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Zika Virus NS3 Mimics a Cellular 14-3-3-Binding Motif to Antagonize RIG-I- and MDA5-Mediated Innate Immunity

Cell host & microbe, 2019-10, Vol.26 (4), p.493-503.e6

2019

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Viral evasion of intracellular DNA and RNA sensing

Nature reviews. Microbiology, 2016-06, Vol.14 (6), p.360-373

2016

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Post-translational Control of Intracellular Pathogen Sensing Pathways

Trends in immunology, 2017-01, Vol.38 (1), p.39-52

2017

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A phosphomimetic-based mechanism of dengue virus to antagonize innate immunity

Nature immunology, 2016-05, Vol.17 (5), p.523-530

2016

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Dephosphorylation of the RNA Sensors RIG-I and MDA5 by the Phosphatase PP1 Is Essential for Innate Immune Signaling

Immunity (Cambridge, Mass.), 2013-03, Vol.38 (3), p.437-449

2013

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RIG-I-like receptors: their regulation and roles in RNA sensing

Nature reviews. Immunology, 2020-09, Vol.20 (9), p.537-551

2020

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An optimized circular polymerase extension reaction-based method for functional analysis of SARS-CoV-2

Virology journal, 2023-04, Vol.20 (1), p.63-63, Article 63

2023

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A network medicine approach to investigation and population-based validation of disease manifestations and drug repurposing for COVID-19

PLoS biology, 2020-11, Vol.18 (11), p.e3000970

2020

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Species-specific inhibition of RIG-I ubiquitination and IFN induction by the influenza A virus NS1 protein

PLoS pathogens, 2012-11, Vol.8 (11), p.e1003059-e1003059

2012

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Regulation of RIG-I-like receptor signaling by host and viral proteins

Cytokine & growth factor reviews, 2014-10, Vol.25 (5), p.491-505

2014

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The Human Papillomavirus E6 Oncoprotein Targets USP15 and TRIM25 To Suppress RIG-I-Mediated Innate Immune Signaling

Journal of virology, 2018-03, Vol.92 (6)

2018

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Evasion of Innate and Intrinsic Antiviral Pathways by the Zika Virus

Viruses, 2019-10, Vol.11 (10), p.970

2019

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Mechanism of TRIM25 Catalytic Activation in the Antiviral RIG-I Pathway

Cell reports (Cambridge), 2016-08, Vol.16 (5), p.1315-1325

2016

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Mutations derived from horseshoe bat ACE2 orthologs enhance ACE2-Fc neutralization of SARS-CoV-2

PLoS pathogens, 2021-04, Vol.17 (4), p.e1009501-e1009501

2021

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ISG15-dependent activation of the sensor MDA5 is antagonized by the SARS-CoV-2 papain-like protease to evade host innate immunity

Nature microbiology, 2021-04, Vol.6 (4), p.467-478

2021

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Viral Evasion of RIG-I-Like Receptor-Mediated Immunity through Dysregulation of Ubiquitination and ISGylation

Viruses, 2021-01, Vol.13 (2), p.182

2021

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High-throughput RNA sequencing of paraformaldehyde-fixed single cells

Nature communications, 2021-09, Vol.12 (1), p.5636-11, Article 5636

2021

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Antagonism of the Phosphatase PP1 by the Measles Virus V Protein Is Required for Innate Immune Escape of MDA5

Cell host & microbe, 2014-07, Vol.16 (1), p.19-30

2014

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Nonsense-mediated decay controls the reactivation of the oncogenic herpesviruses EBV and KSHV

PLoS biology, 2021-02, Vol.19 (2), p.e3001097-e3001097

2021

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TRIM proteins: New players in virus-induced autophagy

PLoS pathogens, 2018-02, Vol.14 (2), p.e1006787-e1006787

2018

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Beclin1-binding UVRAG targets the class C Vps complex to coordinate autophagosome maturation and endocytic trafficking

Nature cell biology, 2008-07, Vol.10 (7), p.776-787

2008

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The ubiquitin-specific protease USP15 promotes RIG-I-mediated antiviral signaling by deubiquitylating TRIM25

Science signaling, 2014-01, Vol.7 (307), p.ra3

2014

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Cytosolic Viral Sensor RIG-I Is a 5′-Triphosphate-Dependent Translocase on Double-Stranded RNA

Science (American Association for the Advancement of Science), 2009-02, Vol.323 (5917), p.1070-1074

2009

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Dysregulation of type I interferon responses in COVID-19

Nature reviews. Immunology, 2020-07, Vol.20 (7), p.397-398

2020

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Prostaglandin E2: the Villain in the Host Response to Influenza Virus

Immunity (Cambridge, Mass.), 2014-04, Vol.40 (4), p.453-454

2014

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Zika Virus NS3 Mimics a Cellular 14-3-3-Binding Motif to Antagonize RIG-I- and MDA5-Mediated Innate Immunity

Viral evasion of intracellular DNA and RNA sensing

Post-translational Control of Intracellular Pathogen Sensing Pathways

A phosphomimetic-based mechanism of dengue virus to antagonize innate immunity

Dephosphorylation of the RNA Sensors RIG-I and MDA5 by the Phosphatase PP1 Is Essential for Innate Immune Signaling

RIG-I-like receptors: their regulation and roles in RNA sensing

An optimized circular polymerase extension reaction-based method for functional analysis of SARS-CoV-2

A network medicine approach to investigation and population-based validation of disease manifestations and drug repurposing for COVID-19

Species-specific inhibition of RIG-I ubiquitination and IFN induction by the influenza A virus NS1 protein

Regulation of RIG-I-like receptor signaling by host and viral proteins

The Human Papillomavirus E6 Oncoprotein Targets USP15 and TRIM25 To Suppress RIG-I-Mediated Innate Immune Signaling

Evasion of Innate and Intrinsic Antiviral Pathways by the Zika Virus

Mechanism of TRIM25 Catalytic Activation in the Antiviral RIG-I Pathway

Mutations derived from horseshoe bat ACE2 orthologs enhance ACE2-Fc neutralization of SARS-CoV-2

ISG15-dependent activation of the sensor MDA5 is antagonized by the SARS-CoV-2 papain-like protease to evade host innate immunity

Viral Evasion of RIG-I-Like Receptor-Mediated Immunity through Dysregulation of Ubiquitination and ISGylation

High-throughput RNA sequencing of paraformaldehyde-fixed single cells

Antagonism of the Phosphatase PP1 by the Measles Virus V Protein Is Required for Innate Immune Escape of MDA5

Nonsense-mediated decay controls the reactivation of the oncogenic herpesviruses EBV and KSHV

TRIM proteins: New players in virus-induced autophagy

Beclin1-binding UVRAG targets the class C Vps complex to coordinate autophagosome maturation and endocytic trafficking

The ubiquitin-specific protease USP15 promotes RIG-I-mediated antiviral signaling by deubiquitylating TRIM25

Cytosolic Viral Sensor RIG-I Is a 5′-Triphosphate-Dependent Translocase on Double-Stranded RNA

Dysregulation of type I interferon responses in COVID-19

Prostaglandin E2: the Villain in the Host Response to Influenza Virus

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