 2006-07, Vol.281 (28), p.19107-19114

2006

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BAG3 facilitates the clearance of endogenous tau in primary neurons

Neurobiology of aging, 2015-01, Vol.36 (1), p.241-248

2015

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Differential modulation of TCF/LEF-1 activity by the soluble LRP6-ICD

PloS one, 2010-07, Vol.5 (7), p.e11821-e11821

2010

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A Caspase Cleaved Form of Tau Is Preferentially Degraded through the Autophagy Pathway

The Journal of biological chemistry, 2010-07, Vol.285 (29), p.21978-21987

2010

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Presenilin 1 Regulates Membrane Homeostatic Pathways that are Dysregulated in Alzheimer’s Disease

Journal of Alzheimer's disease, 2020-01, Vol.77 (3), p.961-977

2020

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Truncated tau and Aβ cooperatively impair mitochondria in primary neurons

Neurobiology of aging, 2012-03, Vol.33 (3), p.619.e25-619.e35

2012

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Tau Post-Translational Modifications: Potentiators of Selective Vulnerability in Sporadic Alzheimer's Disease

Biology (Basel, Switzerland), 2021-10, Vol.10 (10), p.1047

2021

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Neuronal BAG3 attenuates tau hyperphosphorylation, synaptic dysfunction, and cognitive deficits induced by traumatic brain injury via the regulation of autophagy-lysosome pathway

Acta neuropathologica, 2024-10, Vol.148 (1), p.52, Article 52

2024

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Tau phosphorylation in neuronal cell function and dysfunction

The Crosstalk Between Pathological Tau Phosphorylation and Mitochondrial Dysfunction as a Key to Understanding and Treating Alzheimer’s Disease

Impaired mitochondrial dynamics and Nrf2 signaling contribute to compromised responses to oxidative stress in striatal cells expressing full-length mutant huntingtin

Tauopathy-associated tau modifications selectively impact neurodegeneration and mitophagy in a novel C. elegans single-copy transgenic model

Histone deacetylase 6 interacts with the microtubule-associated protein tau

Deletion of Transglutaminase 2 from Mouse Astrocytes Significantly Improves Their Ability to Promote Neurite Outgrowth on an Inhibitory Matrix

All-optical spatiotemporal mapping of ROS dynamics across mitochondrial microdomains in situ

Author Correction: All-optical spatiotemporal mapping of ROS dynamics across mitochondrial microdomains in situ

Transglutaminase and Polyamination of Tubulin: Posttranslational Modification for Stabilizing Axonal Microtubules

Intracellular localization and conformational state of transglutaminase 2: implications for cell death

Nrf2 reduces levels of phosphorylated tau protein by inducing autophagy adaptor protein NDP52

Transglutaminase regulation of cell function

interrelationship between mitochondrial dysfunction and transcriptional dysregulation in Huntington disease

Tau Is Hyperphosphorylated at Multiple Sites in Mouse Brain In Vivo After Streptozotocin-Induced Insulin Deficiency

Cytosolic guanine nucledotide binding deficient form of transglutaminase 2 (R580a) potentiates cell death in oxygen glucose deprivation

Autophagy in Alzheimer's disease

Cyclin‐dependent kinase‐5 in neurodegeneration

Site-specific Phosphorylation and Caspase Cleavage Differentially Impact Tau-Microtubule Interactions and Tau Aggregation

BAG3 facilitates the clearance of endogenous tau in primary neurons

Differential modulation of TCF/LEF-1 activity by the soluble LRP6-ICD

A Caspase Cleaved Form of Tau Is Preferentially Degraded through the Autophagy Pathway

Presenilin 1 Regulates Membrane Homeostatic Pathways that are Dysregulated in Alzheimer’s Disease

Truncated tau and Aβ cooperatively impair mitochondria in primary neurons

Tau Post-Translational Modifications: Potentiators of Selective Vulnerability in Sporadic Alzheimer's Disease

Neuronal BAG3 attenuates tau hyperphosphorylation, synaptic dysfunction, and cognitive deficits induced by traumatic brain injury via the regulation of autophagy-lysosome pathway

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