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Idiopathic Parkinson's disease: possible routes by which vulnerable neuronal types may be subject to neuroinvasion by an unknown pathogen

Journal of Neural Transmission, 2003-05, Vol.110 (5), p.517-536

2003

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Proteasomal Inhibition by α-Synuclein Filaments and Oligomers

The Journal of biological chemistry, 2004-03, Vol.279 (13), p.12924-12934

2004

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Calcium(II) selectively induces α‐synuclein annular oligomers via interaction with the C‐terminal domain

Protein science, 2004-12, Vol.13 (12), p.3245-3252

2004

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Parkin Localizes to the Lewy Bodies of Parkinson Disease and Dementia with Lewy Bodies

The American journal of pathology, 2002-05, Vol.160 (5), p.1655-1667

2002

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Extensive axonal Lewy neurites in Parkinson's disease: a novel pathological feature revealed by α-synuclein immunocytochemistry

Neuroscience letters, 1999-04, Vol.265 (1), p.67-69

1999

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In Situ and in Vitro Study of Colocalization and Segregation of α-Synuclein, Ubiquitin, and Lipids in Lewy Bodies

Experimental neurology, 2000-12, Vol.166 (2), p.324-333

2000

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SUMO-1 marks subdomains within glial cytoplasmic inclusions of multiple system atrophy

Neuroscience letters, 2005-06, Vol.381 (1), p.74-79

2005

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SUMO-1 marks the nuclear inclusions in familial neuronal intranuclear inclusion disease

Experimental neurology, 2003-11, Vol.184 (1), p.436-446

2003

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Septin 3 (G‐septin) is a developmentally regulated phosphoprotein enriched in presynaptic nerve terminals

Journal of neurochemistry, 2004-11, Vol.91 (3), p.579-590

2004

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The solubility of α‐synuclein in multiple system atrophy differs from that of dementia with Lewy bodies and Parkinson's disease

Journal of neurochemistry, 2001-01, Vol.76 (1), p.87-96

2001

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Multiple-system atrophy: a new α-synuclein disease?

The Lancet (British edition), 1998-08, Vol.352 (9127), p.547-548

1998

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Annular α‐synuclein species from purified multiple system atrophy inclusions

Journal of neurochemistry, 2004-07, Vol.90 (2), p.502-512

2004

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α‐Synuclein Immunoisolation of Glial Inclusions from Multiple System Atrophy Brain Tissue Reveals Multiprotein Components

Journal of neurochemistry, 1999-11, Vol.73 (5), p.2093-2100

1999

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Microtubule-associated Protein 1B Is a Component of Cortical Lewy Bodies and Binds α-Synuclein Filaments

The Journal of biological chemistry, 2000-07, Vol.275 (28), p.21500-21507

2000

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Dominantly-inherited adult-onset leukodystrophy with palatal tremor caused by a mutation in the glial fibrillary acidic protein gene

Movement disorders, 2004-10, Vol.19 (10), p.1244-1248

2004

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Parkinson's disease : affection of brain stem nuclei controlling premotor and motor neurons of the somatomotor system

Acta neuropathologica, 2000-05, Vol.99 (5), p.489-495

2000

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Annular alpha-synuclein oligomers are potentially toxic agents in alpha-synucleinopathy. Hypothesis

Neurotoxicity research, 2005, Vol.7 (1-2), p.59-67

2005

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α-Synuclein filaments bind the transcriptional regulator HMGB-1

Neuroreport, 2004-12, Vol.15 (18), p.2735-2739

2004

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α-synuclein fibrils constitute the central core of oligodendroglial inclusion filaments in multiple system atrophy

Experimental neurology, 2003-05, Vol.181 (1), p.68-78

2003

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Nonselenium Glutathione Peroxidase in Human Brain : Elevated Levels in Parkinson's Disease and Dementia with Lewy Bodies

The American journal of pathology, 2002-09, Vol.161 (3), p.885-894

2002

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P25α immunoreactive but α-synuclein immunonegative neuronal inclusions in multiple system atrophy

Acta neuropathologica, 2006-02, Vol.111 (2), p.193-195

2006

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Localization of α-, β-, and γ-synuclein during neuronal development and alterations associated with the neuronal response to axonal trauma

Experimental neurology, 2003-07, Vol.182 (1), p.195-207

2003

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Molecular characterization of a novel dipeptidyl peptidase like 2-short form (DPL2-s) that is highly expressed in the brain and lacks dipeptidyl peptidase activity

Biochimica et biophysica acta, 2006, Vol.1764 (1), p.33-43

2006

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Alpha B-crystallin is a major component of glial cytoplasmic inclusions in multiple system atrophy

Neurotoxicity research, 2005, Vol.7 (1-2), p.77-85

2005

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Ubiquitin-positive degenerating neurites in the brainstem in Parkinson's disease

Brain (London, England : 1878), 1995-12, Vol.118 (6), p.1447-1459

1995

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Idiopathic Parkinson's disease: possible routes by which vulnerable neuronal types may be subject to neuroinvasion by an unknown pathogen

Proteasomal Inhibition by α-Synuclein Filaments and Oligomers

Calcium(II) selectively induces α‐synuclein annular oligomers via interaction with the C‐terminal domain

Parkin Localizes to the Lewy Bodies of Parkinson Disease and Dementia with Lewy Bodies

Extensive axonal Lewy neurites in Parkinson's disease: a novel pathological feature revealed by α-synuclein immunocytochemistry

In Situ and in Vitro Study of Colocalization and Segregation of α-Synuclein, Ubiquitin, and Lipids in Lewy Bodies

SUMO-1 marks subdomains within glial cytoplasmic inclusions of multiple system atrophy

SUMO-1 marks the nuclear inclusions in familial neuronal intranuclear inclusion disease

Septin 3 (G‐septin) is a developmentally regulated phosphoprotein enriched in presynaptic nerve terminals

The solubility of α‐synuclein in multiple system atrophy differs from that of dementia with Lewy bodies and Parkinson's disease

Multiple-system atrophy: a new α-synuclein disease?

Annular α‐synuclein species from purified multiple system atrophy inclusions

α‐Synuclein Immunoisolation of Glial Inclusions from Multiple System Atrophy Brain Tissue Reveals Multiprotein Components

Microtubule-associated Protein 1B Is a Component of Cortical Lewy Bodies and Binds α-Synuclein Filaments

Dominantly-inherited adult-onset leukodystrophy with palatal tremor caused by a mutation in the glial fibrillary acidic protein gene

Parkinson's disease : affection of brain stem nuclei controlling premotor and motor neurons of the somatomotor system

Annular alpha-synuclein oligomers are potentially toxic agents in alpha-synucleinopathy. Hypothesis

α-Synuclein filaments bind the transcriptional regulator HMGB-1

α-synuclein fibrils constitute the central core of oligodendroglial inclusion filaments in multiple system atrophy

Nonselenium Glutathione Peroxidase in Human Brain : Elevated Levels in Parkinson's Disease and Dementia with Lewy Bodies

P25α immunoreactive but α-synuclein immunonegative neuronal inclusions in multiple system atrophy

Localization of α-, β-, and γ-synuclein during neuronal development and alterations associated with the neuronal response to axonal trauma

Molecular characterization of a novel dipeptidyl peptidase like 2-short form (DPL2-s) that is highly expressed in the brain and lacks dipeptidyl peptidase activity

Alpha B-crystallin is a major component of glial cytoplasmic inclusions in multiple system atrophy

Ubiquitin-positive degenerating neurites in the brainstem in Parkinson's disease

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